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Hypertension during Weight Lifting Reduces Flow-Mediated Dilation in Nonathletes

Medicine and science in sports and exercise, 2017-04, Vol.49 (4), p.669-675

2017

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The Human Microcirculation: Regulation of Flow and Beyond

Circulation research, 2016-01, Vol.118 (1), p.157-172

2016

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Role of PGC-1α in Vascular Regulation: Implications for Atherosclerosis

Arteriosclerosis, thrombosis, and vascular biology, 2016-08, Vol.36 (8), p.1467

2016

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Telomerase reverse transcriptase protects against angiotensin II-induced microvascular endothelial dysfunction

American journal of physiology. Heart and circulatory physiology, 2018-05, Vol.314 (5), p.H1053-H1060

2018

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Physiological Consequences of Coronary Arteriolar Dysfunction and Its Influence on Cardiovascular Disease

Physiology (Bethesda, Md.), 2018-09, Vol.33 (5), p.338-347

2018

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The Yin and Yang of endothelium-derived vasodilator factors

American journal of physiology. Heart and circulatory physiology, 2018-05, Vol.314 (5), p.H892-H894

2018

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Microvascular Adaptations to Exercise: Protective Effect of PGC-1 Alpha

American journal of hypertension, 2018-01, Vol.31 (2), p.240-246

2018

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Mitochondrial signaling in the vascular endothelium: beyond reactive oxygen species

Basic Research in Cardiology, 2016-05, Vol.111 (3), p.26-26, Article 26

2016

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PGC-1α (Peroxisome Proliferator–Activated Receptor γ Coactivator 1-α) Overexpression in Coronary Artery Disease Recruits NO and Hydrogen Peroxide During Flow-Mediated Dilation and Protects Against Increased Intraluminal Pressure

Hypertension (Dallas, Tex. 1979), 2017-07, Vol.70 (1), p.166-173

2017

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Friend or foe? Telomerase as a pharmacological target in cancer and cardiovascular disease

Pharmacological research, 2016-09, Vol.111, p.422-433

2016

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Lysophosphatidic acid acts on LPA1 receptor to increase H2O2 during flow‐induced dilation in human adipose arterioles

British journal of pharmacology, 2018-11, Vol.175 (22), p.4266-4280

2018

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The Yin and Yang of endothelium-derived vasodilator factors

American journal of physiology. Regulatory, integrative and comparative physiology, 2018-05, Vol.314 (5), p.H892

2018

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The Yin and Yang of endothelium-derived vasodilator factors

American journal of physiology. Lung cellular and molecular physiology, 2018-05, Vol.314 (5), p.H892

2018

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Telomerase reverse transcriptase protects against angiotensin II-induced microvascular endothelial dysfunction

American journal of physiology. Regulatory, integrative and comparative physiology, 2018-05, Vol.314 (5), p.H1053

2018

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Telomerase reverse transcriptase protects against angiotensin II-induced microvascular endothelial dysfunction

American journal of physiology. Lung cellular and molecular physiology, 2018-05, Vol.314 (5), p.H1053

2018

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LPA‐induced activation of LPA1 receptor leads to the loss of NO‐mediated flow‐induced dilation in human microvessels

The FASEB journal, 2018-04, Vol.32, p.713.15-713.15

2018

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LPA‐induced activation of LPA 1 receptor leads to the loss of NO‐mediated flow‐induced dilation in human microvessels

The FASEB journal, 2018-04, Vol.32 (S1)

2018

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Lysophosphatidic acid acts on LPA 1 receptor to increase H 2 O 2 during flow-induced dilation in human adipose arterioles

British journal of pharmacology, 2018-11, Vol.175 (22), p.4266-4280

2018

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Abstract 247: Mitochondrial Damage Associated Molecular Patterns Promotes Endothelial Dysfunction in the Microcirculation

Circulation research, 2018-08, Vol.123 (Suppl_1)

2018

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Dysbacteriosis an Inciting Cause of Endothelial Dysfunction mediated through Mitochondrial DNA Interactions

The FASEB journal, 2018-04, Vol.32 (S1), p.582.3-582.3

2018

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Lysophosphatidic Acid Promotes a Switch from Nitric Oxide to Hydrogen Peroxide as the Mediator of Flow‐Induced Dilation in Human Arterioles

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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PGC-1alpha Overexpression in Coronary Artery Disease Recruits Nitric Oxide and Hydrogen Peroxide during Flow-mediated Dilation and Protects Against Increased Intraluminal Pressure

Hypertension (Dallas, Tex. 1979), 2017-05, Vol.70 (1), p.166-173

2017

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Anatomical Considerations and Emerging Strategies for Reducing New Onset Conduction Disturbances in Percutaneous Structural Heart Disease Interventions

Structural heart (Online), 2021-07, Vol.5 (4), p.348-356

2021

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Hypertension during Weight Lifting Reduces Flow-Mediated Dilation in Nonathletes

The Human Microcirculation: Regulation of Flow and Beyond

Role of PGC-1α in Vascular Regulation: Implications for Atherosclerosis

Telomerase reverse transcriptase protects against angiotensin II-induced microvascular endothelial dysfunction

Physiological Consequences of Coronary Arteriolar Dysfunction and Its Influence on Cardiovascular Disease

The Yin and Yang of endothelium-derived vasodilator factors

Microvascular Adaptations to Exercise: Protective Effect of PGC-1 Alpha

Mitochondrial signaling in the vascular endothelium: beyond reactive oxygen species

PGC-1α (Peroxisome Proliferator–Activated Receptor γ Coactivator 1-α) Overexpression in Coronary Artery Disease Recruits NO and Hydrogen Peroxide During Flow-Mediated Dilation and Protects Against Increased Intraluminal Pressure

Friend or foe? Telomerase as a pharmacological target in cancer and cardiovascular disease

Lysophosphatidic acid acts on LPA1 receptor to increase H2O2 during flow‐induced dilation in human adipose arterioles

The Yin and Yang of endothelium-derived vasodilator factors

The Yin and Yang of endothelium-derived vasodilator factors

Telomerase reverse transcriptase protects against angiotensin II-induced microvascular endothelial dysfunction

Telomerase reverse transcriptase protects against angiotensin II-induced microvascular endothelial dysfunction

LPA‐induced activation of LPA1 receptor leads to the loss of NO‐mediated flow‐induced dilation in human microvessels

LPA‐induced activation of LPA 1 receptor leads to the loss of NO‐mediated flow‐induced dilation in human microvessels

Lysophosphatidic acid acts on LPA 1 receptor to increase H 2 O 2 during flow-induced dilation in human adipose arterioles

Abstract 247: Mitochondrial Damage Associated Molecular Patterns Promotes Endothelial Dysfunction in the Microcirculation

Dysbacteriosis an Inciting Cause of Endothelial Dysfunction mediated through Mitochondrial DNA Interactions

Lysophosphatidic Acid Promotes a Switch from Nitric Oxide to Hydrogen Peroxide as the Mediator of Flow‐Induced Dilation in Human Arterioles

PGC-1alpha Overexpression in Coronary Artery Disease Recruits Nitric Oxide and Hydrogen Peroxide during Flow-mediated Dilation and Protects Against Increased Intraluminal Pressure

Anatomical Considerations and Emerging Strategies for Reducing New Onset Conduction Disturbances in Percutaneous Structural Heart Disease Interventions

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