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Loss of microRNA cluster miR-29a/b-1 in sporadic Alzheimer's disease correlates with increased BACE1/β-secretase expression

Proceedings of the National Academy of Sciences - PNAS, 2008-04, Vol.105 (17), p.6415-6420

2008

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MicroRNA regulation of Alzheimer's Amyloid precursor protein expression

Neurobiology of disease, 2009-03, Vol.33 (3), p.422-428

2009

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Mutagenesis Mapping of the Presenilin 1 Calcium Leak Conductance Pore

The Journal of biological chemistry, 2011-06, Vol.286 (25), p.22339-22347

2011

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Tetraspanin 6: a pivotal protein of the multiple vesicular body determining exosome release and lysosomal degradation of amyloid precursor protein fragments

Molecular neurodegeneration, 2017-03, Vol.12 (1), p.25-25, Article 25

2017

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Contribution of GABAergic interneurons to amyloid-β plaque pathology in an APP knock-in mouse model

Molecular neurodegeneration, 2020-01, Vol.15 (1), p.3-8, Article 3

2020

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Differential Contribution of the Three Aph1 Genes to γ-Secretase Activity in vivo

Proceedings of the National Academy of Sciences - PNAS, 2005-02, Vol.102 (5), p.1719-1724

2005

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Coordinated and widespread expression of γ-secretase in vivo: evidence for size and molecular heterogeneity

Neurobiology of disease, 2004-11, Vol.17 (2), p.260-272

2004

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MEG3 activates necroptosis in human neuron xenografts modeling Alzheimer’s disease

Science (American Association for the Advancement of Science), 2023-09, Vol.381 (6663), p.1176-1182

2023

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Restoring miR-132 expression rescues adult hippocampal neurogenesis and memory deficits in Alzheimer’s disease

Cell stem cell, 2021-10, Vol.28 (10), p.1805-1821.e8

2021

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Alzheimer’s-Causing Mutations Shift Aβ Length by Destabilizing γ-Secretase-Aβn Interactions

Cell, 2017-07, Vol.170 (3), p.443-456.e14

2017

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Orphan G Protein-Coupled Receptor 3 Modulates Amyloid-Beta Peptide Generation in Neurons

Science (American Association for the Advancement of Science), 2009-02, Vol.323 (5916), p.946-951

2009

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β-arrestin 2 regulates Aβ generation and γ-secretase activity in Alzheimer's disease

Nature medicine, 2013-01, Vol.19 (1), p.43-49

2013

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Astrocyte calcium dysfunction causes early network hyperactivity in Alzheimer’s disease

Cell reports (Cambridge), 2022-08, Vol.40 (8), p.111280-111280, Article 111280

2022

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PARL deficiency in mouse causes Complex III defects, coenzyme Q depletion, and Leigh-like syndrome

Proceedings of the National Academy of Sciences - PNAS, 2019-01, Vol.116 (1), p.277-286

2019

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Loss of GPR3 reduces the amyloid plaque burden and improves memory in Alzheimer's disease mouse models

Science translational medicine, 2015-10, Vol.7 (309), p.309ra164

2015

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Transmembrane Domain 9 of Presenilin Determines the Dynamic Conformation of the Catalytic Site of γ-Secretase

The Journal of biological chemistry, 2008-07, Vol.283 (28), p.19793-19803

2008

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Nuclear import of the DSCAM‐cytoplasmic domain drives signaling capable of inhibiting synapse formation

The EMBO journal, 2019-03, Vol.38 (6), p.n/a

2019

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The dynamic conformational landscape of gamma-secretase

Journal of cell science, 2015-02, Vol.128 (3), p.589-598

2015

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Presenilin clinical mutations can affect γ‐secretase activity by different mechanisms

Journal of neurochemistry, 2006-02, Vol.96 (3), p.732-742

2006

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Postnatal disruption of the disintegrin/metalloproteinase ADAM10 in brain causes epileptic seizures, learning deficits, altered spine morphology, and defective synaptic functions

The Journal of neuroscience, 2013-08, Vol.33 (32), p.12915-12928

2013

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Alzheimer’s-causing mutations shift Aβ length by destabilizing γ-secretase-Aβn interactions

Cell, 2021-04, Vol.184 (8), p.2257-2258

2021

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γ-Secretase Heterogeneity in the Aph1 Subunit: Relevance for Alzheimer's Disease

Science (American Association for the Advancement of Science), 2009-05, Vol.324 (5927), p.639-642

2009

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Astrocytes mediate neuronal network hyperactivity in early AD

Alzheimer's & dementia, 2022-12, Vol.18 (S4), p.n/a

2022

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Contribution of GABAergic interneurons to amyloid-[beta] plaque pathology in an APP knock-in mouse model

Molecular neurodegeneration, 2020-01, Vol.15 (1)

2020

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[beta]-arrestin 2 regulates A[beta] generation and [gamma]-secretase activity in Alzheimer's disease

Nature medicine, 2013-01, Vol.19 (1), p.43

2013

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Loss of microRNA cluster miR-29a/b-1 in sporadic Alzheimer's disease correlates with increased BACE1/β-secretase expression

MicroRNA regulation of Alzheimer's Amyloid precursor protein expression

Mutagenesis Mapping of the Presenilin 1 Calcium Leak Conductance Pore

Tetraspanin 6: a pivotal protein of the multiple vesicular body determining exosome release and lysosomal degradation of amyloid precursor protein fragments

Contribution of GABAergic interneurons to amyloid-β plaque pathology in an APP knock-in mouse model

Differential Contribution of the Three Aph1 Genes to γ-Secretase Activity in vivo

Coordinated and widespread expression of γ-secretase in vivo: evidence for size and molecular heterogeneity

MEG3 activates necroptosis in human neuron xenografts modeling Alzheimer’s disease

Restoring miR-132 expression rescues adult hippocampal neurogenesis and memory deficits in Alzheimer’s disease

Alzheimer’s-Causing Mutations Shift Aβ Length by Destabilizing γ-Secretase-Aβn Interactions

Orphan G Protein-Coupled Receptor 3 Modulates Amyloid-Beta Peptide Generation in Neurons

β-arrestin 2 regulates Aβ generation and γ-secretase activity in Alzheimer's disease

Astrocyte calcium dysfunction causes early network hyperactivity in Alzheimer’s disease

PARL deficiency in mouse causes Complex III defects, coenzyme Q depletion, and Leigh-like syndrome

Loss of GPR3 reduces the amyloid plaque burden and improves memory in Alzheimer's disease mouse models

Transmembrane Domain 9 of Presenilin Determines the Dynamic Conformation of the Catalytic Site of γ-Secretase

Nuclear import of the DSCAM‐cytoplasmic domain drives signaling capable of inhibiting synapse formation

The dynamic conformational landscape of gamma-secretase

Presenilin clinical mutations can affect γ‐secretase activity by different mechanisms

Postnatal disruption of the disintegrin/metalloproteinase ADAM10 in brain causes epileptic seizures, learning deficits, altered spine morphology, and defective synaptic functions

Alzheimer’s-causing mutations shift Aβ length by destabilizing γ-secretase-Aβn interactions

γ-Secretase Heterogeneity in the Aph1 Subunit: Relevance for Alzheimer's Disease

Astrocytes mediate neuronal network hyperactivity in early AD

Contribution of GABAergic interneurons to amyloid-[beta] plaque pathology in an APP knock-in mouse model

[beta]-arrestin 2 regulates A[beta] generation and [gamma]-secretase activity in Alzheimer's disease

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