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A majority of HIV persistence during antiretroviral therapy is due to infected cell proliferation

Nature communications, 2018-11, Vol.9 (1), p.4811-16, Article 4811

2018

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Astrovirus encephalitis in boy with X-linked agammaglobulinemia

Emerging infectious diseases, 2010-06, Vol.16 (6), p.918-925

2010

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An Increasing Proportion of Monotypic HIV-1 DNA Sequences during Antiretroviral Treatment Suggests Proliferation of HIV-Infected Cells

Journal of Virology, 2013-02, Vol.87 (3), p.1770-1778

2013

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Quarter Century of Anti-HIV CAR T Cells

Current HIV/AIDS reports, 2018-04, Vol.15 (2), p.147-154

Wagner, Thor A.

2018

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Proliferation of cells with HIV integrated into cancer genes contributes to persistent infection

Science (American Association for the Advancement of Science), 2014-08, Vol.345 (6196), p.570-573

2014

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Engineering HIV-Resistant, Anti-HIV Chimeric Antigen Receptor T Cells

Molecular therapy, 2017-03, Vol.25 (3), p.570-579

2017

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Efficient modification of CCR5 in primary human hematopoietic cells using a megaTAL nuclease and AAV donor template

Science translational medicine, 2015-09, Vol.7 (307), p.307ra156

2015

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Combining Cell and Gene Therapy in an Effort to Eradicate HIV

AIDS patient care and STDs, 2016-12, Vol.30 (12), p.534

Wagner, Thor A

2016

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CNS Persistence of HIV-1 in Children: the Untapped Reservoir

Current HIV/AIDS reports, 2018-10, Vol.15 (5), p.382-387

2018

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Detection of HIV-1 Drug Resistance in Women Following Administration of a Single Dose of Nevirapine: Comparison of Plasma RNA to Cellular DNA by Consensus Sequencing and by Oligonucleotide Ligation Assay

Journal of Clinical Microbiology, 2010-05, Vol.48 (5), p.1555-1561

2010

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SARS‐CoV‐2 RNA positive pediatric organ donors: A case report

Pediatric transplantation, 2023-08, Vol.27 (5), p.e14452-n/a

2023

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Emerging biomarkers for the diagnosis of severe neonatal infections applicable to low resource settings

Journal of global health, 2011-12, Vol.1 (2), p.210-223

2011

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A global health opportunity: The potential of multiplexed diagnostics in low-resource settings

Journal of global health, 2011-12, Vol.1 (2), p.138-141

2011

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Epidemiology and aetiology of maternal bacterial and viral infections in low- and middle-income countries

Journal of global health, 2011-12, Vol.1 (2), p.171-188

2011

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Yet Another Reason to Treat HIV Infection

Clinical infectious diseases, 2012-04, Vol.54 (7), p.1010-1012

2012

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Antiretroviral treatment for HIV-infected infants exposed to nevirapine

JAMA : the journal of the American Medical Association, 2010-12, Vol.304 (23), p.2589; author reply 2589

2010

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H1N1 influenza vaccination in HIV-infected women on effective antiretroviral treatment did not induce measurable antigen-driven proliferation of the HIV-1 proviral reservoir

AIDS research and therapy, 2017-02, Vol.14 (1), p.7-7, Article 7

2017

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Epidemiology and aetiology of maternal parasitic infections in low- and middle-income countries

Journal of global health, 2011-12, Vol.1 (2), p.189-200

2011

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Potential limitation of CCR5 antagonists : drug resistance more often linked to CXCR4-utilizing than to CCR5-utilizing HIV-1

AIDS (London), 2008-11, Vol.22 (17), p.2393-2395

2008

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Increased mutations in Env and Pol suggest greater HIV-1 replication in sputum-derived viruses compared with blood-derived viruses

AIDS (London), 2009-05, Vol.23 (8), p.923-928

2009

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Quantification of mitochondrial toxicity in HIV‐infected individuals by quantitative PCR compared to flow cytometry

Cytometry. Part B, Clinical cytometry, 2013-01, Vol.84B (1), p.55-58

2013

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452. MegaTAL Disruption of CCR5 To Protect Anti-HIV CAR+ Lymphocytes from HIV Infection

Molecular therapy, 2015-05, Vol.23, p.S179-S180

2015

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Ribozyme Engineering and Early Evolution

Bioscience, 1998-02, Vol.48 (2), p.94-103

1998

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761. Targeted Killing of HIV Infected Cells Using CCR5-Disrupted Anti-HIV-CAR T Cells

Molecular therapy, 2016-05, Vol.24, p.S301-S301

2016

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Potential limitation of CCR5 antagonists: drug resistance more often linked to CXCR4-utilizing than to CCRS-utilizing HIV-1

AIDS (London), 2008-11, Vol.22 (17), p.2393-2395

2008

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A majority of HIV persistence during antiretroviral therapy is due to infected cell proliferation

Astrovirus encephalitis in boy with X-linked agammaglobulinemia

An Increasing Proportion of Monotypic HIV-1 DNA Sequences during Antiretroviral Treatment Suggests Proliferation of HIV-Infected Cells

Quarter Century of Anti-HIV CAR T Cells

Proliferation of cells with HIV integrated into cancer genes contributes to persistent infection

Engineering HIV-Resistant, Anti-HIV Chimeric Antigen Receptor T Cells

Efficient modification of CCR5 in primary human hematopoietic cells using a megaTAL nuclease and AAV donor template

Combining Cell and Gene Therapy in an Effort to Eradicate HIV

CNS Persistence of HIV-1 in Children: the Untapped Reservoir

Detection of HIV-1 Drug Resistance in Women Following Administration of a Single Dose of Nevirapine: Comparison of Plasma RNA to Cellular DNA by Consensus Sequencing and by Oligonucleotide Ligation Assay

SARS‐CoV‐2 RNA positive pediatric organ donors: A case report

Emerging biomarkers for the diagnosis of severe neonatal infections applicable to low resource settings

A global health opportunity: The potential of multiplexed diagnostics in low-resource settings

Epidemiology and aetiology of maternal bacterial and viral infections in low- and middle-income countries

Yet Another Reason to Treat HIV Infection

Antiretroviral treatment for HIV-infected infants exposed to nevirapine

H1N1 influenza vaccination in HIV-infected women on effective antiretroviral treatment did not induce measurable antigen-driven proliferation of the HIV-1 proviral reservoir

Epidemiology and aetiology of maternal parasitic infections in low- and middle-income countries

Potential limitation of CCR5 antagonists : drug resistance more often linked to CXCR4-utilizing than to CCR5-utilizing HIV-1

Increased mutations in Env and Pol suggest greater HIV-1 replication in sputum-derived viruses compared with blood-derived viruses

Quantification of mitochondrial toxicity in HIV‐infected individuals by quantitative PCR compared to flow cytometry

452. MegaTAL Disruption of CCR5 To Protect Anti-HIV CAR+ Lymphocytes from HIV Infection

Ribozyme Engineering and Early Evolution

761. Targeted Killing of HIV Infected Cells Using CCR5-Disrupted Anti-HIV-CAR T Cells

Potential limitation of CCR5 antagonists: drug resistance more often linked to CXCR4-utilizing than to CCRS-utilizing HIV-1

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