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Crosstalk Between the Endoplasmic Reticulum and mTOR Signaling

Circulation research, 2019-01, Vol.124 (1), p.9-11

2019

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Editorial: Metabolic Regulation of Cardiac and Vascular Cell Function: Physiological and Pathophysiological Implications

Frontiers in physiology, 2022-02, Vol.13, p.849869-849869

2022

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Myocardial AKT: the omnipresent nexus

Physiological reviews, 2011-07, Vol.91 (3), p.1023-1070

2011

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Hrd1 and ER-Associated Protein Degradation, ERAD, Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes

Circulation research, 2015-08, Vol.117 (6), p.536-546

2015

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Pim-1 preserves mitochondrial morphology by inhibiting dynamin-related protein 1 translocation

Proceedings of the National Academy of Sciences - PNAS, 2013-04, Vol.110 (15), p.5969-5974

2013

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Mechanistic Target of Rapamycin Complex 2 Protects the Heart From Ischemic Damage

Circulation (New York, N.Y.), 2013-11, Vol.128 (19), p.2132-2144

2013

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Fibronectin Is Essential for Reparative Cardiac Progenitor Cell Response After Myocardial Infarction

Circulation research, 2013-07, Vol.113 (2), p.115-125

2013

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Cardiac S100A1 protein levels determine contractile performance and propensity toward heart failure after myocardial infarction

Circulation (New York, N.Y.), 2006-09, Vol.114 (12), p.1258-1268

2006

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Pathological hypertrophy amelioration by PRAS40-mediated inhibition of mTORC1

Proceedings of the National Academy of Sciences - PNAS, 2013-07, Vol.110 (31), p.12661-12666

2013

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S100A4 protects the myocardium against ischemic stress

Journal of molecular and cellular cardiology, 2016-11, Vol.100, p.54-63

2016

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Identification of Cardiac Troponin I Sequence Motifs Leading to Heart Failure by Induction of Myocardial Inflammation and Fibrosis

Circulation (New York, N.Y.), 2008-11, Vol.118 (20), p.2063-2072

2008

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Pim-1 Kinase Protects Mitochondrial Integrity in Cardiomyocytes

Circulation research, 2010-04, Vol.106 (7), p.1265-1274

2010

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Mitochondrial translocation of Nur77 mediates cardiomyocyte apoptosis

European heart journal, 2011-09, Vol.32 (17), p.2179-2188

2011

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S100A1 is released from ischemic cardiomyocytes and signals myocardial damage via Toll‐like receptor 4

EMBO molecular medicine, 2014-06, Vol.6 (6), p.778-794

2014

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Comparative Transcriptomics of Immune Checkpoint Inhibitor Myocarditis Identifies Guanylate Binding Protein 5 and 6 Dysregulation

Cancers, 2021-05, Vol.13 (10), p.2498

2021

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Corrigendum: A Multi-Network Comparative Analysis of Transcriptome and Translatome Identifies Novel Hub Genes in Cardiac Remodeling

Frontiers in genetics, 2021-06, Vol.12, p.706542-706542

2021

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Nucleolar stress is an early response to myocardial damage involving nucleolar proteins nucleostemin and nucleophosmin

Proceedings of the National Academy of Sciences - PNAS, 2011-04, Vol.108 (15), p.6145-6150

2011

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Inhibition of Cardiac Kir Current (IK1) by Protein Kinase C Critically Depends on PKCβ and Kir2.2

PloS one, 2016-05, Vol.11 (5), p.e0156181-e0156181

2016

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Regulation of Cardiac Hypertrophic Signaling by Prolyl Isomerase Pin1

Circulation research, 2013-04, Vol.112 (9), p.1244-1252

2013

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A Multi-Network Comparative Analysis of Transcriptome and Translatome Identifies Novel Hub Genes in Cardiac Remodeling

Frontiers in genetics, 2020-11, Vol.11, p.583124-583124

2020

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TIP30 counteracts cardiac hypertrophy and failure by inhibiting translational elongation

EMBO molecular medicine, 2019-10, Vol.11 (10), p.e10018-n/a

2019

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Myoscape controls cardiac calcium cycling and contractility via regulation of L-type calcium channel surface expression

Nature communications, 2016-04, Vol.7 (1), p.11317-16, Article 11317

2016

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Analysis of myocardial cellular gene expression during pressure overload reveals matrix based functional intercellular communication

iScience, 2022-03, Vol.25 (3), p.103965-103965, Article 103965

2022

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Pim-1 kinase inhibits pathological injury by promoting cardioprotective signaling

Journal of molecular and cellular cardiology, 2011-10, Vol.51 (4), p.554-558

2011

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Crosstalk Between the Endoplasmic Reticulum and mTOR Signaling

Editorial: Metabolic Regulation of Cardiac and Vascular Cell Function: Physiological and Pathophysiological Implications

Myocardial AKT: the omnipresent nexus

Hrd1 and ER-Associated Protein Degradation, ERAD, Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes

Pim-1 preserves mitochondrial morphology by inhibiting dynamin-related protein 1 translocation

Mechanistic Target of Rapamycin Complex 2 Protects the Heart From Ischemic Damage

Fibronectin Is Essential for Reparative Cardiac Progenitor Cell Response After Myocardial Infarction

Cardiac S100A1 protein levels determine contractile performance and propensity toward heart failure after myocardial infarction

Pathological hypertrophy amelioration by PRAS40-mediated inhibition of mTORC1

S100A4 protects the myocardium against ischemic stress

Identification of Cardiac Troponin I Sequence Motifs Leading to Heart Failure by Induction of Myocardial Inflammation and Fibrosis

Pim-1 Kinase Protects Mitochondrial Integrity in Cardiomyocytes

Mitochondrial translocation of Nur77 mediates cardiomyocyte apoptosis

S100A1 is released from ischemic cardiomyocytes and signals myocardial damage via Toll‐like receptor 4

Comparative Transcriptomics of Immune Checkpoint Inhibitor Myocarditis Identifies Guanylate Binding Protein 5 and 6 Dysregulation

Genomic structural variations lead to dysregulation of important coding and non‐coding RNA species in dilated cardiomyopathy

Corrigendum: A Multi-Network Comparative Analysis of Transcriptome and Translatome Identifies Novel Hub Genes in Cardiac Remodeling

Nucleolar stress is an early response to myocardial damage involving nucleolar proteins nucleostemin and nucleophosmin

Inhibition of Cardiac Kir Current (IK1) by Protein Kinase C Critically Depends on PKCβ and Kir2.2

Regulation of Cardiac Hypertrophic Signaling by Prolyl Isomerase Pin1

A Multi-Network Comparative Analysis of Transcriptome and Translatome Identifies Novel Hub Genes in Cardiac Remodeling

TIP30 counteracts cardiac hypertrophy and failure by inhibiting translational elongation

Myoscape controls cardiac calcium cycling and contractility via regulation of L-type calcium channel surface expression

Analysis of myocardial cellular gene expression during pressure overload reveals matrix based functional intercellular communication

Pim-1 kinase inhibits pathological injury by promoting cardioprotective signaling

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