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TMEM106B is a receptor mediating ACE2-independent SARS-CoV-2 cell entry

Cell, 2023-08, Vol.186 (16), p.3427-3442.e22

2023

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A common Alu insertion in the 3'UTR of TMEM106B is associated with risk of dementia

Alzheimer's & dementia, 2024-07, Vol.20 (7), p.5071-5077

2024

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Emerging Trends in Cryo-EM-based Structural Studies of Neuropathological Amyloids

Journal of molecular biology, 2023-12, Vol.435 (24), p.168361-168361, Article 168361

Mishra, Suman

2023

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Homotypic fibrillization of TMEM106B across diverse neurodegenerative diseases

Cell, 2022-04, Vol.185 (8), p.1346-1355.e15

2022

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Gene replacement‐Alzheimer's disease (GR‐AD): Modeling the genetics of human dementias in mice

Alzheimer's & dementia, 2024-04, Vol.20 (4), p.3080-3087

2024

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Cryo-EM structures of pathogenic fibrils and their impact on neurodegenerative disease research

Neuron (Cambridge, Mass.), 2024-07, Vol.112 (14), p.2269-2288

2024

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Lysosome dysfunction as a cause of neurodegenerative diseases: Lessons from frontotemporal dementia and amyotrophic lateral sclerosis

Neurobiology of disease, 2021-07, Vol.154, p.105360-105360, Article 105360

2021

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The lysosomal function of progranulin, a guardian against neurodegeneration

Acta neuropathologica, 2018-07, Vol.136 (1), p.1-17

2018

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The endolysosomal pathway and ALS/FTD

Trends in neurosciences (Regular ed.), 2023-12, Vol.46 (12), p.1025-1041

2023

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Genetics of FTLD: overview and what else we can expect from genetic studies

Journal of neurochemistry, 2016-08, Vol.138 (S1), p.32-53

2016

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Loss of TMEM106B and PGRN leads to severe lysosomal abnormalities and neurodegeneration in mice

EMBO reports, 2020-10, Vol.21 (10), p.e50219-n/a

2020

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rs1990622 variant associates with Alzheimer's disease and regulates TMEM106B expression in human brain tissues

BMC medicine, 2021-01, Vol.19 (1), p.11-11, Article 11

2021

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Loss of TMEM106B Ameliorates Lysosomal and Frontotemporal Dementia-Related Phenotypes in Progranulin-Deficient Mice

Neuron (Cambridge, Mass.), 2017-07, Vol.95 (2), p.281-296.e6

2017

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Loss of Tmem106b exacerbates FTLD pathologies and causes motor deficits in progranulin‐deficient mice

EMBO reports, 2020-10, Vol.21 (10), p.e50197-n/a

2020

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Loss of TMEM106B potentiates lysosomal and FTLD‐like pathology in progranulin‐deficient mice

EMBO reports, 2020-10, Vol.21 (10), p.e50241-n/a

2020

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Loss of TMEM106B leads to myelination deficits: implications for frontotemporal dementia treatment strategies

Brain (London, England : 1878), 2020-06, Vol.143 (6), p.1905-1919

2020

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Differential diagnosis of amnestic dementia patients based on an FDG‐PET signature of autopsy‐confirmed LATE‐NC

Alzheimer's & dementia, 2023-04, Vol.19 (4), p.1234-1244

2023

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C-terminal TMEM106B fragments in human brain correlate with disease-associated TMEM106B haplotypes

Brain (London, England : 1878), 2023-10, Vol.146 (10), p.4055-4064

2023

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Physiological and pathological functions of TMEM106B: a gene associated with brain aging and multiple brain disorders

Acta neuropathologica, 2021-03, Vol.141 (3), p.327-339

2021

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A role of the frontotemporal lobar degeneration risk factor TMEM106B in myelination

Brain (London, England : 1878), 2020-07, Vol.143 (7), p.2255-2271

2020

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A Dementia-Associated Risk Variant near TMEM106B Alters Chromatin Architecture and Gene Expression

American journal of human genetics, 2017-11, Vol.101 (5), p.643-663

2017

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Accumulation of TMEM106B C-terminal fragments in neurodegenerative disease and aging

Acta neuropathologica, 2023-03, Vol.145 (3), p.285-302

2023

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Differential Aging Analysis in Human Cerebral Cortex Identifies Variants in TMEM106B and GRN that Regulate Aging Phenotypes

Cell systems, 2017-04, Vol.4 (4), p.404-415.e5

2017

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A recurrent de novo mutation in TMEM106B causes hypomyelinating leukodystrophy

Brain (London, England : 1878), 2017-12, Vol.140 (12), p.3105-3111

2017

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Pathological, imaging and genetic characteristics support the existence of distinct TDP-43 types in non-FTLD brains

Acta neuropathologica, 2019-02, Vol.137 (2), p.227-238

2019

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TMEM106B is a receptor mediating ACE2-independent SARS-CoV-2 cell entry

A common Alu insertion in the 3'UTR of TMEM106B is associated with risk of dementia

Emerging Trends in Cryo-EM-based Structural Studies of Neuropathological Amyloids

Homotypic fibrillization of TMEM106B across diverse neurodegenerative diseases

Gene replacement‐Alzheimer's disease (GR‐AD): Modeling the genetics of human dementias in mice

Cryo-EM structures of pathogenic fibrils and their impact on neurodegenerative disease research

Lysosome dysfunction as a cause of neurodegenerative diseases: Lessons from frontotemporal dementia and amyotrophic lateral sclerosis

The lysosomal function of progranulin, a guardian against neurodegeneration

The endolysosomal pathway and ALS/FTD

Genetics of FTLD: overview and what else we can expect from genetic studies

Loss of TMEM106B and PGRN leads to severe lysosomal abnormalities and neurodegeneration in mice

rs1990622 variant associates with Alzheimer's disease and regulates TMEM106B expression in human brain tissues

Loss of TMEM106B Ameliorates Lysosomal and Frontotemporal Dementia-Related Phenotypes in Progranulin-Deficient Mice

Loss of Tmem106b exacerbates FTLD pathologies and causes motor deficits in progranulin‐deficient mice

Loss of TMEM106B potentiates lysosomal and FTLD‐like pathology in progranulin‐deficient mice

Loss of TMEM106B leads to myelination deficits: implications for frontotemporal dementia treatment strategies

Differential diagnosis of amnestic dementia patients based on an FDG‐PET signature of autopsy‐confirmed LATE‐NC

C-terminal TMEM106B fragments in human brain correlate with disease-associated TMEM106B haplotypes

Physiological and pathological functions of TMEM106B: a gene associated with brain aging and multiple brain disorders

A role of the frontotemporal lobar degeneration risk factor TMEM106B in myelination

A Dementia-Associated Risk Variant near TMEM106B Alters Chromatin Architecture and Gene Expression

Accumulation of TMEM106B C-terminal fragments in neurodegenerative disease and aging

Differential Aging Analysis in Human Cerebral Cortex Identifies Variants in TMEM106B and GRN that Regulate Aging Phenotypes

A recurrent de novo mutation in TMEM106B causes hypomyelinating leukodystrophy

Pathological, imaging and genetic characteristics support the existence of distinct TDP-43 types in non-FTLD brains

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