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Ceramide Changes the Mediator of Flow-Induced Vasodilation From Nitric Oxide to Hydrogen Peroxide in the Human Microcirculation

Circulation research, 2014-08, Vol.115 (5), p.525-532

2014

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Lipid phosphate phosphatase 3 maintains NO‐mediated flow‐mediated dilatation in human adipose resistance arterioles

The Journal of physiology, 2023-02, Vol.601 (3), p.469-481

2023

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Critical Role for Telomerase in the Mechanism of Flow-Mediated Dilation in the Human Microcirculation

Circulation research, 2016-03, Vol.118 (5), p.856-866

2016

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PGC-1α (Peroxisome Proliferator–Activated Receptor γ Coactivator 1-α) Overexpression in Coronary Artery Disease Recruits NO and Hydrogen Peroxide During Flow-Mediated Dilation and Protects Against Increased Intraluminal Pressure

Hypertension (Dallas, Tex. 1979), 2017-07, Vol.70 (1), p.166-173

2017

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Blockade of endothelial Mas receptor restores the vasomotor response to phenylephrine in human resistance arterioles pretreated with captopril and exposed to propofol

BMC anesthesiology, 2022-07, Vol.22 (1), p.1-240, Article 240

2022

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Manipulation of the Sphingolipid Rheostat Influences the Mediator of Flow-Induced Dilation in the Human Microvasculature

Journal of the American Heart Association, 2019-09, Vol.8 (17), p.e013153

2019

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Lysophosphatidic acid acts on LPA1 receptor to increase H2O2 during flow‐induced dilation in human adipose arterioles

British journal of pharmacology, 2018-11, Vol.175 (22), p.4266-4280

2018

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Telomerase reverse transcriptase protects against angiotensin II-induced microvascular endothelial dysfunction

American journal of physiology. Heart and circulatory physiology, 2018-05, Vol.314 (5), p.H1053-H1060

2018

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Critical Interaction Between Telomerase and Autophagy in Mediating Flow-Induced Human Arteriolar Vasodilation

Arteriosclerosis, thrombosis, and vascular biology, 2021-01, Vol.41 (1), p.446

2021

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Telomerase reverse transcriptase protects against angiotensin II-induced microvascular endothelial dysfunction

American journal of physiology. Regulatory, integrative and comparative physiology, 2018-05, Vol.314 (5), p.H1053

2018

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An acute rise in intraluminal pressure shifts the mediator of flow-mediated dilation from nitric oxide to hydrogen peroxide in human arterioles

American journal of physiology. Heart and circulatory physiology, 2014-12, Vol.307 (11), p.H1587-H1593

2014

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Telomerase reverse transcriptase protects against angiotensin II-induced microvascular endothelial dysfunction

American journal of physiology. Lung cellular and molecular physiology, 2018-05, Vol.314 (5), p.H1053

2018

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Abstract P121: Vascular Protective Effects of Telomerase Activity Are Independent of Nuclear Function

Hypertension (Dallas, Tex. 1979), 2015-09, Vol.66 (suppl_1)

2015

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Decreased Telomerase Activity Sensitizes the Resistance Vasculature to Stress‐Induced Endothelial Dysfunction

The FASEB journal, 2013-04, Vol.27 (S1)

2013

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Neoadjuvant Chemotherapy Decreases Angiogenesis Potential and Microvascular Function in Human Breast Cancer Patients

The FASEB journal, 2018-04, Vol.32 (S1), p.845.6-845.6

2018

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LPA‐induced activation of LPA 1 receptor leads to the loss of NO‐mediated flow‐induced dilation in human microvessels

The FASEB journal, 2018-04, Vol.32 (S1)

2018

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LPA‐induced activation of LPA1 receptor leads to the loss of NO‐mediated flow‐induced dilation in human microvessels

The FASEB journal, 2018-04, Vol.32, p.713.15-713.15

2018

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ISH ADA-01 Critical role of telomerase in regulating cerebral vascular function and redox environment

Journal of hypertension, 2016-09, Vol.34 Suppl 1 - ISH 2016 Abstract Book (Supplement 1), p.e39-e39

2016

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Inhibition of the vascular renin‐angiotensin system preserves nitric oxide‐mediated vasodilation in human adipose arterioles after transient high pressure stress (676.9)

The FASEB journal, 2014-04, Vol.28 (S1), p.n/a

2014

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Lysophosphatidic acid acts on LPA 1 receptor to increase H 2 O 2 during flow-induced dilation in human adipose arterioles

British journal of pharmacology, 2018-11, Vol.175 (22), p.4266-4280

2018

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Abstract 247: Mitochondrial Damage Associated Molecular Patterns Promotes Endothelial Dysfunction in the Microcirculation

Circulation research, 2018-08, Vol.123 (Suppl_1)

2018

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Critical role of telomerase in regulating cerebral vascular function and redox environment

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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PGC‐1α Activation Confers Plasticity in the Mediator of Dilation in Healthy and Diseased Human Arterioles

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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Dysbacteriosis an Inciting Cause of Endothelial Dysfunction mediated through Mitochondrial DNA Interactions

The FASEB journal, 2018-04, Vol.32 (S1), p.582.3-582.3

2018

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5,6-δ-DHTL, a stable metabolite of arachidonic acid, is a potential EDHF that mediates microvascular dilation

Free radical biology & medicine, 2017-02, Vol.103, p.87-94

2017

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Ceramide Changes the Mediator of Flow-Induced Vasodilation From Nitric Oxide to Hydrogen Peroxide in the Human Microcirculation

Lipid phosphate phosphatase 3 maintains NO‐mediated flow‐mediated dilatation in human adipose resistance arterioles

Critical Role for Telomerase in the Mechanism of Flow-Mediated Dilation in the Human Microcirculation

PGC-1α (Peroxisome Proliferator–Activated Receptor γ Coactivator 1-α) Overexpression in Coronary Artery Disease Recruits NO and Hydrogen Peroxide During Flow-Mediated Dilation and Protects Against Increased Intraluminal Pressure

Blockade of endothelial Mas receptor restores the vasomotor response to phenylephrine in human resistance arterioles pretreated with captopril and exposed to propofol

Manipulation of the Sphingolipid Rheostat Influences the Mediator of Flow-Induced Dilation in the Human Microvasculature

Lysophosphatidic acid acts on LPA1 receptor to increase H2O2 during flow‐induced dilation in human adipose arterioles

Telomerase reverse transcriptase protects against angiotensin II-induced microvascular endothelial dysfunction

Critical Interaction Between Telomerase and Autophagy in Mediating Flow-Induced Human Arteriolar Vasodilation

Telomerase reverse transcriptase protects against angiotensin II-induced microvascular endothelial dysfunction

An acute rise in intraluminal pressure shifts the mediator of flow-mediated dilation from nitric oxide to hydrogen peroxide in human arterioles

Telomerase reverse transcriptase protects against angiotensin II-induced microvascular endothelial dysfunction

Abstract P121: Vascular Protective Effects of Telomerase Activity Are Independent of Nuclear Function

Decreased Telomerase Activity Sensitizes the Resistance Vasculature to Stress‐Induced Endothelial Dysfunction

Neoadjuvant Chemotherapy Decreases Angiogenesis Potential and Microvascular Function in Human Breast Cancer Patients

LPA‐induced activation of LPA 1 receptor leads to the loss of NO‐mediated flow‐induced dilation in human microvessels

LPA‐induced activation of LPA1 receptor leads to the loss of NO‐mediated flow‐induced dilation in human microvessels

ISH ADA-01 Critical role of telomerase in regulating cerebral vascular function and redox environment

Inhibition of the vascular renin‐angiotensin system preserves nitric oxide‐mediated vasodilation in human adipose arterioles after transient high pressure stress (676.9)

Lysophosphatidic acid acts on LPA 1 receptor to increase H 2 O 2 during flow-induced dilation in human adipose arterioles

Abstract 247: Mitochondrial Damage Associated Molecular Patterns Promotes Endothelial Dysfunction in the Microcirculation

Critical role of telomerase in regulating cerebral vascular function and redox environment

PGC‐1α Activation Confers Plasticity in the Mediator of Dilation in Healthy and Diseased Human Arterioles

Dysbacteriosis an Inciting Cause of Endothelial Dysfunction mediated through Mitochondrial DNA Interactions

5,6-δ-DHTL, a stable metabolite of arachidonic acid, is a potential EDHF that mediates microvascular dilation

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