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MicroRNA-125b induces tau hyperphosphorylation and cognitive deficits in Alzheimer's disease

The EMBO journal, 2014-08, Vol.33 (15), p.1667-1680

2014

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Staging of Alzheimer disease-associated neurofibrillary pathology using paraffin sections and immunocytochemistry

Acta neuropathologica, 2006-10, Vol.112 (4), p.389-404

2006

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The FTD‐like syndrome causing TREM2 T66M mutation impairs microglia function, brain perfusion, and glucose metabolism

The EMBO journal, 2017-07, Vol.36 (13), p.1837-1853

2017

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Alpha-Synuclein defects autophagy by impairing SNAP29-mediated autophagosome-lysosome fusion

Cell death & disease, 2021-09, Vol.12 (10), p.854-854, Article 854

2021

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Dipeptide repeat protein pathology in C9ORF72 mutation cases: clinico-pathological correlations

Acta neuropathologica, 2013-12, Vol.126 (6), p.859-879

2013

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C9orf72 FTLD/ALS-associated Gly-Ala dipeptide repeat proteins cause neuronal toxicity and Unc119 sequestration

Acta neuropathologica, 2014-10, Vol.128 (4), p.485-503

2014

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The phenotypic spectrum of progressive supranuclear palsy: A retrospective multicenter study of 100 definite cases

Movement disorders, 2014-12, Vol.29 (14), p.1758-1766

2014

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Distribution of dipeptide repeat proteins in cellular models and C9orf72 mutation cases suggests link to transcriptional silencing

Acta neuropathologica, 2015-10, Vol.130 (4), p.537-555

2015

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In situ architecture of neuronal α-Synuclein inclusions

Nature communications, 2021-04, Vol.12 (1), p.2110-2110, Article 2110

2021

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The relation between tau pathology and granulovacuolar degeneration of neurons

Neurobiology of disease, 2021-01, Vol.147, p.105138-105138, Article 105138

2021

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PERK activation mitigates tau pathology in vitro and in vivo

EMBO molecular medicine, 2017-03, Vol.9 (3), p.371-384

2017

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Which ante mortem clinical features predict progressive supranuclear palsy pathology?

Movement disorders, 2017-07, Vol.32 (7), p.995-1005

2017

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Poly-glycine–alanine exacerbates C9orf72 repeat expansion-mediated DNA damage via sequestration of phosphorylated ATM and loss of nuclear hnRNPA3

Acta neuropathologica, 2020-01, Vol.139 (1), p.99-118

2020

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Parkinson's disease: SNCA- , PARK2-, and LRRK2- targeting microRNAs elevated in cingulate gyrus

Parkinsonism & related disorders, 2016-12, Vol.33, p.115-121

2016

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DNA methylation analysis on purified neurons and glia dissects age and Alzheimer's disease-specific changes in the human cortex

Epigenetics & chromatin, 2018-07, Vol.11 (1), p.41-41, Article 41

2018

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Common pathobiochemical hallmarks of progranulin-associated frontotemporal lobar degeneration and neuronal ceroid lipofuscinosis

Acta neuropathologica, 2014-06, Vol.127 (6), p.845-860

2014

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TDP-43 loss of function inhibits endosomal trafficking and alters trophic signaling in neurons

The EMBO journal, 2016-11, Vol.35 (21), p.2350-2370

2016

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Long-term in vivo imaging of fibrillar tau in the retina of P301S transgenic mice

PloS one, 2012-12, Vol.7 (12), p.e53547-e53547

2012

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Active poly‐GA vaccination prevents microglia activation and motor deficits in a C9orf72 mouse model

EMBO molecular medicine, 2020-02, Vol.12 (2), p.e10919-n/a

2020

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Genome-wide association study in essential tremor identifies three new loci

Brain (London, England : 1878), 2016-12, Vol.139 (Pt 12), p.3163-3169

2016

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Proteomics and C9orf72 neuropathology identify ribosomes as poly-GR/PR interactors driving toxicity

Life science alliance, 2018-05, Vol.1 (2), p.e201800070-e201800070

2018

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First symptom guides diagnosis and prognosis in neurodegenerative diseases—a retrospective study of autopsy proven cases

European journal of neurology, 2021-06, Vol.28 (6), p.1801-1811

2021

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Monomethylated and unmethylated FUS exhibit increased binding to Transportin and distinguish FTLD-FUS from ALS-FUS

Acta neuropathologica, 2016-04, Vol.131 (4), p.587-604

2016

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MicroRNA-125b induces tau hyperphosphorylation and cognitive deficits in Alzheimer's disease

Staging of Alzheimer disease-associated neurofibrillary pathology using paraffin sections and immunocytochemistry

The FTD‐like syndrome causing TREM2 T66M mutation impairs microglia function, brain perfusion, and glucose metabolism

Alpha-Synuclein defects autophagy by impairing SNAP29-mediated autophagosome-lysosome fusion

Dipeptide repeat protein pathology in C9ORF72 mutation cases: clinico-pathological correlations

C9orf72 FTLD/ALS-associated Gly-Ala dipeptide repeat proteins cause neuronal toxicity and Unc119 sequestration

The phenotypic spectrum of progressive supranuclear palsy: A retrospective multicenter study of 100 definite cases

Distribution of dipeptide repeat proteins in cellular models and C9orf72 mutation cases suggests link to transcriptional silencing

In situ architecture of neuronal α-Synuclein inclusions

How to apply the movement disorder society criteria for diagnosis of progressive supranuclear palsy

The relation between tau pathology and granulovacuolar degeneration of neurons

PERK activation mitigates tau pathology in vitro and in vivo

Which ante mortem clinical features predict progressive supranuclear palsy pathology?

Poly-glycine–alanine exacerbates C9orf72 repeat expansion-mediated DNA damage via sequestration of phosphorylated ATM and loss of nuclear hnRNPA3

Spinal poly-GA inclusions in a C9orf72 mouse model trigger motor deficits and inflammation without neuron loss

Parkinson's disease: SNCA- , PARK2-, and LRRK2- targeting microRNAs elevated in cingulate gyrus

DNA methylation analysis on purified neurons and glia dissects age and Alzheimer's disease-specific changes in the human cortex

Common pathobiochemical hallmarks of progranulin-associated frontotemporal lobar degeneration and neuronal ceroid lipofuscinosis

TDP-43 loss of function inhibits endosomal trafficking and alters trophic signaling in neurons

Long-term in vivo imaging of fibrillar tau in the retina of P301S transgenic mice

Active poly‐GA vaccination prevents microglia activation and motor deficits in a C9orf72 mouse model

Genome-wide association study in essential tremor identifies three new loci

Proteomics and C9orf72 neuropathology identify ribosomes as poly-GR/PR interactors driving toxicity

First symptom guides diagnosis and prognosis in neurodegenerative diseases—a retrospective study of autopsy proven cases

Monomethylated and unmethylated FUS exhibit increased binding to Transportin and distinguish FTLD-FUS from ALS-FUS

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