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The Pseudokinase MLKL and the Kinase RIPK3 Have Distinct Roles in Autoimmune Disease Caused by Loss of Death-Receptor-Induced Apoptosis

Immunity (Cambridge, Mass.), 2016-09, Vol.45 (3), p.513-526

2016

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Data-independent acquisition method for ubiquitinome analysis reveals regulation of circadian biology

Nature communications, 2021-01, Vol.12 (1), p.254-13, Article 254

2021

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The structural context of posttranslational modifications at a proteome-wide scale

PLoS biology, 2022-05, Vol.20 (5), p.e3001636-e3001636

2022

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Phosphoproteome profiling uncovers a key role for CDKs in TNF signaling

Nature communications, 2021-10, Vol.12 (1), p.6053-6053, Article 6053

2021

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NLRP3 inflammasome activation downstream of cytoplasmic LPS recognition by both caspase‐4 and caspase‐5

European journal of immunology, 2015-10, Vol.45 (10), p.2918-2926

2015

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Activation of the pseudokinase MLKL unleashes the four-helix bundle domain to induce membrane localization and necroptotic cell death

Proceedings of the National Academy of Sciences - PNAS, 2014-10, Vol.111 (42), p.15072-15077

2014

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Quantitative and Dynamic Catalogs of Proteins Released during Apoptotic and Necroptotic Cell Death

Cell reports (Cambridge), 2020-01, Vol.30 (4), p.1260-1270.e5

2020

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You are what you eat and how you digest it! A discussion on inflammatory efferocytosis

Frontiers in cell and developmental biology, 2023-02, Vol.11, p.1132696-1132696

Tanzer, Maria C

2023

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Conformational switching of the pseudokinase domain promotes human MLKL tetramerization and cell death by necroptosis

Nature communications, 2018-06, Vol.9 (1), p.2422-15, Article 2422

2018

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The brace helices of MLKL mediate interdomain communication and oligomerisation to regulate cell death by necroptosis

Cell death and differentiation, 2018-09, Vol.25 (9), p.1567-1580

2018

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Necroptosis signalling is tuned by phosphorylation of MLKL residues outside the pseudokinase domain activation loop

Biochemical journal, 2015-10, Vol.471 (2), p.255-265

2015

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Insights into the evolution of divergent nucleotide-binding mechanisms among pseudokinases revealed by crystal structures of human and mouse MLKL

Biochemical journal, 2014-02, Vol.457 (3), p.369-377

2014

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A proteomic perspective on TNF-mediated signalling and cell death

Biochemical Society transactions, 2022-02, Vol.50 (1), p.13

Tanzer, Maria C

2022

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Editorial: Cell death: from its induction to the removal of dying cells

Frontiers in cell and developmental biology, 2023-04, Vol.11, p.1195775-1195775

2023

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Laser-mediated rupture of chlamydial inclusions triggers pathogen egress and host cell necrosis

Nature communications, 2017-03, Vol.8 (1), p.14729-14729, Article 14729

2017

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Multilevel proteomics reveals host perturbations by SARS-CoV-2 and SARS-CoV

Nature (London), 2021-06, Vol.594 (7862), p.246-252

2021

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Rapid and In-Depth Coverage of the (Phospho-)Proteome With Deep Libraries and Optimal Window Design for dia-PASEF

Molecular & cellular proteomics, 2022-09, Vol.21 (9), p.100279-100279, Article 100279

2022

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The caspase-8 inhibitor emricasan combines with the SMAC mimetic birinapant to induce necroptosis and treat acute myeloid leukemia

Science translational medicine, 2016-05, Vol.8 (339), p.339ra69

2016

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Phosphorylation of muramyl peptides by NAGK is required for NOD2 activation

Nature (London), 2022-09, Vol.609 (7927), p.590-596

2022

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The polycomb repressive complex 2 governs life and death of peripheral T cells

Blood, 2014-07, Vol.124 (5), p.737-749

2014

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Combination of IAP antagonist and IFNγ activates novel caspase-10- and RIPK1-dependent cell death pathways

Cell death and differentiation, 2017-03, Vol.24 (3), p.481-491

2017

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Potent Inhibition of Necroptosis by Simultaneously Targeting Multiple Effectors of the Pathway

ACS chemical biology, 2020-10, Vol.15 (10), p.2702-2713

2020

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Gene-selective transcription promotes the inhibition of tissue reparative macrophages by TNF

Life science alliance, 2022-04, Vol.5 (4), p.e202101315

2022

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The Pseudokinase MLKL and the Kinase RIPK3 Have Distinct Roles in Autoimmune Disease Caused by Loss of Death-Receptor-Induced Apoptosis

Data-independent acquisition method for ubiquitinome analysis reveals regulation of circadian biology

The structural context of posttranslational modifications at a proteome-wide scale

Phosphoproteome profiling uncovers a key role for CDKs in TNF signaling

NLRP3 inflammasome activation downstream of cytoplasmic LPS recognition by both caspase‐4 and caspase‐5

Activation of the pseudokinase MLKL unleashes the four-helix bundle domain to induce membrane localization and necroptotic cell death

Quantitative and Dynamic Catalogs of Proteins Released during Apoptotic and Necroptotic Cell Death

You are what you eat and how you digest it! A discussion on inflammatory efferocytosis

Conformational switching of the pseudokinase domain promotes human MLKL tetramerization and cell death by necroptosis

The brace helices of MLKL mediate interdomain communication and oligomerisation to regulate cell death by necroptosis

Necroptosis signalling is tuned by phosphorylation of MLKL residues outside the pseudokinase domain activation loop

Insights into the evolution of divergent nucleotide-binding mechanisms among pseudokinases revealed by crystal structures of human and mouse MLKL

A proteomic perspective on TNF-mediated signalling and cell death

TRAF2 regulates TNF and NF-κB signalling to suppress apoptosis and skin inflammation independently of Sphingosine kinase 1

Editorial: Cell death: from its induction to the removal of dying cells

Laser-mediated rupture of chlamydial inclusions triggers pathogen egress and host cell necrosis

Multilevel proteomics reveals host perturbations by SARS-CoV-2 and SARS-CoV

Rapid and In-Depth Coverage of the (Phospho-)Proteome With Deep Libraries and Optimal Window Design for dia-PASEF

The caspase-8 inhibitor emricasan combines with the SMAC mimetic birinapant to induce necroptosis and treat acute myeloid leukemia

Phosphorylation of muramyl peptides by NAGK is required for NOD2 activation

The polycomb repressive complex 2 governs life and death of peripheral T cells

Combination of IAP antagonist and IFNγ activates novel caspase-10- and RIPK1-dependent cell death pathways

Potent Inhibition of Necroptosis by Simultaneously Targeting Multiple Effectors of the Pathway

Gene-selective transcription promotes the inhibition of tissue reparative macrophages by TNF

Correction: TRAF2 regulates TNF and NF-κB signalling to suppress apoptosis and skin inflammation independently of Sphingosine kinase 1

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