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The Validation Process in the IELTS Reading Component: Reading Requirements for Preparing International Students

Journal of lanugage and education, 2018-03, Vol.4 (1), p.63-78

Kovalenko, Marina

2018

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CAG repeat expansion in the Huntington’s disease gene shapes linear and circular RNAs biogenesis

PLoS genetics, 2023-10, Vol.19 (10), p.e1010988-e1010988

2023

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Splice modulators target PMS1 to reduce somatic expansion of the Huntington's disease-associated CAG repeat

Nature communications, 2024-04, Vol.15 (1), p.3182-3182

2024

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Msh2 acts in medium-spiny striatal neurons as an enhancer of CAG instability and mutant huntingtin phenotypes in Huntington's disease knock-in mice

PloS one, 2012-09, Vol.7 (9), p.e44273

2012

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Histone deacetylase knockouts modify transcription, CAG instability and nuclear pathology in Huntington disease mice

eLife, 2020-09, Vol.9

2020

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Size structures and comparative phenology of syntopic populations of Hirudo verbana and Hirudo medicinalis in eastern Ukraine

Biológia, 2012-10, Vol.67 (5), p.934-938

2012

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The Global Education Programme in the Era of the Knowledge Based Economy: Context and Implications

Mir Rossii, 2017-06, Vol.26 (3), p.117-141

Kovalenko, Marina

2017

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Somatic CAG expansion in Huntington's disease is dependent on the MLH3 endonuclease domain, which can be excluded via splice redirection

Nucleic acids research, 2021-04, Vol.49 (7), p.3907

2021

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Problematically Conditioned Subject Content of Discursive Speech Materials in the Integrative Approach to Teaching Interpreters Listening Comprehension

Yazyk i kul'tura, 2020-12 (52), p.232-245

2020

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B1 HTT CAG knock-in mice with pure and interrupted repeat tracts provide insight into the role of somatic expansion in HD pathogenesis

Journal of neurology, neurosurgery and psychiatry, 2016-09, Vol.87 (Suppl 1), p.A9-A9

2016

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HttQ111/+ Huntington's Disease Knock-in Mice Exhibit Brain Region-Specific Morphological Changes and Synaptic Dysfunction

Journal of Huntington's disease, 2018-01, Vol.7 (1), p.17

2018

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Selecting features from multiple feature sets for SVM committee-based screening of human larynx

Expert systems with applications, 2010, Vol.37 (10), p.6957-6962

2010

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Chromosome substitution strain assessment of a Huntington’s disease modifier locus

Mammalian genome, 2015-04, Vol.26 (3-4), p.119-130

2015

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Insulin- and Glucose-Induced Phosphorylation of the Na+,K+-Adenosine Triphosphatase α-Subunits in Rat Skeletal Muscle

Endocrinology (Philadelphia), 2001-08, Vol.142 (8), p.3474-3482

2001

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Site-selective Dephosphorylation of the Platelet-derived Growth Factor β-Receptor by the Receptor-like Protein-tyrosine Phosphatase DEP-1

The Journal of biological chemistry, 2000-05, Vol.275 (21), p.16219-16226

2000

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Selective platelet-derived growth factor receptor kinase blockers reverse sis-transformation

Cancer research (Chicago, Ill.), 1994-12, Vol.54 (23), p.6106-6114

1994

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The protein-tyrosine phosphatase DEP-1 modulates growth factor-stimulated cell migration and cell-matrix adhesion

Oncogene, 2003-07, Vol.22 (27), p.4175-4185

2003

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Phosphorylation Site-Specific Inhibition of Platelet-Derived Growth Factor β-Receptor Autophosphorylation by the Receptor Blocking Tyrphostin AG1296

Biochemistry (Easton), 1997-05, Vol.36 (21), p.6260-6269

1997

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A Broad Phenotypic Screen Identifies Novel Phenotypes Driven by a Single Mutant Allele in Huntingtons Disease CAG Knock-In Mice: e80923

PloS one, 2013-11, Vol.8 (11)

2013

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Platelet-Derived Growth Factor Receptor Family

Reference Module in Life Sciences, 2017

2017

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Integrative Teaching of Foreign-Language Listening and Speaking Skills to Engineering Students

Integrating Engineering Education and Humanities for Global Intercultural Perspectives, p.387-395

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Site-selective Dephosphorylation of the Platelet-derived Growth Factor Î²-Receptor by the Receptor-like Protein-tyrosine Phosphatase DEP-1

The Journal of biological chemistry, 2000-05, Vol.275 (21), p.16219

2000

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Tyrosine Phosphorylation of Gsα and Inhibition of Bradykinin-induced Activation of the Cyclic AMP Pathway in A431 Cells by Epidermal Growth Factor Receptor

The Journal of biological chemistry, 1996-12, Vol.271 (49), p.31098-31105

1996

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The Validation Process in the IELTS Reading Component: Reading Requirements for Preparing International Students

CAG repeat expansion in the Huntington’s disease gene shapes linear and circular RNAs biogenesis

Splice modulators target PMS1 to reduce somatic expansion of the Huntington's disease-associated CAG repeat

Msh2 acts in medium-spiny striatal neurons as an enhancer of CAG instability and mutant huntingtin phenotypes in Huntington's disease knock-in mice

Histone deacetylase knockouts modify transcription, CAG instability and nuclear pathology in Huntington disease mice

A broad phenotypic screen identifies novel phenotypes driven by a single mutant allele in Huntington's disease CAG knock-in mice

Size structures and comparative phenology of syntopic populations of Hirudo verbana and Hirudo medicinalis in eastern Ukraine

The Global Education Programme in the Era of the Knowledge Based Economy: Context and Implications

Somatic CAG expansion in Huntington's disease is dependent on the MLH3 endonuclease domain, which can be excluded via splice redirection

Problematically Conditioned Subject Content of Discursive Speech Materials in the Integrative Approach to Teaching Interpreters Listening Comprehension

High resolution time-course mapping of early transcriptomic, molecular and cellular phenotypes in Huntington's disease CAG knock-in mice across multiple genetic backgrounds

B1 HTT CAG knock-in mice with pure and interrupted repeat tracts provide insight into the role of somatic expansion in HD pathogenesis

HttQ111/+ Huntington's Disease Knock-in Mice Exhibit Brain Region-Specific Morphological Changes and Synaptic Dysfunction

Selecting features from multiple feature sets for SVM committee-based screening of human larynx

Chromosome substitution strain assessment of a Huntington’s disease modifier locus

Insulin- and Glucose-Induced Phosphorylation of the Na+,K+-Adenosine Triphosphatase α-Subunits in Rat Skeletal Muscle

Site-selective Dephosphorylation of the Platelet-derived Growth Factor β-Receptor by the Receptor-like Protein-tyrosine Phosphatase DEP-1

Selective platelet-derived growth factor receptor kinase blockers reverse sis-transformation

The protein-tyrosine phosphatase DEP-1 modulates growth factor-stimulated cell migration and cell-matrix adhesion

Phosphorylation Site-Specific Inhibition of Platelet-Derived Growth Factor β-Receptor Autophosphorylation by the Receptor Blocking Tyrphostin AG1296

A Broad Phenotypic Screen Identifies Novel Phenotypes Driven by a Single Mutant Allele in Huntingtons Disease CAG Knock-In Mice: e80923

Platelet-Derived Growth Factor Receptor Family

Integrative Teaching of Foreign-Language Listening and Speaking Skills to Engineering Students

Site-selective Dephosphorylation of the Platelet-derived Growth Factor Î²-Receptor by the Receptor-like Protein-tyrosine Phosphatase DEP-1

Tyrosine Phosphorylation of Gsα and Inhibition of Bradykinin-induced Activation of the Cyclic AMP Pathway in A431 Cells by Epidermal Growth Factor Receptor

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