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Pharmacologic ATF6 activation confers global protection in widespread disease models by reprograming cellular proteostasis

Nature communications, 2019-01, Vol.10 (1), p.187-16, Article 187

2019

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ATF6 Regulates Cardiac Hypertrophy by Transcriptional Induction of the mTORC1 Activator, Rheb

Circulation research, 2019-01, Vol.124 (1), p.79-93

2019

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Pharmacologic ATF6 activating compounds are metabolically activated to selectively modify endoplasmic reticulum proteins

eLife, 2018-08, Vol.7

2018

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Dietary choline intake is necessary to prevent systems‐wide organ pathology and reduce Alzheimer's disease hallmarks

Aging cell, 2023-02, Vol.22 (2), p.e13775-n/a

2023

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ATF6 as a Nodal Regulator of Proteostasis in the Heart

Frontiers in physiology, 2020-04, Vol.11, p.267-267

2020

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The ER Unfolded Protein Response Effector, ATF6, Reduces Cardiac Fibrosis and Decreases Activation of Cardiac Fibroblasts

International journal of molecular sciences, 2020-02, Vol.21 (4), p.1373

2020

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Sledgehammer to Scalpel: Broad Challenges to the Heart and Other Tissues Yield Specific Cellular Responses via Transcriptional Regulation of the ER-Stress Master Regulator ATF6α

International journal of molecular sciences, 2020-02, Vol.21 (3), p.1134

2020

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Integrating ER and Mitochondrial Proteostasis in the Healthy and Diseased Heart

Frontiers in cardiovascular medicine, 2020-01, Vol.6, p.193-193

2020

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Designing Novel Therapies to Mend Broken Hearts: ATF6 and Cardiac Proteostasis

Cells (Basel, Switzerland), 2020-03, Vol.9 (3), p.602

2020

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ATF6 Decreases Myocardial Ischemia/Reperfusion Damage and Links ER Stress and Oxidative Stress Signaling Pathways in the Heart

Circulation research, 2017-03, Vol.120 (5), p.862-875

2017

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Unfolding the Roles of Mitochondria as Therapeutic Targets for Heart Disease

Journal of the American College of Cardiology, 2019-04, Vol.73 (14), p.1807-1810

2019

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The peroxisomal enzyme, FAR1, is induced during ER stress in an ATF6-dependent manner in cardiac myocytes

American journal of physiology. Heart and circulatory physiology, 2021-05, Vol.320 (5), p.H1813-H1821

2021

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Optimizing Adeno-Associated Virus Serotype 9 for Studies of Cardiac Chamber-Specific Gene Regulation

Circulation (New York, N.Y.), 2021-05, Vol.143 (20), p.2025-2027

2021

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Mesencephalic astrocyte–derived neurotrophic factor is an ER-resident chaperone that protects against reductive stress in the heart

The Journal of biological chemistry, 2020-05, Vol.295 (22), p.7566-7583

2020

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Design and Production of Heart Chamber-Specific AAV9 Vectors

Methods in molecular biology (Clifton, N.J.), 2022, Vol.2573, p.89

2022

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ATF6 protects against protein misfolding during cardiac hypertrophy

Journal of molecular and cellular cardiology, 2024-04, Vol.189, p.12-24

2024

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CaMKIIδ subtypes differentially regulate infarct formation following ex vivo myocardial ischemia/reperfusion through NF-κB and TNF-α

Journal of molecular and cellular cardiology, 2017-02, Vol.103, p.48-55

2017

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ER-Specific Autophagy or ER-Phagy in Cardiac Myocytes Protects the Heart Against Doxorubicin-Induced Cardiotoxicity

JACC CardioOncology, 2023-10, Vol.5 (5), p.671-673

2023

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Noncanonical Form of ERAD Regulates Cardiac Hypertrophy

Circulation (New York, N.Y.), 2023-01, Vol.147 (1), p.66-82

2023

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Proteomic analysis of the cardiac myocyte secretome reveals extracellular protective functions for the ER stress response

Journal of molecular and cellular cardiology, 2020-06, Vol.143, p.132-144

2020

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Reactive Oxygen Species (ROS)-Activatable Prodrug for Selective Activation of ATF6 after Ischemia/Reperfusion Injury

ACS medicinal chemistry letters, 2020-03, Vol.11 (3), p.292-297

2020

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Optimization of Large‐Scale Adeno‐Associated Virus (AAV) Production

Current protocols, 2023-05, Vol.3 (5), p.e757-n/a

2023

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Abstract 292: The Role of the ATF6 Branch of the ER Stress Response in the Endocrine Heart

Circulation research, 2016-07, Vol.119 (suppl_1)

2016

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ATF6β is an Adaptive Transcription Factor in Cardiac Myocyte

The FASEB journal, 2020-04, Vol.34 (S1), p.1-1

2020

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Small Nppa and Myl2 Promoters Are Sufficient to Maintain Chamber‐specific Expression on an AAV9 Platform

The FASEB journal, 2020-04, Vol.34 (S1), p.1-1

2020

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Pharmacologic ATF6 activation confers global protection in widespread disease models by reprograming cellular proteostasis

ATF6 Regulates Cardiac Hypertrophy by Transcriptional Induction of the mTORC1 Activator, Rheb

Pharmacologic ATF6 activating compounds are metabolically activated to selectively modify endoplasmic reticulum proteins

Dietary choline intake is necessary to prevent systems‐wide organ pathology and reduce Alzheimer's disease hallmarks

ATF6 as a Nodal Regulator of Proteostasis in the Heart

The ER Unfolded Protein Response Effector, ATF6, Reduces Cardiac Fibrosis and Decreases Activation of Cardiac Fibroblasts

Sledgehammer to Scalpel: Broad Challenges to the Heart and Other Tissues Yield Specific Cellular Responses via Transcriptional Regulation of the ER-Stress Master Regulator ATF6α

Integrating ER and Mitochondrial Proteostasis in the Healthy and Diseased Heart

Designing Novel Therapies to Mend Broken Hearts: ATF6 and Cardiac Proteostasis

ATF6 Decreases Myocardial Ischemia/Reperfusion Damage and Links ER Stress and Oxidative Stress Signaling Pathways in the Heart

Unfolding the Roles of Mitochondria as Therapeutic Targets for Heart Disease

The peroxisomal enzyme, FAR1, is induced during ER stress in an ATF6-dependent manner in cardiac myocytes

Optimizing Adeno-Associated Virus Serotype 9 for Studies of Cardiac Chamber-Specific Gene Regulation

Mesencephalic astrocyte–derived neurotrophic factor is an ER-resident chaperone that protects against reductive stress in the heart

Design and Production of Heart Chamber-Specific AAV9 Vectors

ATF6 protects against protein misfolding during cardiac hypertrophy

CaMKIIδ subtypes differentially regulate infarct formation following ex vivo myocardial ischemia/reperfusion through NF-κB and TNF-α

ER-Specific Autophagy or ER-Phagy in Cardiac Myocytes Protects the Heart Against Doxorubicin-Induced Cardiotoxicity

Noncanonical Form of ERAD Regulates Cardiac Hypertrophy

Proteomic analysis of the cardiac myocyte secretome reveals extracellular protective functions for the ER stress response

Reactive Oxygen Species (ROS)-Activatable Prodrug for Selective Activation of ATF6 after Ischemia/Reperfusion Injury

Optimization of Large‐Scale Adeno‐Associated Virus (AAV) Production

Abstract 292: The Role of the ATF6 Branch of the ER Stress Response in the Endocrine Heart

ATF6β is an Adaptive Transcription Factor in Cardiac Myocyte

Small Nppa and Myl2 Promoters Are Sufficient to Maintain Chamber‐specific Expression on an AAV9 Platform

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