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The Human Microcirculation: Regulation of Flow and Beyond

Circulation research, 2016-01, Vol.118 (1), p.157-172

2016

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Role of PGC-1α in Vascular Regulation: Implications for Atherosclerosis

Arteriosclerosis, thrombosis, and vascular biology, 2016-08, Vol.36 (8), p.1467

2016

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Critical Interaction Between Telomerase and Autophagy in Mediating Flow-Induced Human Arteriolar Vasodilation

Arteriosclerosis, thrombosis, and vascular biology, 2021-01, Vol.41 (1), p.446

2021

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Lipid phosphate phosphatase 3 maintains NO‐mediated flow‐mediated dilatation in human adipose resistance arterioles

The Journal of physiology, 2023-02, Vol.601 (3), p.469-481

2023

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Crossing signals: bioactive lipids in the microvasculature

American journal of physiology. Heart and circulatory physiology, 2020-05, Vol.318 (5), p.H1185-H1197

2020

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PGC-1α (Peroxisome Proliferator–Activated Receptor γ Coactivator 1-α) Overexpression in Coronary Artery Disease Recruits NO and Hydrogen Peroxide During Flow-Mediated Dilation and Protects Against Increased Intraluminal Pressure

Hypertension (Dallas, Tex. 1979), 2017-07, Vol.70 (1), p.166-173

2017

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Lysophosphatidic acid acts on LPA1 receptor to increase H2O2 during flow‐induced dilation in human adipose arterioles

British journal of pharmacology, 2018-11, Vol.175 (22), p.4266-4280

2018

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Shaker‐related voltage‐gated K+ channel expression and vasomotor function in human coronary resistance arteries

Microcirculation (New York, N.Y. 1994), 2018-01, Vol.25 (1), p.n/a

2018

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Contribution of K V 1.5 Channel to Hydrogen Peroxide-Induced Human Arteriolar Dilation and Its Modulation by Coronary Artery Disease

Circulation research, 2017-02, Vol.120 (4), p.658-669

2017

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Contribution of KV1.5 Channel to H2O2-Induced Human Arteriolar Dilation and its Modulation by Coronary Artery Disease

Circulation research, 2016-11, Vol.120 (4), p.658-669

2016

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Contribution of KV1.5 Channel to Hydrogen Peroxide–Induced Human Arteriolar Dilation and Its Modulation by Coronary Artery Disease

Circulation research, 2017-02, Vol.120 (4), p.658-669

2017

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LPA‐induced activation of LPA 1 receptor leads to the loss of NO‐mediated flow‐induced dilation in human microvessels

The FASEB journal, 2018-04, Vol.32 (S1)

2018

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LPA‐induced activation of LPA1 receptor leads to the loss of NO‐mediated flow‐induced dilation in human microvessels

The FASEB journal, 2018-04, Vol.32, p.713.15-713.15

2018

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Lysophosphatidic acid acts on LPA 1 receptor to increase H 2 O 2 during flow-induced dilation in human adipose arterioles

British journal of pharmacology, 2018-11, Vol.175 (22), p.4266-4280

2018

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Abstract 247: Mitochondrial Damage Associated Molecular Patterns Promotes Endothelial Dysfunction in the Microcirculation

Circulation research, 2018-08, Vol.123 (Suppl_1)

2018

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Dysbacteriosis an Inciting Cause of Endothelial Dysfunction mediated through Mitochondrial DNA Interactions

The FASEB journal, 2018-04, Vol.32 (S1), p.582.3-582.3

2018

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Lysophosphatidic Acid Promotes a Switch from Nitric Oxide to Hydrogen Peroxide as the Mediator of Flow‐Induced Dilation in Human Arterioles

The FASEB journal, 2016-04, Vol.30 (S1)

2016

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PGC-1alpha Overexpression in Coronary Artery Disease Recruits Nitric Oxide and Hydrogen Peroxide during Flow-mediated Dilation and Protects Against Increased Intraluminal Pressure

Hypertension (Dallas, Tex. 1979), 2017-05, Vol.70 (1), p.166-173

2017

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KV1 channel expression and vasomotor function in human coronary resistance arteries

Microcirculation (New York, N.Y. 1994), 2018-01, Vol.25 (1)

2018

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Unveiling the Mechanism of Coronary Metabolic Vasodilation: Voltage-Gated Potassium Channels and Hydrogen Peroxide

Circulation research, 2015-09, Vol.117 (7), p.589-591

2015

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Mice, rats, and guinea pigs differ in FMOs expression and tissue concentration of TMAO, a gut bacteria-derived biomarker of cardiovascular and metabolic diseases

PloS one, 2024-01, Vol.19 (1), p.e0297474-e0297474

2024

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Hypertensive rats show increased renal excretion and decreased tissue concentrations of glycine betaine, a protective osmolyte with diuretic properties

PloS one, 2024-01, Vol.19 (1), p.e0294926-e0294926

2024

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Abstract P326: Trimethylamine, A Gut Bacteria Metabolite And Air Pollutant, Increases Blood Pressure, Proteinuria And Markers Of Kidney Damage In Rats

Hypertension (Dallas, Tex. 1979), 2022-09, Vol.79 (Suppl_1)

2022

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Deoxycholic Acid, a Secondary Bile Acid, Increases Cardiac Output and Blood Pressure in Rats

Nutrients, 2023-12, Vol.16 (1), p.32

2023

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Indole-3-propionic acid, a tryptophan-derived bacterial metabolite, increases blood pressure via cardiac and vascular mechanisms in rats

American journal of physiology. Regulatory, integrative and comparative physiology, 2021-12, Vol.321 (6), p.R969-R981

2021

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The Human Microcirculation: Regulation of Flow and Beyond

Role of PGC-1α in Vascular Regulation: Implications for Atherosclerosis

Critical Interaction Between Telomerase and Autophagy in Mediating Flow-Induced Human Arteriolar Vasodilation

Lipid phosphate phosphatase 3 maintains NO‐mediated flow‐mediated dilatation in human adipose resistance arterioles

Crossing signals: bioactive lipids in the microvasculature

PGC-1α (Peroxisome Proliferator–Activated Receptor γ Coactivator 1-α) Overexpression in Coronary Artery Disease Recruits NO and Hydrogen Peroxide During Flow-Mediated Dilation and Protects Against Increased Intraluminal Pressure

Lysophosphatidic acid acts on LPA1 receptor to increase H2O2 during flow‐induced dilation in human adipose arterioles

Shaker‐related voltage‐gated K+ channel expression and vasomotor function in human coronary resistance arteries

Contribution of K V 1.5 Channel to Hydrogen Peroxide-Induced Human Arteriolar Dilation and Its Modulation by Coronary Artery Disease

Contribution of KV1.5 Channel to H2O2-Induced Human Arteriolar Dilation and its Modulation by Coronary Artery Disease

Contribution of KV1.5 Channel to Hydrogen Peroxide–Induced Human Arteriolar Dilation and Its Modulation by Coronary Artery Disease

LPA‐induced activation of LPA 1 receptor leads to the loss of NO‐mediated flow‐induced dilation in human microvessels

LPA‐induced activation of LPA1 receptor leads to the loss of NO‐mediated flow‐induced dilation in human microvessels

Lysophosphatidic acid acts on LPA 1 receptor to increase H 2 O 2 during flow-induced dilation in human adipose arterioles

Abstract 247: Mitochondrial Damage Associated Molecular Patterns Promotes Endothelial Dysfunction in the Microcirculation

Dysbacteriosis an Inciting Cause of Endothelial Dysfunction mediated through Mitochondrial DNA Interactions

Lysophosphatidic Acid Promotes a Switch from Nitric Oxide to Hydrogen Peroxide as the Mediator of Flow‐Induced Dilation in Human Arterioles

PGC-1alpha Overexpression in Coronary Artery Disease Recruits Nitric Oxide and Hydrogen Peroxide during Flow-mediated Dilation and Protects Against Increased Intraluminal Pressure

KV1 channel expression and vasomotor function in human coronary resistance arteries

Unveiling the Mechanism of Coronary Metabolic Vasodilation: Voltage-Gated Potassium Channels and Hydrogen Peroxide

Mice, rats, and guinea pigs differ in FMOs expression and tissue concentration of TMAO, a gut bacteria-derived biomarker of cardiovascular and metabolic diseases

Hypertensive rats show increased renal excretion and decreased tissue concentrations of glycine betaine, a protective osmolyte with diuretic properties

Abstract P326: Trimethylamine, A Gut Bacteria Metabolite And Air Pollutant, Increases Blood Pressure, Proteinuria And Markers Of Kidney Damage In Rats

Deoxycholic Acid, a Secondary Bile Acid, Increases Cardiac Output and Blood Pressure in Rats

Indole-3-propionic acid, a tryptophan-derived bacterial metabolite, increases blood pressure via cardiac and vascular mechanisms in rats

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