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Converging Mechanisms in ALS and FTD: Disrupted RNA and Protein Homeostasis

Neuron (Cambridge, Mass.), 2013-08, Vol.79 (3), p.416-438

2013

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The Seeds of Neurodegeneration: Prion-like Spreading in ALS

Cell, 2011-10, Vol.147 (3), p.498-508

2011

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Rebuilding Chromosomes After Catastrophe: Emerging Mechanisms of Chromothripsis

Trends in cell biology, 2017-12, Vol.27 (12), p.917-930

2017

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ALS: A Disease of Motor Neurons and Their Nonneuronal Neighbors

Neuron (Cambridge, Mass.), 2006-10, Vol.52 (1), p.39-59

2006

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Understanding the role of TDP-43 and FUS/TLS in ALS and beyond

Current opinion in neurobiology, 2011-12, Vol.21 (6), p.904-919

2011

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Non-cell autonomous toxicity in neurodegenerative disorders: ALS and beyond

The Journal of cell biology, 2009-12, Vol.187 (6), p.761-772

2009

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Antisense Oligonucleotide Therapies for Neurodegenerative Diseases

Annual review of neuroscience, 2019-07, Vol.42, p.385

2019

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The SARS-CoV-2 nucleocapsid phosphoprotein forms mutually exclusive condensates with RNA and the membrane-associated M protein

Nature communications, 2021-01, Vol.12 (1), p.502-502, Article 502

2021

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Prion-like spread of protein aggregates in neurodegeneration

The Journal of experimental medicine, 2012-05, Vol.209 (5), p.889-893

2012

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Chromoanagenesis and cancer: mechanisms and consequences of localized, complex chromosomal rearrangements

Nature medicine, 2012-11, Vol.18 (11), p.1630-1638

2012

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Cytoplasmic TDP-43 De-mixing Independent of Stress Granules Drives Inhibition of Nuclear Import, Loss of Nuclear TDP-43, and Cell Death

Neuron (Cambridge, Mass.), 2019-04, Vol.102 (2), p.339-357.e7

2019

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Losing balance: the origin and impact of aneuploidy in cancer

EMBO reports, 2012-06, Vol.13 (6), p.501-514

2012

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DNA Sequence-Specific Binding of CENP-B Enhances the Fidelity of Human Centromere Function

Developmental cell, 2015-05, Vol.33 (3), p.314-327

2015

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Single-Stranded RNAs Use RNAi to Potently and Allele-Selectively Inhibit Mutant Huntingtin Expression

Cell, 2012-08, Vol.150 (5), p.895-908

2012

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Centrosome Amplification Is Sufficient to Promote Spontaneous Tumorigenesis in Mammals

Developmental cell, 2017-02, Vol.40 (3), p.313-322.e5

2017

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Sustained Therapeutic Reversal of Huntington's Disease by Transient Repression of Huntingtin Synthesis

Neuron (Cambridge, Mass.), 2012-06, Vol.74 (6), p.1031-1044

2012

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Inducible, reversible system for the rapid and complete degradation of proteins in mammalian cells

Proceedings of the National Academy of Sciences - PNAS, 2012-12, Vol.109 (49), p.E3350-E3357

2012

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Chromothripsis drives the evolution of gene amplification in cancer

Nature (London), 2021-03, Vol.591 (7848), p.137-141

2021

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Premature polyadenylation-mediated loss of stathmin-2 is a hallmark of TDP-43-dependent neurodegeneration

Nature neuroscience, 2019-02, Vol.22 (2), p.180

2019

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Degeneration and impaired regeneration of gray matter oligodendrocytes in amyotrophic lateral sclerosis

Nature neuroscience, 2013-05, Vol.16 (5), p.571-579

2013

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Muscle Expression of Mutant Androgen Receptor Accounts for Systemic and Motor Neuron Disease Phenotypes in Spinal and Bulbar Muscular Atrophy

Neuron (Cambridge, Mass.), 2014-04, Vol.82 (2), p.295-307

2014

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Selective Y centromere inactivation triggers chromosome shattering in micronuclei and repair by non-homologous end joining

Nature cell biology, 2017-01, Vol.19 (1), p.68

2017

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Macrophage Migration Inhibitory Factor as a Chaperone Inhibiting Accumulation of Misfolded SOD1

Neuron (Cambridge, Mass.), 2015-04, Vol.86 (1), p.218-232

2015

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C9ORF72 GGGGCC repeat-associated non-AUG translation is upregulated by stress through eIF2α phosphorylation

Nature communications, 2018-01, Vol.9 (1), p.51-12, Article 51

2018

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Chromosome segregation errors generate a diverse spectrum of simple and complex genomic rearrangements

Nature genetics, 2019-04, Vol.51 (4), p.705-715

2019

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Converging Mechanisms in ALS and FTD: Disrupted RNA and Protein Homeostasis

The Seeds of Neurodegeneration: Prion-like Spreading in ALS

Rebuilding Chromosomes After Catastrophe: Emerging Mechanisms of Chromothripsis

ALS: A Disease of Motor Neurons and Their Nonneuronal Neighbors

Understanding the role of TDP-43 and FUS/TLS in ALS and beyond

Non-cell autonomous toxicity in neurodegenerative disorders: ALS and beyond

Antisense Oligonucleotide Therapies for Neurodegenerative Diseases

The SARS-CoV-2 nucleocapsid phosphoprotein forms mutually exclusive condensates with RNA and the membrane-associated M protein

Prion-like spread of protein aggregates in neurodegeneration

Chromoanagenesis and cancer: mechanisms and consequences of localized, complex chromosomal rearrangements

Cytoplasmic TDP-43 De-mixing Independent of Stress Granules Drives Inhibition of Nuclear Import, Loss of Nuclear TDP-43, and Cell Death

Losing balance: the origin and impact of aneuploidy in cancer

DNA Sequence-Specific Binding of CENP-B Enhances the Fidelity of Human Centromere Function

Single-Stranded RNAs Use RNAi to Potently and Allele-Selectively Inhibit Mutant Huntingtin Expression

Centrosome Amplification Is Sufficient to Promote Spontaneous Tumorigenesis in Mammals

Sustained Therapeutic Reversal of Huntington's Disease by Transient Repression of Huntingtin Synthesis

Inducible, reversible system for the rapid and complete degradation of proteins in mammalian cells

Chromothripsis drives the evolution of gene amplification in cancer

Premature polyadenylation-mediated loss of stathmin-2 is a hallmark of TDP-43-dependent neurodegeneration

Degeneration and impaired regeneration of gray matter oligodendrocytes in amyotrophic lateral sclerosis

Muscle Expression of Mutant Androgen Receptor Accounts for Systemic and Motor Neuron Disease Phenotypes in Spinal and Bulbar Muscular Atrophy

Selective Y centromere inactivation triggers chromosome shattering in micronuclei and repair by non-homologous end joining

Macrophage Migration Inhibitory Factor as a Chaperone Inhibiting Accumulation of Misfolded SOD1

C9ORF72 GGGGCC repeat-associated non-AUG translation is upregulated by stress through eIF2α phosphorylation

Chromosome segregation errors generate a diverse spectrum of simple and complex genomic rearrangements

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