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Structural changes of the ligand and of the receptor alters the receptor preference for neutrophil activating peptides starting with a formylmethionyl group

Biochimica et biophysica acta, 2015-01, Vol.1853 (1), p.192-200

2015

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N-Formyl Peptide Receptor 3 (FPR3) Departs from the Homologous FPR2/ALX Receptor with Regard to the Major Processes Governing Chemoattractant Receptor Regulation, Expression at the Cell Surface, and Phosphorylation

The Journal of biological chemistry, 2011-07, Vol.286 (30), p.26718-26731

2011

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Structural characterization and inhibitory profile of formyl peptide receptor 2 selective peptides descending from a PIP2-binding domain of gelsolin

The Journal of immunology (1950), 2012-07, Vol.189 (2), p.629-637

2012

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The N-formyl peptide receptors and the anaphylatoxin C5a receptors: An overview

Biochimie, 2007-09, Vol.89 (9), p.1089-1106

2007

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Human mitochondria‐derived N‐formylated peptides are novel agonists equally active on FPR and FPRL1, while Listeria monocytogenes‐derived peptides preferentially activate FPR

European journal of immunology, 2005-08, Vol.35 (8), p.2486-2495

2005

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Serum amyloid A mediates human neutrophil production of reactive oxygen species through a receptor independent of formyl peptide receptor like-1

Journal of leukocyte biology, 2008-02, Vol.83 (2), p.245-253

2008

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Injection of Pseudomonas aeruginosa Exo toxins into host cells can be modulated by host factors at the level of translocon assembly and/or activity

PloS one, 2012-01, Vol.7 (1), p.e30488

2012

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Identification of Novel Small-Molecule Agonists for Human Formyl Peptide Receptors and Pharmacophore Models of Their Recognition

Molecular pharmacology, 2010-02, Vol.77 (2), p.159-170

2010

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House dust mite allergen activates human eosinophils via formyl peptide receptor and formyl peptide receptor-like 1

European journal of immunology, 2007-07, Vol.37 (7), p.1966-1977

2007

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The Synthetic Peptide Trp-Lys-Tyr-Met-Val-Met-NH2 Specifically Activates Neutrophils through FPRL1/Lipoxin A4 Receptors and Is an Agonist for the Orphan Monocyte-expressed Chemoattractant Receptor FPRL2

The Journal of biological chemistry, 2001-06, Vol.276 (24), p.21585-21593

2001

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The role of β-arrestins in the formyl peptide receptor-like 1 internalization and signaling

Cellular signalling, 2007-09, Vol.19 (9), p.1939-1948

2007

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Complement Component 5a Receptor Oligomerization and Homologous Receptor Down-regulation

The Journal of biological chemistry, 2008-11, Vol.283 (45), p.31038-31046

2008

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The FPR2-specific ligand MMK-1 activates the neutrophil NADPH-oxidase, but triggers no unique pathway for opening of plasma membrane calcium channels

Cell calcium (Edinburgh), 2009-05, Vol.45 (5), p.431-438

2009

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Human formyl peptide receptor 2 senses highly pathogenic Staphylococcus aureus

Cell host & microbe, 2010-06, Vol.7 (6), p.463-473

2010

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The annexin I sequence gln(9)-ala(10)-trp(11)-phe(12) is a core structure for interaction with the formyl peptide receptor 1

The Journal of biological chemistry, 2010-05, Vol.285 (19), p.14338

2010

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The Annexin I Sequence Gln9-Ala10-Trp11-Phe12 Is a Core Structure for Interaction with the Formyl Peptide Receptor 1

The Journal of biological chemistry, 2010-05, Vol.285 (19), p.14338-14345

2010

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The Chemoattractant Receptors FPR and C5aR: Same Functions – Different Fates

Traffic (Copenhagen, Denmark), 2005-02, Vol.6 (2), p.83-86

2005

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Formyl peptide receptor-mediated proinflammatory consequences of peptide deformylase inhibition in Staphylococcus aureus

Microbes and infection, 2010-05, Vol.12 (5), p.415-419

2010

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6-Methyl-2,4-Disubstituted Pyridazin-3(2H)-ones: A Novel Class of Small-Molecule Agonists for Formyl Peptide Receptors

Journal of medicinal chemistry, 2009-08, Vol.52 (16), p.5044-5057

2009

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Thrombin-induced increase in endothelial permeability is associated with changes in cell-to-cell junction organization

Arteriosclerosis, thrombosis, and vascular biology, 1996-03, Vol.16 (3), p.488

1996

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The leukocyte chemotactic receptor FPR2, but not the closely related FPR1, is sensitive to cell-penetrating pepducins with amino acid sequences descending from the third intracellular receptor loop

Biochimica et biophysica acta, 2013-08, Vol.1833 (8), p.1914-1923

2013

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Human mitochondria-derivedN-formylated peptides are novel agonists equally active on FPR and FPRL1, whileListeria monocytogenes-derived peptides preferentially activate FPR

European journal of immunology, 2005-08, Vol.35 (8), p.2486-2495

2005

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Direct binding of a fragment of the Wiskott-Aldrich syndrome protein to the C-terminal end of the anaphylatoxin C5a receptor

Biochemical journal, 2003-06, Vol.372 (Pt 2), p.453-463

2003

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Overexpression of Wild-Type and Catalytically Inactive Forms of GRK2 and GRK6 Fails to Alter the Agonist-Induced Phosphorylation of the C5a Receptor (CD88): Evidence That GRK6 Is Autophosphorylated in COS-7 Cells

Biochemical and biophysical research communications, 1999-05, Vol.259 (1), p.224-229

1999

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Isolation and Characterization of a Variant HL60 Cell Line Defective in the Activation of the NADPH Oxidase by Phorbol Myristate Acetate

The Journal of immunology (1950), 1998-12, Vol.161 (12), p.6885-6895

1998

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Structural changes of the ligand and of the receptor alters the receptor preference for neutrophil activating peptides starting with a formylmethionyl group

N-Formyl Peptide Receptor 3 (FPR3) Departs from the Homologous FPR2/ALX Receptor with Regard to the Major Processes Governing Chemoattractant Receptor Regulation, Expression at the Cell Surface, and Phosphorylation

Structural characterization and inhibitory profile of formyl peptide receptor 2 selective peptides descending from a PIP2-binding domain of gelsolin

The N-formyl peptide receptors and the anaphylatoxin C5a receptors: An overview

Human mitochondria‐derived N‐formylated peptides are novel agonists equally active on FPR and FPRL1, while Listeria monocytogenes‐derived peptides preferentially activate FPR

Serum amyloid A mediates human neutrophil production of reactive oxygen species through a receptor independent of formyl peptide receptor like-1

Injection of Pseudomonas aeruginosa Exo toxins into host cells can be modulated by host factors at the level of translocon assembly and/or activity

Identification of Novel Small-Molecule Agonists for Human Formyl Peptide Receptors and Pharmacophore Models of Their Recognition

House dust mite allergen activates human eosinophils via formyl peptide receptor and formyl peptide receptor-like 1

The Synthetic Peptide Trp-Lys-Tyr-Met-Val-Met-NH2 Specifically Activates Neutrophils through FPRL1/Lipoxin A4 Receptors and Is an Agonist for the Orphan Monocyte-expressed Chemoattractant Receptor FPRL2

The role of β-arrestins in the formyl peptide receptor-like 1 internalization and signaling

Complement Component 5a Receptor Oligomerization and Homologous Receptor Down-regulation

The FPR2-specific ligand MMK-1 activates the neutrophil NADPH-oxidase, but triggers no unique pathway for opening of plasma membrane calcium channels

Human formyl peptide receptor 2 senses highly pathogenic Staphylococcus aureus

The annexin I sequence gln(9)-ala(10)-trp(11)-phe(12) is a core structure for interaction with the formyl peptide receptor 1

The Annexin I Sequence Gln9-Ala10-Trp11-Phe12 Is a Core Structure for Interaction with the Formyl Peptide Receptor 1

The Chemoattractant Receptors FPR and C5aR: Same Functions – Different Fates

Formyl peptide receptor-mediated proinflammatory consequences of peptide deformylase inhibition in Staphylococcus aureus

6-Methyl-2,4-Disubstituted Pyridazin-3(2H)-ones: A Novel Class of Small-Molecule Agonists for Formyl Peptide Receptors

Thrombin-induced increase in endothelial permeability is associated with changes in cell-to-cell junction organization

The leukocyte chemotactic receptor FPR2, but not the closely related FPR1, is sensitive to cell-penetrating pepducins with amino acid sequences descending from the third intracellular receptor loop

Human mitochondria-derivedN-formylated peptides are novel agonists equally active on FPR and FPRL1, whileListeria monocytogenes-derived peptides preferentially activate FPR

Direct binding of a fragment of the Wiskott-Aldrich syndrome protein to the C-terminal end of the anaphylatoxin C5a receptor

Overexpression of Wild-Type and Catalytically Inactive Forms of GRK2 and GRK6 Fails to Alter the Agonist-Induced Phosphorylation of the C5a Receptor (CD88): Evidence That GRK6 Is Autophosphorylated in COS-7 Cells

Isolation and Characterization of a Variant HL60 Cell Line Defective in the Activation of the NADPH Oxidase by Phorbol Myristate Acetate

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