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TREM2 mutations implicated in neurodegeneration impair cell surface transport and phagocytosis

Science translational medicine, 2014-07, Vol.6 (243), p.243ra86

2014

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Casein Kinase 2 dependent phosphorylation of eIF4B regulates BACE1 expression in Alzheimer's disease

Cell death & disease, 2021-08, Vol.12 (8), p.769-14, Article 769

2021

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Low Cholesterol Stimulates the Nonamyloidogenic Pathway by Its Effect on the α-Secretase ADAM 10

Proceedings of the National Academy of Sciences - PNAS, 2001-05, Vol.98 (10), p.5815-5820

2001

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Translational Repression of the Disintegrin and Metalloprotease ADAM10 by a Stable G-quadruplex Secondary Structure in Its 5′-Untranslated Region

The Journal of biological chemistry, 2011-12, Vol.286 (52), p.45063-45072

2011

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Constitutive and Regulated α -secretase Cleavage of Alzheimer's Amyloid Precursor Protein by a Disintegrin Metalloprotease

Proceedings of the National Academy of Sciences - PNAS, 1999-03, Vol.96 (7), p.3922-3927

1999

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Furin-, ADAM 10-, and γ-Secretase-Mediated Cleavage of a Receptor Tyrosine Phosphatase and Regulation of β-Catenin's Transcriptional Activity

Molecular and Cellular Biology, 2006-05, Vol.26 (10), p.3917-3934

2006

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Involvement of presenilin holoprotein upregulation in calcium dyshomeostasis of Alzheimer's disease

Journal of cellular and molecular medicine, 2013-02, Vol.17 (2), p.293-302

2013

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Expression of the Anti-amyloidogenic Secretase ADAM10 Is Suppressed by Its 5′-Untranslated Region

The Journal of biological chemistry, 2010-05, Vol.285 (21), p.15753-15760

2010

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Expression of the Alzheimer protease BACE1 is suppressed via its 5'-untranslated region

EMBO reports, 2004-06, Vol.5 (6), p.620-625

2004

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hnRNP A3 binds to GGGGCC repeats and is a constituent of p62-positive/TDP43-negative inclusions in the hippocampus of patients with C9orf72 mutations

Acta neuropathologica, 2013-03, Vol.125 (3), p.413-423

2013

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Postnatal disruption of the disintegrin/metalloproteinase ADAM10 in brain causes epileptic seizures, learning deficits, altered spine morphology, and defective synaptic functions

The Journal of neuroscience, 2013-08, Vol.33 (32), p.12915-12928

2013

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Function, regulation and therapeutic properties of β-secretase (BACE1)

Seminars in cell & developmental biology, 2009-04, Vol.20 (2), p.175-182

2009

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Phosphorylation of the Translation Initiation Factor eIF2α Increases BACE1 Levels and Promotes Amyloidogenesis

Neuron (Cambridge, Mass.), 2008-12, Vol.60 (6), p.988-1009

2008

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Rescue of progranulin deficiency associated with frontotemporal lobar degeneration by alkalizing reagents and inhibition of vacuolar ATPase

The Journal of neuroscience, 2011-02, Vol.31 (5), p.1885-1894

2011

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Insulin-degrading Enzyme Rapidly Removes the β-Amyloid Precursor Protein Intracellular Domain (AICD)

The Journal of biological chemistry, 2002-04, Vol.277 (16), p.13389-13393

2002

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Presenilin-dependent Intramembrane Proteolysis of CD44 Leads to the Liberation of Its Intracellular Domain and the Secretion of an Aβ-like Peptide

The Journal of biological chemistry, 2002-11, Vol.277 (47), p.44754-44759

2002

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Phosphorylation Regulates Intracellular Trafficking of β-Secretase

The Journal of biological chemistry, 2001-05, Vol.276 (18), p.14634-14641

2001

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Presenilin-1 Affects Trafficking and Processing of βAPP and Is Targeted in a Complex with Nicastrin to the Plasma Membrane

The Journal of cell biology, 2002-08, Vol.158 (3), p.551-561

2002

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Maturation and Pro-peptide Cleavage of β-Secretase

The Journal of biological chemistry, 2000-10, Vol.275 (40), p.30849-30854

2000

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Requirement for small side chain residues within the GxGD-motif of presenilin for γ-secretase substrate cleavage

Journal of neurochemistry, 2010-02, Vol.112 (4), p.940-950

2010

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Requirement for small side chain residues within the GxGD-motif of presenilin for g-secretase substrate cleavage

Journal of neurochemistry, 2010-02, Vol.112 (4), p.940-950

2010

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Requirement for small side chain residues within the GxGD-motif of presenilin for [gamma]-secretase substrate cleavage

Journal of neurochemistry, 2010-02, Vol.112 (4), p.940

2010

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P1‐157: γ‐Secretase substrates APP, Notch1 and CD44 show distinct processing in NPC cells

Alzheimer's & dementia, 2009-07, Vol.5 (4S_Part_7), p.P223-P224

2009

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Identification of a beta-secretase activity, which truncates amyloid beta-peptide after its presenilin-dependent generation

The Journal of biological chemistry, 2003-02, Vol.278 (8), p.5531-5538

2003

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A stable G-quadruplex within the ADAM10 5'-UTR is involved in translational repression of ADAM10

Alzheimer's & dementia, 2011-07, Vol.7 (4), p.S408-S409

2011

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TREM2 mutations implicated in neurodegeneration impair cell surface transport and phagocytosis

Casein Kinase 2 dependent phosphorylation of eIF4B regulates BACE1 expression in Alzheimer's disease

Low Cholesterol Stimulates the Nonamyloidogenic Pathway by Its Effect on the α-Secretase ADAM 10

Translational Repression of the Disintegrin and Metalloprotease ADAM10 by a Stable G-quadruplex Secondary Structure in Its 5′-Untranslated Region

Constitutive and Regulated α -secretase Cleavage of Alzheimer's Amyloid Precursor Protein by a Disintegrin Metalloprotease

Furin-, ADAM 10-, and γ-Secretase-Mediated Cleavage of a Receptor Tyrosine Phosphatase and Regulation of β-Catenin's Transcriptional Activity

Involvement of presenilin holoprotein upregulation in calcium dyshomeostasis of Alzheimer's disease

Expression of the Anti-amyloidogenic Secretase ADAM10 Is Suppressed by Its 5′-Untranslated Region

Expression of the Alzheimer protease BACE1 is suppressed via its 5'-untranslated region

hnRNP A3 binds to GGGGCC repeats and is a constituent of p62-positive/TDP43-negative inclusions in the hippocampus of patients with C9orf72 mutations

Postnatal disruption of the disintegrin/metalloproteinase ADAM10 in brain causes epileptic seizures, learning deficits, altered spine morphology, and defective synaptic functions

Function, regulation and therapeutic properties of β-secretase (BACE1)

Phosphorylation of the Translation Initiation Factor eIF2α Increases BACE1 Levels and Promotes Amyloidogenesis

Rescue of progranulin deficiency associated with frontotemporal lobar degeneration by alkalizing reagents and inhibition of vacuolar ATPase

Insulin-degrading Enzyme Rapidly Removes the β-Amyloid Precursor Protein Intracellular Domain (AICD)

Presenilin-dependent Intramembrane Proteolysis of CD44 Leads to the Liberation of Its Intracellular Domain and the Secretion of an Aβ-like Peptide

Phosphorylation Regulates Intracellular Trafficking of β-Secretase

Presenilin-1 Affects Trafficking and Processing of βAPP and Is Targeted in a Complex with Nicastrin to the Plasma Membrane

Maturation and Pro-peptide Cleavage of β-Secretase

Requirement for small side chain residues within the GxGD-motif of presenilin for γ-secretase substrate cleavage

Requirement for small side chain residues within the GxGD-motif of presenilin for g-secretase substrate cleavage

Requirement for small side chain residues within the GxGD-motif of presenilin for [gamma]-secretase substrate cleavage

P1‐157: γ‐Secretase substrates APP, Notch1 and CD44 show distinct processing in NPC cells

Identification of a beta-secretase activity, which truncates amyloid beta-peptide after its presenilin-dependent generation

A stable G-quadruplex within the ADAM10 5'-UTR is involved in translational repression of ADAM10

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