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The key role of solvent in condensation: Mapping water in liquid-liquid phase-separated FUS

Biophysical journal, 2021-04, Vol.120 (7), p.1266-1275

2021

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The two faces of protein misfolding: gain- and loss-of-function in neurodegenerative diseases

The EMBO journal, 2008-01, Vol.27 (2), p.336-349

2008

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The G127V variant of the prion protein interferes with dimer formation in vitro but not in cellulo

Scientific reports, 2021-02, Vol.11 (1), p.3116-13, Article 3116

2021

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The N-terminus of the prion protein is a toxic effector regulated by the C-terminus

eLife, 2017-05, Vol.6

2017

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Prion Disease: A Tale of Folds and Strains

Brain pathology (Zurich, Switzerland), 2013-05, Vol.23 (3), p.321-332

2013

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Loss of Parkin or PINK1 Function Increases Drp1-dependent Mitochondrial Fragmentation

The Journal of biological chemistry, 2009-08, Vol.284 (34), p.22938-22951

2009

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The cellular prion protein interacts with and promotes the activity of Na,K-ATPases

PloS one, 2021-11, Vol.16 (11), p.e0258682-e0258682

2021

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GPI-anchor signal sequence influences PrPC sorting, shedding and signalling, and impacts on different pathomechanistic aspects of prion disease in mice

PLoS pathogens, 2019-01, Vol.15 (1), p.e1007520-e1007520

2019

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Cardiac glycoside-mediated turnover of Na, K-ATPases as a rational approach to reducing cell surface levels of the cellular prion protein

PloS one, 2022-07, Vol.17 (7), p.e0270915-e0270915

2022

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The E3 Ligase Parkin Maintains Mitochondrial Integrity by Increasing Linear Ubiquitination of NEMO

Molecular cell, 2013-03, Vol.49 (5), p.908-921

2013

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Bivalent metal ions induce formation of α-synuclein fibril polymorphs with different cytotoxicities

Scientific reports, 2022-07, Vol.12 (1), p.11898-11898, Article 11898

2022

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SecY-mediated quality control prevents the translocation of non-gated porins

Scientific reports, 2020-10, Vol.10 (1), p.16347-16347, Article 16347

2020

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Parkin cooperates with GDNF/RET signaling to prevent dopaminergic neuron degeneration

The Journal of clinical investigation, 2015-05, Vol.125 (5), p.1873-1885

2015

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Alterations in the brain interactome of the intrinsically disordered N-terminal domain of the cellular prion protein (PrPC) in Alzheimer's disease

PloS one, 2018-05, Vol.13 (5), p.e0197659

2018

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The N-terminus of Sec61p plays key roles in ER protein import and ERAD

PloS one, 2019-04, Vol.14 (4), p.e0215950-e0215950

2019

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Transgenic Overexpression of the Disordered Prion Protein N1 Fragment in Mice Does Not Protect Against Neurodegenerative Diseases Due to Impaired ER Translocation

Molecular neurobiology, 2020-06, Vol.57 (6), p.2812-2829

2020

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Enhancement of binding avidity by bivalent binding enables PrPSc-specific detection by anti-PrP monoclonal antibody 132

PloS one, 2019-06, Vol.14 (6), p.e0217944-e0217944

2019

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Structural and mechanistic aspects influencing the ADAM10-mediated shedding of the prion protein

Molecular neurodegeneration, 2018-04, Vol.13 (1), p.18-18, Article 18

2018

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p53 Aggregates penetrate cells and induce the co-aggregation of intracellular p53

PloS one, 2013-07, Vol.8 (7), p.e69242-e69242

2013

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NEMO reshapes the α-Synuclein aggregate interface and acts as an autophagy adapter by co-condensation with p62

Nature communications, 2023-12, Vol.14 (1), p.8368-8368, Article 8368

2023

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Dimerization of the cellular prion protein inhibits propagation of scrapie prions

The Journal of biological chemistry, 2018-05, Vol.293 (21), p.8020-8031

2018

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Biological Functions of the Intrinsically Disordered N-Terminal Domain of the Prion Protein: A Possible Role of Liquid-Liquid Phase Separation

Biomolecules (Basel, Switzerland), 2021-08, Vol.11 (8), p.1201

2021

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Genome-Wide Screening of Genes Required for Glycosylphosphatidylinositol Biosynthesis

PloS one, 2015-09, Vol.10 (9), p.e0138553

2015

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Substitutions of PrP N-terminal histidine residues modulate scrapie disease pathogenesis and incubation time in transgenic mice

PloS one, 2017-12, Vol.12 (12), p.e0188989-e0188989

2017

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The key role of solvent in condensation: Mapping water in liquid-liquid phase-separated FUS

The two faces of protein misfolding: gain- and loss-of-function in neurodegenerative diseases

The G127V variant of the prion protein interferes with dimer formation in vitro but not in cellulo

The N-terminus of the prion protein is a toxic effector regulated by the C-terminus

Prion Disease: A Tale of Folds and Strains

Loss of Parkin or PINK1 Function Increases Drp1-dependent Mitochondrial Fragmentation

The cellular prion protein interacts with and promotes the activity of Na,K-ATPases

GPI-anchor signal sequence influences PrPC sorting, shedding and signalling, and impacts on different pathomechanistic aspects of prion disease in mice

Cardiac glycoside-mediated turnover of Na, K-ATPases as a rational approach to reducing cell surface levels of the cellular prion protein

The E3 Ligase Parkin Maintains Mitochondrial Integrity by Increasing Linear Ubiquitination of NEMO

A protein quality control pathway regulated by linear ubiquitination

Bivalent metal ions induce formation of α-synuclein fibril polymorphs with different cytotoxicities

SecY-mediated quality control prevents the translocation of non-gated porins

Parkin cooperates with GDNF/RET signaling to prevent dopaminergic neuron degeneration

Alterations in the brain interactome of the intrinsically disordered N-terminal domain of the cellular prion protein (PrPC) in Alzheimer's disease

The N-terminus of Sec61p plays key roles in ER protein import and ERAD

Transgenic Overexpression of the Disordered Prion Protein N1 Fragment in Mice Does Not Protect Against Neurodegenerative Diseases Due to Impaired ER Translocation

Enhancement of binding avidity by bivalent binding enables PrPSc-specific detection by anti-PrP monoclonal antibody 132

Structural and mechanistic aspects influencing the ADAM10-mediated shedding of the prion protein

p53 Aggregates penetrate cells and induce the co-aggregation of intracellular p53

NEMO reshapes the α-Synuclein aggregate interface and acts as an autophagy adapter by co-condensation with p62

Dimerization of the cellular prion protein inhibits propagation of scrapie prions

Biological Functions of the Intrinsically Disordered N-Terminal Domain of the Prion Protein: A Possible Role of Liquid-Liquid Phase Separation

Genome-Wide Screening of Genes Required for Glycosylphosphatidylinositol Biosynthesis

Substitutions of PrP N-terminal histidine residues modulate scrapie disease pathogenesis and incubation time in transgenic mice

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