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ATF6 Decreases Myocardial Ischemia/Reperfusion Damage and Links ER Stress and Oxidative Stress Signaling Pathways in the Heart

Circulation research, 2017-03, Vol.120 (5), p.862-875

2017

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ER Stress-Induced Secretion of Proteins and Their Extracellular Functions in the Heart

Cells (Basel, Switzerland), 2020-09, Vol.9 (9), p.2066

2020

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Ischemia Activates the ATF6 Branch of the Endoplasmic Reticulum Stress Response

The Journal of biological chemistry, 2009-10, Vol.284 (43), p.29735-29745

2009

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Pathological hypertrophy amelioration by PRAS40-mediated inhibition of mTORC1

Proceedings of the National Academy of Sciences - PNAS, 2013-07, Vol.110 (31), p.12661-12666

2013

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VARS2 Depletion Leads to Activation of the Integrated Stress Response and Disruptions in Mitochondrial Fatty Acid Oxidation

International journal of molecular sciences, 2022-06, Vol.23 (13), p.7327

2022

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Transcriptional Effects of Candidate COVID-19 Treatments on Cardiac Myocytes

Frontiers in cardiovascular medicine, 2022-05, Vol.9, p.844441-844441

2022

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Corrigendum: A Multi-Network Comparative Analysis of Transcriptome and Translatome Identifies Novel Hub Genes in Cardiac Remodeling

Frontiers in genetics, 2021-06, Vol.12, p.706542-706542

2021

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S100A4 protects the myocardium against ischemic stress

Journal of molecular and cellular cardiology, 2016-11, Vol.100, p.54-63

2016

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A Multi-Network Comparative Analysis of Transcriptome and Translatome Identifies Novel Hub Genes in Cardiac Remodeling

Frontiers in genetics, 2020-11, Vol.11, p.583124-583124

2020

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TIP30 counteracts cardiac hypertrophy and failure by inhibiting translational elongation

EMBO molecular medicine, 2019-10, Vol.11 (10), p.e10018-n/a

2019

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S100A4 as a Target of the E3-Ligase Asb2β and Its Effect on Engineered Heart Tissue

Frontiers in physiology, 2018-09, Vol.9, p.1292-1292

2018

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IRE1/XBP1 and endoplasmic reticulum signaling — from basic to translational research for cardiovascular disease

Current opinion in physiology, 2022-08, Vol.28, p.100552, Article 100552

2022

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Protein Misfolding in Cardiac Disease

Circulation (New York, N.Y.), 2019-04, Vol.139 (18), p.2085-2088

2019

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New concepts of endoplasmic reticulum function in the heart: Programmed to conserve

Journal of molecular and cellular cardiology, 2013-02, Vol.55, p.85-91

2013

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The cardiokine story unfolds: ischemic stress-induced protein secretion in the heart

Trends in molecular medicine, 2011-04, Vol.17 (4), p.207-214

2011

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Mesencephalic Astrocyte-derived Neurotrophic Factor Protects the Heart from Ischemic Damage and Is Selectively Secreted upon Sarco/endoplasmic Reticulum Calcium Depletion

The Journal of biological chemistry, 2012-07, Vol.287 (31), p.25893-25904

2012

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ATF6 Regulates Cardiac Hypertrophy by Transcriptional Induction of the mTORC1 Activator, Rheb

Circulation research, 2019-01, Vol.124 (1), p.79-93

2019

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Abstract MP217: Nuclease-deficient Cas9 Transcription Factors Restore Krueppel-like Factor 15 Expression In Stressed Mouse And Human Cardiomyocytes

Circulation research, 2021-09, Vol.129 (Suppl_1)

2021

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Abstract 260: The ER Unfolded Protein Response Effector, ATF6, Reduces Fibrosis and Moderates Activation of Cardiac Fibroblasts

Circulation research, 2019-08, Vol.125 (Suppl_1)

2019

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Abstract 17305: The ER Unfolded Protein Response Effector, ATF6, Promotes Proliferation and Maintains Pluripotency in Cardiac Stem Cells

Circulation (New York, N.Y.), 2018-11, Vol.138 (Suppl_1 Suppl 1), p.A17305-A17305

2018

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Mesencephalic astrocyte–derived neurotrophic factor is an ER-resident chaperone that protects against reductive stress in the heart

The Journal of biological chemistry, 2020-05, Vol.295 (22), p.7566-7583

2020

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Monitoring Cell-Type–Specific Gene Expression Using Ribosome Profiling In Vivo During Cardiac Hemodynamic Stress

Circulation research, 2019-08, Vol.125 (4), p.431-448

2019

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CRISPR-Mediated Activation of Endogenous Gene Expression in the Postnatal Heart

Circulation research, 2020-01, Vol.126 (1), p.6-24

2020

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Hrd1 and ER-Associated Protein Degradation, ERAD, Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes

Circulation research, 2015-08, Vol.117 (6), p.536-546

2015

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Mesencephalic astrocyte–derived neurotrophic factor is an ER‐resident chaperone that protects against reductive stress in the heart

The FASEB journal, 2021-05, Vol.35 (S1), p.n/a, Article fasebj.2021.35.S1.04510

2021

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ATF6 Decreases Myocardial Ischemia/Reperfusion Damage and Links ER Stress and Oxidative Stress Signaling Pathways in the Heart

ER Stress-Induced Secretion of Proteins and Their Extracellular Functions in the Heart

Ischemia Activates the ATF6 Branch of the Endoplasmic Reticulum Stress Response

Pathological hypertrophy amelioration by PRAS40-mediated inhibition of mTORC1

VARS2 Depletion Leads to Activation of the Integrated Stress Response and Disruptions in Mitochondrial Fatty Acid Oxidation

Transcriptional Effects of Candidate COVID-19 Treatments on Cardiac Myocytes

Corrigendum: A Multi-Network Comparative Analysis of Transcriptome and Translatome Identifies Novel Hub Genes in Cardiac Remodeling

S100A4 protects the myocardium against ischemic stress

A Multi-Network Comparative Analysis of Transcriptome and Translatome Identifies Novel Hub Genes in Cardiac Remodeling

TIP30 counteracts cardiac hypertrophy and failure by inhibiting translational elongation

S100A4 as a Target of the E3-Ligase Asb2β and Its Effect on Engineered Heart Tissue

IRE1/XBP1 and endoplasmic reticulum signaling — from basic to translational research for cardiovascular disease

Protein Misfolding in Cardiac Disease

New concepts of endoplasmic reticulum function in the heart: Programmed to conserve

The cardiokine story unfolds: ischemic stress-induced protein secretion in the heart

Mesencephalic Astrocyte-derived Neurotrophic Factor Protects the Heart from Ischemic Damage and Is Selectively Secreted upon Sarco/endoplasmic Reticulum Calcium Depletion

ATF6 Regulates Cardiac Hypertrophy by Transcriptional Induction of the mTORC1 Activator, Rheb

Abstract MP217: Nuclease-deficient Cas9 Transcription Factors Restore Krueppel-like Factor 15 Expression In Stressed Mouse And Human Cardiomyocytes

Abstract 260: The ER Unfolded Protein Response Effector, ATF6, Reduces Fibrosis and Moderates Activation of Cardiac Fibroblasts

Abstract 17305: The ER Unfolded Protein Response Effector, ATF6, Promotes Proliferation and Maintains Pluripotency in Cardiac Stem Cells

Mesencephalic astrocyte–derived neurotrophic factor is an ER-resident chaperone that protects against reductive stress in the heart

Monitoring Cell-Type–Specific Gene Expression Using Ribosome Profiling In Vivo During Cardiac Hemodynamic Stress

CRISPR-Mediated Activation of Endogenous Gene Expression in the Postnatal Heart

Hrd1 and ER-Associated Protein Degradation, ERAD, Are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes

Mesencephalic astrocyte–derived neurotrophic factor is an ER‐resident chaperone that protects against reductive stress in the heart

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