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The Fate of a Hapten - From the Skin to Modification of Macrophage Migration Inhibitory Factor (MIF) in Lymph Nodes

Scientific reports, 2018-02, Vol.8 (1), p.2895-11, Article 2895

2018

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Danger signals derived from stressed and necrotic epithelial cells activate human eosinophils

Immunology, 2004-08, Vol.112 (4), p.605-614

2004

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The pilosebaceous unit—a phthalate-induced pathway to skin sensitization

Toxicology and applied pharmacology, 2012-10, Vol.264 (1), p.114-120

2012

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Interplay between signaling via the formyl peptide receptor (FPR) and chemokine receptor 3 (CCR3) in human eosinophils

Journal of leukocyte biology, 2009-08, Vol.86 (2), p.327-336

2009

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Photodegradation of Dibenzoylmethanes: Potential Cause of Photocontact Allergy to Sunscreens

Chemical research in toxicology, 2009-11, Vol.22 (11), p.1881-1892

2009

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Cyclosporin H, Boc-MLF and Boc-FLFLF are antagonists that preferentially inhibit activity triggered through the formyl peptide receptor

Inflammation, 2007-12, Vol.30 (6), p.224-229

2007

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Caged Fluorescent Haptens Reveal the Generation of Cryptic Epitopes in Allergic Contact Dermatitis

Journal of investigative dermatology, 2011-07, Vol.131 (7), p.1486-1493

2011

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An attempt to design an isoluminol-hydrogen peroxidase-amplified CL that measures intracellularly produced H2O2 in phagocytes: sensitivity for H2O2 is not high enough to allow detection

Luminescence, 2007-09, Vol.22 (5), p.507-510

2007

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Modification and Expulsion of Keratins by Human Epidermal Keratinocytes upon Hapten Exposure in Vitro

Chemical research in toxicology, 2011-05, Vol.24 (5), p.737-743

2011

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The FPR2-specific ligand MMK-1 activates the neutrophil NADPH-oxidase, but triggers no unique pathway for opening of plasma membrane calcium channels

Cell calcium (Edinburgh), 2009-05, Vol.45 (5), p.431-438

2009

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Changes in the ratio between FPR and FPRL1 triggered superoxide production in human neutrophils—A tool in analysing receptor specific events

Journal of immunological methods, 2008-02, Vol.331 (1), p.50-58

2008

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An attempt to design an isoluminol–hydrogen peroxidase‐amplified CL that measures intracellularly produced H 2 O 2 in phagocytes: sensitivity for H 2 O 2 is not high enough to allow detection

Luminescence (Chichester, England), 2007-09, Vol.22 (5), p.507-510

2007

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Danger signals derived from stressed and necrotic epithelial cells activate human eosinophils

Immunology, 2004, Vol.112 (4), p.605

2004

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The Fate of a Hapten - From the Skin to Modification of Macrophage Migration Inhibitory Factor (MIF) in Lymph Nodes

Danger signals derived from stressed and necrotic epithelial cells activate human eosinophils

The pilosebaceous unit—a phthalate-induced pathway to skin sensitization

Interplay between signaling via the formyl peptide receptor (FPR) and chemokine receptor 3 (CCR3) in human eosinophils

Photodegradation of Dibenzoylmethanes: Potential Cause of Photocontact Allergy to Sunscreens

Cyclosporin H, Boc-MLF and Boc-FLFLF are antagonists that preferentially inhibit activity triggered through the formyl peptide receptor

Caged Fluorescent Haptens Reveal the Generation of Cryptic Epitopes in Allergic Contact Dermatitis

An attempt to design an isoluminol-hydrogen peroxidase-amplified CL that measures intracellularly produced H2O2 in phagocytes: sensitivity for H2O2 is not high enough to allow detection

Modification and Expulsion of Keratins by Human Epidermal Keratinocytes upon Hapten Exposure in Vitro

The FPR2-specific ligand MMK-1 activates the neutrophil NADPH-oxidase, but triggers no unique pathway for opening of plasma membrane calcium channels

Changes in the ratio between FPR and FPRL1 triggered superoxide production in human neutrophils—A tool in analysing receptor specific events

An attempt to design an isoluminol–hydrogen peroxidase‐amplified CL that measures intracellularly produced H 2 O 2 in phagocytes: sensitivity for H 2 O 2 is not high enough to allow detection

Danger signals derived from stressed and necrotic epithelial cells activate human eosinophils

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