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Intracellular generation of superoxide by the phagocyte NADPH oxidase: How, where, and what for?

Free radical biology & medicine, 2010-12, Vol.49 (12), p.1834-1845

2010

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International Union of Basic and Clinical Pharmacology. LXXIII. Nomenclature for the formyl peptide receptor (FPR) family

Pharmacological reviews, 2009-06, Vol.61 (2), p.119

2009

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Host defense peptide LL-37 selectively reduces proinflammatory macrophage responses

The Journal of immunology (1950), 2011-05, Vol.186 (9), p.5497-5505

2011

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Porphyromonas gingivalis Produce Neutrophil Specific Chemoattractants Including Short Chain Fatty Acids

Frontiers in cellular and infection microbiology, 2021-01, Vol.10, p.620681-620681

2021

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The Host Defense Peptide LL-37 Selectively Permeabilizes Apoptotic Leukocytes

Antimicrobial Agents and Chemotherapy, 2009-03, Vol.53 (3), p.1027-1038

2009

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Interleukin-8-Derived Peptide Has Antibacterial Activity

Antimicrobial Agents and Chemotherapy, 2005-09, Vol.49 (9), p.3889-3895

2005

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Molecular docking of 2-(benzimidazol-2-ylthio)-N-phenylacetamide-derived small-molecule agonists of human formyl peptide receptor 1

Journal of molecular modeling, 2012-06, Vol.18 (6), p.2831-2843

2012

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Changes in Activation States of Murine Polymorphonuclear Leukocytes (PMN) during Inflammation: a Comparison of Bone Marrow and Peritoneal Exudate PMN

Clinical and Vaccine Immunology, 2006-05, Vol.13 (5), p.575-583

2006

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Neutrophil NADPH-oxidase activation by an annexin AI peptide is transduced by the formyl peptide receptor (FPR), whereas an inhibitory signal is generated independently of the FPR family receptors

Journal of leukocyte biology, 2005-09, Vol.78 (3), p.762-771

2005

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Lipopolysaccharide-Induced Gelatinase Granule Mobilization Primes Neutrophils for Activation by Galectin-3 and Formylmethionyl-Leu-Phe

Infection and Immunity, 2001-02, Vol.69 (2), p.832-837

2001

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Interplay between signaling via the formyl peptide receptor (FPR) and chemokine receptor 3 (CCR3) in human eosinophils

Journal of leukocyte biology, 2009-08, Vol.86 (2), p.327-336

2009

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Structural Determinants in the Staphylococcus aureus -Derived Phenol-Soluble Modulin α2 Peptide Required for Neutrophil Formyl Peptide Receptor Activation

The Journal of immunology (1950), 2022-04, Vol.208 (7), p.1632-1641

2022

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Staphylococcus aureus -Derived PSMα Peptides Activate Neutrophil FPR2 but Lack the Ability to Mediate β-Arrestin Recruitment and Chemotaxis

The Journal of immunology (1950), 2019-12, Vol.203 (12), p.3349-3360

2019

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A new chemiluminescence paradox: selective inhibition of isoluminol-amplified activity in phagocytes by peptides from annexin AI

Luminescence (Chichester, England), 2008-05, Vol.23 (3), p.139-143

2008

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WHAT FORMYL PEPTIDE RECEPTORS, IF ANY, ARE TRIGGERED BY COMPOUND 43 AND LIPOXIN A(4) ?

Scandinavian journal of immunology, 2011-09

2011

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The annexin I sequence gln(9)-ala(10)-trp(11)-phe(12) is a core structure for interaction with the formyl peptide receptor 1

The Journal of biological chemistry, 2010-05, Vol.285 (19), p.14338

2010

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Lipoxin A(4) metabolites/analogues from two commercial sources have no effects on TNF-alpha-mediated priming or activation through the neutrophil formyl peptide receptors

Scandinavian journal of immunology, 2009-10, Vol.70 (4), p.396-402

2009

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Further studies on 2-arylacetamide pyridazin-3(2H)-ones: Design, synthesis and evaluation of 4,6-disubstituted analogs as formyl peptide receptors (FPRs) agonists

European journal of medicinal chemistry, 2013-06, Vol.64, p.512-528

2013

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N-formyl peptide receptor 3 (FPR3) departs from the homologous FPR2/ALX with regard to the major processes governing chemoattractant receptor regulation, expression at the cell surface and phosphorylation

The Journal of biological chemistry, 2011

2011

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A methodological approach to studies of desensitization of the formyl peptide receptor: Role of the read out system, reactive oxygen species and the specific agonist used to trigger neutrophils

Journal of immunological methods, 2010-01, Vol.352 (1), p.45-53

2010

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A non-peptide receptor inhibitor with selectivity for one of the neutrophil formyl peptide receptors, FPR 1

Biochemical pharmacology, 2012-06, Vol.83 (12), p.1655-1662

2012

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The mechanism for activation of the neutrophil NADPH‐oxidase by the peptides formyl‐Met‐Leu‐Phe and Trp‐Lys‐Tyr‐Met‐Val‐Met differs from that for interleukin‐8

Immunology, 2004-06, Vol.112 (2), p.201-210

2004

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Oxygen radical production and severity of the Guillain–Barré syndrome

Journal of neuroimmunology, 2007-12, Vol.192 (1), p.186-191

2007

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The FPR2-specific ligand MMK-1 activates the neutrophil NADPH-oxidase, but triggers no unique pathway for opening of plasma membrane calcium channels

Cell calcium (Edinburgh), 2009-05, Vol.45 (5), p.431-438

2009

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A Monocyte-Specific Peptide from Herpes Simplex Virus Type 2 Glycoprotein G Activates the NADPH-Oxidase but Not Chemotaxis through a G-Protein-Coupled Receptor Distinct from the Members of the Formyl Peptide Receptor Family

The Journal of immunology (1950), 2007-11, Vol.179 (9), p.6080-6087

2007

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Intracellular generation of superoxide by the phagocyte NADPH oxidase: How, where, and what for?

International Union of Basic and Clinical Pharmacology. LXXIII. Nomenclature for the formyl peptide receptor (FPR) family

Host defense peptide LL-37 selectively reduces proinflammatory macrophage responses

Porphyromonas gingivalis Produce Neutrophil Specific Chemoattractants Including Short Chain Fatty Acids

The Host Defense Peptide LL-37 Selectively Permeabilizes Apoptotic Leukocytes

Interleukin-8-Derived Peptide Has Antibacterial Activity

Molecular docking of 2-(benzimidazol-2-ylthio)-N-phenylacetamide-derived small-molecule agonists of human formyl peptide receptor 1

Changes in Activation States of Murine Polymorphonuclear Leukocytes (PMN) during Inflammation: a Comparison of Bone Marrow and Peritoneal Exudate PMN

Neutrophil NADPH-oxidase activation by an annexin AI peptide is transduced by the formyl peptide receptor (FPR), whereas an inhibitory signal is generated independently of the FPR family receptors

Lipopolysaccharide-Induced Gelatinase Granule Mobilization Primes Neutrophils for Activation by Galectin-3 and Formylmethionyl-Leu-Phe

Interplay between signaling via the formyl peptide receptor (FPR) and chemokine receptor 3 (CCR3) in human eosinophils

Structural Determinants in the Staphylococcus aureus -Derived Phenol-Soluble Modulin α2 Peptide Required for Neutrophil Formyl Peptide Receptor Activation

Staphylococcus aureus -Derived PSMα Peptides Activate Neutrophil FPR2 but Lack the Ability to Mediate β-Arrestin Recruitment and Chemotaxis

A new chemiluminescence paradox: selective inhibition of isoluminol-amplified activity in phagocytes by peptides from annexin AI

WHAT FORMYL PEPTIDE RECEPTORS, IF ANY, ARE TRIGGERED BY COMPOUND 43 AND LIPOXIN A(4) ?

The annexin I sequence gln(9)-ala(10)-trp(11)-phe(12) is a core structure for interaction with the formyl peptide receptor 1

Lipoxin A(4) metabolites/analogues from two commercial sources have no effects on TNF-alpha-mediated priming or activation through the neutrophil formyl peptide receptors

Further studies on 2-arylacetamide pyridazin-3(2H)-ones: Design, synthesis and evaluation of 4,6-disubstituted analogs as formyl peptide receptors (FPRs) agonists

N-formyl peptide receptor 3 (FPR3) departs from the homologous FPR2/ALX with regard to the major processes governing chemoattractant receptor regulation, expression at the cell surface and phosphorylation

A methodological approach to studies of desensitization of the formyl peptide receptor: Role of the read out system, reactive oxygen species and the specific agonist used to trigger neutrophils

A non-peptide receptor inhibitor with selectivity for one of the neutrophil formyl peptide receptors, FPR 1

The mechanism for activation of the neutrophil NADPH‐oxidase by the peptides formyl‐Met‐Leu‐Phe and Trp‐Lys‐Tyr‐Met‐Val‐Met differs from that for interleukin‐8

Oxygen radical production and severity of the Guillain–Barré syndrome

The FPR2-specific ligand MMK-1 activates the neutrophil NADPH-oxidase, but triggers no unique pathway for opening of plasma membrane calcium channels

A Monocyte-Specific Peptide from Herpes Simplex Virus Type 2 Glycoprotein G Activates the NADPH-Oxidase but Not Chemotaxis through a G-Protein-Coupled Receptor Distinct from the Members of the Formyl Peptide Receptor Family

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