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Targeting Alterations in the RAF-MEK Pathway

Cancer discovery, 2019-03, Vol.9 (3), p.329-341

2019

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Overcoming Adaptive Resistance to KRAS Inhibitors Through Vertical Pathway Targeting

Clinical cancer research, 2020-04, Vol.26 (7), p.1538-1540

2020

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Chromothripsis drives the evolution of gene amplification in cancer

Nature (London), 2021-03, Vol.591 (7848), p.137-141

2021

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S1014 Immune Checkpoint Inhibitors for Colorectal Cancer in Patients With Inflammatory Bowel Disease

The American journal of gastroenterology, 2022-10, Vol.117 (10S), p.e734-e735

2022

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Sotorasib in KRAS p.G12C–Mutated Advanced Pancreatic Cancer

The New England journal of medicine, 2023-01, Vol.388 (1), p.33-43

2023

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Heat Shock Factor 1-dependent extracellular matrix remodeling mediates the transition from chronic intestinal inflammation to colon cancer

Nature communications, 2020-12, Vol.11 (1), p.6245-6245, Article 6245

2020

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Mechanisms of Acquired Resistance to BRAF V600E Inhibition in Colon Cancers Converge on RAF Dimerization and Are Sensitive to Its Inhibition

Cancer research (Chicago, Ill.), 2017-12, Vol.77 (23), p.6513-6523

2017

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ctDNA applications and integration in colorectal cancer: an NCI Colon and Rectal-Anal Task Forces whitepaper

Nature reviews. Clinical oncology, 2020-12, Vol.17 (12), p.757-770

2020

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RAF inhibitor PLX8394 selectively disrupts BRAF dimers and RAS-independent BRAF-mutant-driven signaling

Nature medicine, 2019-02, Vol.25 (2), p.284-291

2019

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Different hotspot p53 mutants exert distinct phenotypes and predict outcome of colorectal cancer patients

Nature communications, 2022-05, Vol.13 (1), p.2800-2800, Article 2800

2022

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Colorectal Carcinomas Containing Hypermethylated MLH1 Promoter and Wild-Type BRAF/KRAS Are Enriched for Targetable Kinase Fusions

Cancer research (Chicago, Ill.), 2019-03, Vol.79 (6), p.1047-1053

2019

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Pilot trial of combined BRAF and EGFR inhibition in BRAF-mutant metastatic colorectal cancer patients

Clinical cancer research, 2015-03, Vol.21 (6), p.1313-1320

2015

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Clinical Features and Outcomes of Patients with Colorectal Cancers Harboring NRAS Mutations

Clinical cancer research, 2017-08, Vol.23 (16), p.4753-4760

2017

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Retained mismatch repair protein expression occurs in approximately 6% of microsatellite instability-high cancers and is associated with missense mutations in mismatch repair genes

Modern pathology, 2020-05, Vol.33 (5), p.871-879

2020

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Combination Therapy and Appropriate Dosing to Target KRAS in Colorectal Cancer

The New England journal of medicine, 2023-12, Vol.389 (23), p.2197-2199

Yaeger, Rona

2023

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Allele-Specific Mechanisms of Activation of MEK1 Mutants Determine Their Properties

Cancer discovery, 2018-05, Vol.8 (5), p.648-661

2018

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Genomic Alterations Observed in Colitis-Associated Cancers Are Distinct From Those Found in Sporadic Colorectal Cancers and Vary by Type of Inflammatory Bowel Disease

Gastroenterology (New York, N.Y. 1943), 2016-08, Vol.151 (2), p.278-287.e6

2016

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Comparative sequencing analysis reveals high genomic concordance between matched primary and metastatic colorectal cancer lesions

Genome biology, 2014-08, Vol.15 (8), p.454-454, Article 454

2014

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Lineage Reversion Drives WNT Independence in Intestinal Cancer

Cancer discovery, 2020-10, Vol.10 (10), p.1590-1609

2020

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Association of Preoperative and Postoperative Serum Carcinoembryonic Antigen and Colon Cancer Outcome

JAMA oncology, 2018-03, Vol.4 (3), p.309

2018

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Targeting Alterations in the RAF-MEK Pathway

Overcoming Adaptive Resistance to KRAS Inhibitors Through Vertical Pathway Targeting

Chromothripsis drives the evolution of gene amplification in cancer

S1014 Immune Checkpoint Inhibitors for Colorectal Cancer in Patients With Inflammatory Bowel Disease

Encorafenib, Binimetinib, and Cetuximab in BRAF V600E–Mutated Colorectal Cancer

Sotorasib in KRAS p.G12C–Mutated Advanced Pancreatic Cancer

Heat Shock Factor 1-dependent extracellular matrix remodeling mediates the transition from chronic intestinal inflammation to colon cancer

Clinical Sequencing Defines the Genomic Landscape of Metastatic Colorectal Cancer

Emergence of KRAS mutations and acquired resistance to anti-EGFR therapy in colorectal cancer

Mechanisms of Acquired Resistance to BRAF V600E Inhibition in Colon Cancers Converge on RAF Dimerization and Are Sensitive to Its Inhibition

ctDNA applications and integration in colorectal cancer: an NCI Colon and Rectal-Anal Task Forces whitepaper

RAF inhibitor PLX8394 selectively disrupts BRAF dimers and RAS-independent BRAF-mutant-driven signaling

Different hotspot p53 mutants exert distinct phenotypes and predict outcome of colorectal cancer patients

Colorectal Carcinomas Containing Hypermethylated MLH1 Promoter and Wild-Type BRAF/KRAS Are Enriched for Targetable Kinase Fusions

Pilot trial of combined BRAF and EGFR inhibition in BRAF-mutant metastatic colorectal cancer patients

Clinical Features and Outcomes of Patients with Colorectal Cancers Harboring NRAS Mutations

Retained mismatch repair protein expression occurs in approximately 6% of microsatellite instability-high cancers and is associated with missense mutations in mismatch repair genes

Resistance to TRK inhibition mediated by convergent MAPK pathway activation

Combination Therapy and Appropriate Dosing to Target KRAS in Colorectal Cancer

Allele-Specific Mechanisms of Activation of MEK1 Mutants Determine Their Properties

Genomic Alterations Observed in Colitis-Associated Cancers Are Distinct From Those Found in Sporadic Colorectal Cancers and Vary by Type of Inflammatory Bowel Disease

Comparative sequencing analysis reveals high genomic concordance between matched primary and metastatic colorectal cancer lesions

Lineage Reversion Drives WNT Independence in Intestinal Cancer

Association of Preoperative and Postoperative Serum Carcinoembryonic Antigen and Colon Cancer Outcome

Functional landscapes of POLE and POLD1 mutations in checkpoint blockade-dependent antitumor immunity

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