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Monomeric amyloid-β reduced amyloid-β oligomer-induced synapse damage in neuronal cultures

Neurobiology of disease, 2018-03, Vol.111, p.48-58

2018

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Can we switch production of toxic Aβ oligomers to neuroprotective Aβ monomers to allow synapse regeneration

Neural regeneration research, 2017-09, Vol.12 (9), p.1437-1438

Bate, Clive

2017

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Sialic Acid on the Glycosylphosphatidylinositol Anchor Regulates PrP-mediated Cell Signaling and Prion Formation

The Journal of biological chemistry, 2016-01, Vol.291 (1), p.160-170

2016

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Enhanced neuronal degradation of amyloid-β oligomers allows synapse regeneration

Neural regeneration research, 2015-05, Vol.10 (5), p.700-701

Bate, Clive

2015

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Sialic Acid within the Glycosylphosphatidylinositol Anchor Targets the Cellular Prion Protein to Synapses

The Journal of biological chemistry, 2016-08, Vol.291 (33), p.17093-17101

2016

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GPI-anchor signal sequence influences PrPC sorting, shedding and signalling, and impacts on different pathomechanistic aspects of prion disease in mice

PLoS pathogens, 2019-01, Vol.15 (1), p.e1007520-e1007520

2019

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cAMP-Inhibits Cytoplasmic Phospholipase A₂ and Protects Neurons against Amyloid-β-Induced Synapse Damage

Biology (Basel, Switzerland), 2015-09, Vol.4 (3), p.591-606

2015

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α-Synuclein-induced synapse damage in cultured neurons is mediated by cholesterol-sensitive activation of cytoplasmic phospholipase A2

Biomolecules (Basel, Switzerland), 2015-03, Vol.5 (1), p.178-193

2015

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Squalestatin Cures Prion-infected Neurons and Protects Against Prion Neurotoxicity

The Journal of biological chemistry, 2004-04, Vol.279 (15), p.14983-14990

2004

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The glycosylphosphatidylinositol anchor is a major determinant of prion binding and replication

Biochemical journal, 2010-04, Vol.428 (1), p.95-101

2010

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Amyloid-β-induced Synapse Damage Is Mediated via Cross-linkage of Cellular Prion Proteins

The Journal of biological chemistry, 2011-11, Vol.286 (44), p.37955-37963

2011

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A camelid anti-PrP antibody abrogates PrP replication in prion-permissive neuroblastoma cell lines

PloS one, 2010-03, Vol.5 (3), p.e9804

2010

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Glycosylphosphatidylinositols: More than just an anchor?

Communicative & integrative biology, 2016-03, Vol.9 (2), p.e1149671

2016

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Monoacylated Cellular Prion Proteins Reduce Amyloid-β-Induced Activation of Cytoplasmic Phospholipase A2 and Synapse Damage

Biology (Basel, Switzerland), 2015-06, Vol.4 (2), p.367-382

2015

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Interferon-γ increases neuronal death in response to amyloid-β1-42

Journal of neuroinflammation, 2006-03, Vol.3 (1), p.7-7

2006

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Clustering of sialylated glycosylphosphatidylinositol anchors mediates PrP-induced activation of cytoplasmic phospholipase A2 and synapse damage

Prion, 2012-09, Vol.6 (4), p.350-353

2012

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Sequestration of free cholesterol in cell membranes by prions correlates with cytoplasmic phospholipase A2 activation

BMC biology, 2008-02, Vol.6 (1), p.8-8

2008

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Neurodegeneration Induced by Clustering of Sialylated Glycosylphosphatidylinositols of Prion Proteins

The Journal of biological chemistry, 2012-03, Vol.287 (11), p.7935-7944

2012

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Phospholipase A2 Inhibitors or Platelet-activating Factor Antagonists Prevent Prion Replication

The Journal of biological chemistry, 2004-08, Vol.279 (35), p.36405-36411

2004

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Cholesterol ester hydrolase inhibitors reduce the production of synaptotoxic amyloid-β oligomers

Biochimica et biophysica acta. Molecular basis of disease, 2018-03, Vol.1864 (3), p.649-659

2018

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Can we switch production of toxic A[beta] oligomers to neuroprotective A[beta] monomers to allow synapse regeneration?

Neural regeneration research, 2017-09, Vol.12 (9), p.1437

Bate, Clive

2017

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The cellular prion protein with a monoacylated glycosylphosphatidylinositol anchor modifies cell membranes, inhibits cell signaling and reduces prion formation

Prion, 2011-04, Vol.5 (2), p.65-68

2011

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Ethanol protects cultured neurons against amyloid-β and α-synuclein-induced synapse damage

Neuropharmacology, 2011-12, Vol.61 (8), p.1406-1412

2011

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Cholesterol synthesis inhibitors protect against platelet-activating factor-induced neuronal damage

Journal of neuroinflammation, 2007, Vol.4 (1), p.5-5

2007

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Polyunsaturated Fatty Acids Protect Against Prion-Mediated Synapse Damage In Vitro

Neurotoxicity research, 2010-04, Vol.17 (3), p.203-214

2010

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Monomeric amyloid-β reduced amyloid-β oligomer-induced synapse damage in neuronal cultures

Can we switch production of toxic Aβ oligomers to neuroprotective Aβ monomers to allow synapse regeneration

Sialic Acid on the Glycosylphosphatidylinositol Anchor Regulates PrP-mediated Cell Signaling and Prion Formation

Enhanced neuronal degradation of amyloid-β oligomers allows synapse regeneration

Sialic Acid within the Glycosylphosphatidylinositol Anchor Targets the Cellular Prion Protein to Synapses

GPI-anchor signal sequence influences PrPC sorting, shedding and signalling, and impacts on different pathomechanistic aspects of prion disease in mice

cAMP-Inhibits Cytoplasmic Phospholipase A₂ and Protects Neurons against Amyloid-β-Induced Synapse Damage

α-Synuclein-induced synapse damage in cultured neurons is mediated by cholesterol-sensitive activation of cytoplasmic phospholipase A2

Squalestatin Cures Prion-infected Neurons and Protects Against Prion Neurotoxicity

The glycosylphosphatidylinositol anchor is a major determinant of prion binding and replication

Amyloid-β-induced Synapse Damage Is Mediated via Cross-linkage of Cellular Prion Proteins

A camelid anti-PrP antibody abrogates PrP replication in prion-permissive neuroblastoma cell lines

Glycosylphosphatidylinositols: More than just an anchor?

Monoacylated Cellular Prion Proteins Reduce Amyloid-β-Induced Activation of Cytoplasmic Phospholipase A2 and Synapse Damage

Interferon-γ increases neuronal death in response to amyloid-β1-42

Clustering of sialylated glycosylphosphatidylinositol anchors mediates PrP-induced activation of cytoplasmic phospholipase A2 and synapse damage

Sequestration of free cholesterol in cell membranes by prions correlates with cytoplasmic phospholipase A2 activation

Neurodegeneration Induced by Clustering of Sialylated Glycosylphosphatidylinositols of Prion Proteins

Phospholipase A2 Inhibitors or Platelet-activating Factor Antagonists Prevent Prion Replication

Cholesterol ester hydrolase inhibitors reduce the production of synaptotoxic amyloid-β oligomers

Can we switch production of toxic A[beta] oligomers to neuroprotective A[beta] monomers to allow synapse regeneration?

The cellular prion protein with a monoacylated glycosylphosphatidylinositol anchor modifies cell membranes, inhibits cell signaling and reduces prion formation

Ethanol protects cultured neurons against amyloid-β and α-synuclein-induced synapse damage

Cholesterol synthesis inhibitors protect against platelet-activating factor-induced neuronal damage

Polyunsaturated Fatty Acids Protect Against Prion-Mediated Synapse Damage In Vitro

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