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Microglial Inflammatory Signaling Orchestrates the Hypothalamic Immune Response to Dietary Excess and Mediates Obesity Susceptibility

Cell metabolism, 2017-07, Vol.26 (1), p.185-197.e3

2017

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Sex differences in microglial CX3CR1 signalling determine obesity susceptibility in mice

Nature communications, 2017-02, Vol.8 (1), p.14556-14556, Article 14556

2017

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Hypothalamic Inflammation: Marker or Mechanism of Obesity Pathogenesis?

Diabetes (New York, N.Y.), 2013-08, Vol.62 (8), p.2629-2634

2013

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Central injection of fibroblast growth factor 1 induces sustained remission of diabetic hyperglycemia in rodents

Nature medicine, 2016-07, Vol.22 (7), p.800-806

2016

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Functional identification of a neurocircuit regulating blood glucose

Proceedings of the National Academy of Sciences - PNAS, 2016-04, Vol.113 (14), p.E2073-E2082

2016

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Kisspeptin Receptor Haplo-insufficiency Causes Premature Ovarian Failure Despite Preserved Gonadotropin Secretion

Endocrinology (Philadelphia), 2014-08, Vol.155 (8), p.3088-3097

2014

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CX3CL1 Action on Microglia Protects from Diet-Induced Obesity by Restoring POMC Neuronal Excitability and Melanocortin System Activity Impaired by High-Fat Diet Feeding

International journal of molecular sciences, 2022-06, Vol.23 (12), p.6380

2022

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Microglial Inflammatory Signaling Orchestrates the Hypothalamic Immune Response to Dietary Excess and Mediates Obesity Susceptibility

Cell metabolism, 2018-06, Vol.27 (6), p.1356-1356

2018

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Loss of Ntrk2/Kiss1r Signaling in Oocytes Causes Premature Ovarian Failure

Endocrinology (Philadelphia), 2014-08, Vol.155 (8), p.3098-3111

2014

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Glia: silent partners in energy homeostasis and obesity pathogenesis

Diabetologia, 2017-02, Vol.60 (2), p.226-236

2017

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Hypothalamic inflammation and gliosis in obesity

Current opinion in endocrinology, diabetes, and obesity, 2015-10, Vol.22 (5), p.325-330

2015

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Prostaglandin PGE2 Receptor EP4 Regulates Microglial Phagocytosis and Increases Susceptibility to Diet-Induced Obesity

Diabetes (New York, N.Y.), 2023-02, Vol.72 (2), p.233-244

2023

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Obesity-associated microglial inflammatory activation paradoxically improves glucose tolerance

Cell metabolism, 2023-09, Vol.35 (9), p.1613-1629.e8

2023

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1759-P: Prostaglandin PGE2 Receptor EP4 Signaling Deficiency in Microglia Reduces Weight Gain with Transient Glucose Improvements in Diet-Induced Obesity

Diabetes (New York, N.Y.), 2020-06, Vol.69 (Supplement_1)

2020

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Central androgen action reverses hypothalamic astrogliosis and atherogenic risk factors induced by orchiectomy and high-fat diet feeding in male mice

American journal of physiology: endocrinology and metabolism, 2023-05, Vol.324 (5), p.E461-E475

2023

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152-OR: Evidence Linking Loss of Hypothalamic Perineuronal Nets to Obesity Pathogenesis

Diabetes (New York, N.Y.), 2022-06, Vol.71 (Supplement_1)

2022

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1769-P: Microglial Activation and Inactivation via Designer Receptors Exclusively Activated by Designer Drugs (DREADDs) Alters Peripheral Glucose Homeostasis

Diabetes (New York, N.Y.), 2020-06, Vol.69 (Supplement_1)

2020

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212-OR: Testosterone Deficiency Increases Hypothalamic Gliosis and Metabolic Risk

Diabetes (New York, N.Y.), 2020-06, Vol.69 (Supplement_1)

2020

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Deletion of Protein Kinase C λ in POMC Neurons Predisposes to Diet-Induced Obesity

Diabetes (New York, N.Y.), 2017-04, Vol.66 (4), p.920-934

2017

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Evidence That in Uncontrolled Diabetes, Hyperglucagonemia Is Required for Ketosis but Not for Increased Hepatic Glucose Production or Hyperglycemia

Diabetes (New York, N.Y.), 2015-07, Vol.64 (7), p.2376-2387

2015

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Leptin Action in the Ventromedial Hypothalamic Nucleus Is Sufficient, But Not Necessary, to Normalize Diabetic Hyperglycemia

Endocrinology (Philadelphia), 2013-09, Vol.154 (9), p.3067-3076

2013

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Neurotrophins Acting Via TRKB Receptors Activate the JAGGED1-NOTCH2 Cell-Cell Communication Pathway to Facilitate Early Ovarian Development

Endocrinology (Philadelphia), 2011-12, Vol.152 (12), p.5005-5016

2011

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Deletion of Protein Kinase C [lamda] in POMC Neurons Predisposes to Diet-Induced Obesity

Diabetes (New York, N.Y.), 2017-04, Vol.66 (4), p.920

2017

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Randomized Study Comparing Radiofrequency Ablation with the PVAC Gold System vs. Antiarrhythmic Drugs in Elderly Patients with Symptomatic Atrial Fibrillation

Arquivos brasileiros de cardiologia, 2024-06, Vol.121 (6), p.e20230684

2024

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Microglial Inflammatory Signaling Orchestrates the Hypothalamic Immune Response to Dietary Excess and Mediates Obesity Susceptibility

Sex differences in microglial CX3CR1 signalling determine obesity susceptibility in mice

Hypothalamic Inflammation: Marker or Mechanism of Obesity Pathogenesis?

Central injection of fibroblast growth factor 1 induces sustained remission of diabetic hyperglycemia in rodents

Functional identification of a neurocircuit regulating blood glucose

Kisspeptin Receptor Haplo-insufficiency Causes Premature Ovarian Failure Despite Preserved Gonadotropin Secretion

CX3CL1 Action on Microglia Protects from Diet-Induced Obesity by Restoring POMC Neuronal Excitability and Melanocortin System Activity Impaired by High-Fat Diet Feeding

Microglial Inflammatory Signaling Orchestrates the Hypothalamic Immune Response to Dietary Excess and Mediates Obesity Susceptibility

Loss of Ntrk2/Kiss1r Signaling in Oocytes Causes Premature Ovarian Failure

Glia: silent partners in energy homeostasis and obesity pathogenesis

Hypothalamic inflammation and gliosis in obesity

Prostaglandin PGE2 Receptor EP4 Regulates Microglial Phagocytosis and Increases Susceptibility to Diet-Induced Obesity

Obesity-associated microglial inflammatory activation paradoxically improves glucose tolerance

1759-P: Prostaglandin PGE2 Receptor EP4 Signaling Deficiency in Microglia Reduces Weight Gain with Transient Glucose Improvements in Diet-Induced Obesity

Central androgen action reverses hypothalamic astrogliosis and atherogenic risk factors induced by orchiectomy and high-fat diet feeding in male mice

152-OR: Evidence Linking Loss of Hypothalamic Perineuronal Nets to Obesity Pathogenesis

1769-P: Microglial Activation and Inactivation via Designer Receptors Exclusively Activated by Designer Drugs (DREADDs) Alters Peripheral Glucose Homeostasis

212-OR: Testosterone Deficiency Increases Hypothalamic Gliosis and Metabolic Risk

Deletion of Protein Kinase C λ in POMC Neurons Predisposes to Diet-Induced Obesity

Evidence That in Uncontrolled Diabetes, Hyperglucagonemia Is Required for Ketosis but Not for Increased Hepatic Glucose Production or Hyperglycemia

Leptin Action in the Ventromedial Hypothalamic Nucleus Is Sufficient, But Not Necessary, to Normalize Diabetic Hyperglycemia

Neurotrophins Acting Via TRKB Receptors Activate the JAGGED1-NOTCH2 Cell-Cell Communication Pathway to Facilitate Early Ovarian Development

Deletion of Protein Kinase C [lamda] in POMC Neurons Predisposes to Diet-Induced Obesity

Randomized Study Comparing Radiofrequency Ablation with the PVAC Gold System vs. Antiarrhythmic Drugs in Elderly Patients with Symptomatic Atrial Fibrillation

Estudo Randomizado Comparando a Ablação por Cateter com o PVAC Gold vs. Tratamento com Fármacos Antiarrítmicos em Pacientes Idosos com Fibrilação Atrial Sintomática

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