UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Einfacher Volltextzugriff von (fast) überall

Installieren Sie die LibKey Nomad-Browsererweiterung und erhalten Sie von vielen Webseiten einfachen Zugriff auf lizenzierte Volltexte oder Open Access Publikationen. Mehr erfahren...

Recurrent gain of function mutation in calcium channel CACNA1H causes early-onset hypertension with primary aldosteronism

eLife, 2015-04, Vol.4, p.e06315-e06315

2015

Volltextzugriff (PDF)

Determination of oligomeric states of proteins via dual-color colocalization with single molecule localization microscopy

eLife, 2022-10, Vol.11

2022

Volltextzugriff (PDF)

Elevated aldosterone and blood pressure in a mouse model of familial hyperaldosteronism with ClC-2 mutation

Nature communications, 2019-11, Vol.10 (1), p.5155-14, Article 5155

2019

Volltextzugriff (PDF)

Enhanced Ca2+ signaling, mild primary aldosteronism, and hypertension in a familial hyperaldosteronism mouse model (Cacna1hM1560V/+)

Proceedings of the National Academy of Sciences - PNAS, 2021-04, Vol.118 (17), p.1

2021

Volltextzugriff (PDF)

Author Correction: Elevated aldosterone and blood pressure in a mouse model of familial hyperaldosteronism with ClC-2 mutation

Nature communications, 2022-05, Vol.13 (1), p.3066-3066, Article 3066

2022

Volltextzugriff (PDF)

Pathogenesis of Familial Hyperaldosteronism Type II: New Concepts Involving Anion Channels

Current hypertension reports, 2019-04, Vol.21 (4), p.31-10, Article 31

2019

Volltextzugriff (PDF)

Dual-color Colocalization in Single-molecule Localization Microscopy to Determine the Oligomeric State of Proteins in the Plasma Membrane

Bio-protocol, 2023-07, Vol.13 (13), p.e4749-e4749

2023

Volltextzugriff (PDF)

A K+-selective CNG channel orchestrates Ca2+ signalling in zebrafish sperm

eLife, 2015-12, Vol.4

2015

Volltextzugriff (PDF)

Reduced Membrane Insertion of CLC-K by V33L Barttin Results in Loss of Hearing, but Leaves Kidney Function Intact

Frontiers in physiology, 2017-05, Vol.8, p.269-269

2017

Volltextzugriff (PDF)

Barttin Regulates the Subcellular Localization and Posttranslational Modification of Human Cl - /H + Antiporter ClC-5

Frontiers in physiology, 2018-10, Vol.9, p.1490-1490

2018

Volltextzugriff (PDF)

Regulation of ClC-2 gating by intracellular ATP

Pflügers Archiv, 2013-10, Vol.465 (10), p.1423-1437

2013

Volltextzugriff (PDF)

Isradipine therapy in Cacna1dIle772Met/+ mice ameliorates primary aldosteronism and neurologic abnormalities

JCI insight, 2023-10, Vol.8 (20)

2023

Volltextzugriff (PDF)

Rapid Turnover of the Cardiac L-Type CaV1.2 Channel by Endocytic Recycling Regulates Its Cell Surface Availability

iScience, 2018-09, Vol.7, p.1-15

2018

Volltextzugriff (PDF)

CACNA1H Calcium Channel Mutations in Primary Aldosteronism – Is Sodium the Culprit?

Endocrinology (Philadelphia), 2020-12, Vol.161 (12), p.1

2020

Volltextzugriff (PDF)

Adrenal Anion Channels: New Roles in Zona Glomerulosa Physiology and in the Pathophysiology of Primary Aldosteronism

Handbook of experimental pharmacology, 2024, Vol.283, p.59

2024

Volltextzugriff (PDF)

Ca V 3.2 (CACNA1H) in Primary Aldosteronism

Handbook of experimental pharmacology, 2023, Vol.279, p.249

2023

Volltextzugriff (PDF)

T- and L-Type Calcium Channels Maintain Calcium Oscillations in the Murine Zona Glomerulosa

Hypertension (Dallas, Tex. 1979), 2024-04, Vol.81 (4), p.811-822

2024

Volltextzugriff (PDF)

CLCN2 chloride channel mutations in familial hyperaldosteronism type II

Nature genetics, 2018-03, Vol.50 (3), p.349-354

2018

Volltextzugriff (PDF)

Carboxyl-terminal Truncations of ClC-Kb Abolish Channel Activation by Barttin Via Modified Common Gating and Trafficking

The Journal of biological chemistry, 2015-12, Vol.290 (51), p.30406-30416

2015

Volltextzugriff (PDF)

ClC-1 and ClC-2 form hetero-dimeric channels with novel protopore functions

Pflügers Archiv, 2014-12, Vol.466 (12), p.2191-2204

2014

Volltextzugriff (PDF)

CLC channel function and dysfunction in health and disease

Frontiers in physiology, 2014-10, Vol.5, p.378-378

2014

Volltextzugriff (PDF)

A novel cross‐species inhibitor to study the function of CatSper Ca2+ channels in sperm

British journal of pharmacology, 2018-08, Vol.175 (15), p.3144-3161

2018

Volltextzugriff (PDF)

Direct Interaction of CaVβ with Actin Up-regulates L-type Calcium Currents in HL-1 Cardiomyocytes

The Journal of biological chemistry, 2015-02, Vol.290 (8), p.4561-4572

2015

Volltextzugriff (PDF)

CaV3.2 (CACNA1H) in Primary Aldosteronism

Voltage-gated Ca2+ Channels: Pharmacology, Modulation and their Role in Human Disease, p.249-262

Volltextzugriff (PDF)

1 – 25 von 44

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(41)
Mit Volltextzugriff
(36)

Publikationsform

Sprache

Englisch
(44)
Japanisch
(6)

Erscheinungsjahr

n.n

Thema

15 weitere anzeigen...

Weniger anzeigen...

Zeitschrift / Serie

5 weitere anzeigen...

Weniger anzeigen...

Neu hinzugefügt

... seit 30 Tagen
(1)

Menü

Weitere Dienste

Einstellungen

Somatic and germline CACNA1D calcium channel mutations in aldosterone-producing adenomas and primary aldosteronism

Recurrent gain of function mutation in calcium channel CACNA1H causes early-onset hypertension with primary aldosteronism

Determination of oligomeric states of proteins via dual-color colocalization with single molecule localization microscopy

Elevated aldosterone and blood pressure in a mouse model of familial hyperaldosteronism with ClC-2 mutation

Enhanced Ca2+ signaling, mild primary aldosteronism, and hypertension in a familial hyperaldosteronism mouse model (Cacna1hM1560V/+)

Author Correction: Elevated aldosterone and blood pressure in a mouse model of familial hyperaldosteronism with ClC-2 mutation

Pathogenesis of Familial Hyperaldosteronism Type II: New Concepts Involving Anion Channels

Dual-color Colocalization in Single-molecule Localization Microscopy to Determine the Oligomeric State of Proteins in the Plasma Membrane

A K+-selective CNG channel orchestrates Ca2+ signalling in zebrafish sperm

Reduced Membrane Insertion of CLC-K by V33L Barttin Results in Loss of Hearing, but Leaves Kidney Function Intact

Barttin Regulates the Subcellular Localization and Posttranslational Modification of Human Cl - /H + Antiporter ClC-5

Regulation of ClC-2 gating by intracellular ATP

Isradipine therapy in Cacna1dIle772Met/+ mice ameliorates primary aldosteronism and neurologic abnormalities

Rapid Turnover of the Cardiac L-Type CaV1.2 Channel by Endocytic Recycling Regulates Its Cell Surface Availability

CACNA1H Calcium Channel Mutations in Primary Aldosteronism – Is Sodium the Culprit?

Adrenal Anion Channels: New Roles in Zona Glomerulosa Physiology and in the Pathophysiology of Primary Aldosteronism

Ca V 3.2 (CACNA1H) in Primary Aldosteronism

T- and L-Type Calcium Channels Maintain Calcium Oscillations in the Murine Zona Glomerulosa

CLCN2 chloride channel mutations in familial hyperaldosteronism type II

Carboxyl-terminal Truncations of ClC-Kb Abolish Channel Activation by Barttin Via Modified Common Gating and Trafficking

ClC-1 and ClC-2 form hetero-dimeric channels with novel protopore functions

CLC channel function and dysfunction in health and disease

A novel cross‐species inhibitor to study the function of CatSper Ca2+ channels in sperm

Direct Interaction of CaVβ with Actin Up-regulates L-type Calcium Currents in HL-1 Cardiomyocytes

CaV3.2 (CACNA1H) in Primary Aldosteronism

Suchergebnisse filtern