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The arrhythmogenic consequences of increasing late INa in the cardiomyocyte

Cardiovascular research, 2013-09, Vol.99 (4), p.600-611

2013

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An increase of late sodium current induces delayed afterdepolarizations and sustained triggered activity in atrial myocytes

American journal of physiology. Heart and circulatory physiology, 2008-05, Vol.294 (5), p.H2031-H2039

2008

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Basal late sodium current is a significant contributor to the duration of action potential of guinea pig ventricular myocytes

Physiological reports, 2017-05, Vol.5 (10), p.e13295-n/a

2017

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Blocking late sodium current reduces hydrogen peroxide-induced arrhythmogenic activity and contractile dysfunction

The Journal of pharmacology and experimental therapeutics, 2006-07, Vol.318 (1), p.214

2006

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A slowly inactivating sodium current contributes to spontaneous diastolic depolarization of atrial myocytes

American journal of physiology. Heart and circulatory physiology, 2009-10, Vol.297 (4), p.H1254-H1262

2009

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Antagonism by Ranolazine of the Pro-Arrhythmic Effects of Increasing Late INa in Guinea Pig Ventricular Myocytes

Journal of cardiovascular pharmacology, 2004-08, Vol.44 (2), p.192-199

2004

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The late Na+ current (INa) inhibitor ranolazine attenuates effects of palmitoyl-L-carnitine to increase late INa and cause ventricular diastolic dysfunction

The Journal of pharmacology and experimental therapeutics, 2009-08, Vol.330 (2), p.550

2009

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Antiarrhythmic effects of ranolazine in a guinea pig in vitro model of long-QT syndrome

The Journal of pharmacology and experimental therapeutics, 2004-08, Vol.310 (2), p.599

2004

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Enhanced basal late sodium current appears to underlie the age-related prolongation of action potential duration in guinea pig ventricular myocytes

Journal of applied physiology (1985), 2018-10, Vol.125 (4), p.1329-1338

2018

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An increase in late sodium current potentiates the proarrhythmic activities of low-risk QT-prolonging drugs in female rabbit hearts

The Journal of pharmacology and experimental therapeutics, 2006-02, Vol.316 (2), p.718

2006

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Ranolazine Attenuates Hypoxia- and Hydrogen Peroxide-induced Increases in Sodium Channel Late Openings in Ventricular Myocytes

Journal of cardiovascular pharmacology, 2014-07, Vol.64 (1), p.60-68

2014

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Probing an intelligent predictive maintenance approach with deep learning and augmented reality for machine tools in IoT-enabled manufacturing

Robotics and computer-integrated manufacturing, 2022-10, Vol.77, p.102357, Article 102357

2022

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The role of late I Na in development of cardiac arrhythmias

Handbook of experimental pharmacology, 2014, Vol.221, p.137

2014

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Use of preclinical assays to predict risk of drug-induced torsades de pointes

Heart rhythm, 2005-11, Vol.2 (11), p.S16-S22

2005

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Abstract 17251: Enhanced Late Sodium Current Predisposes Cardiomyocytes From Aged Guinea Pigs to the Arrhythmogenic Effects of Hydrogen Peroxide

Circulation (New York, N.Y.), 2015-11, Vol.132 (Suppl_3 Suppl 3), p.A17251-A17251

2015

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Abstract 17193: Inhibiting Late Sodium Current Attenuates Isoproterenol-Induced Transient Inward Current and Delayed Afterdepolarizations in Ventricular Myocytes

Circulation (New York, N.Y.), 2015-11, Vol.132 (Suppl_3 Suppl 3), p.A17193-A17193

2015

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Contribution of frequency-augmented inward Ca2+ current to myocardial contractility

Canadian journal of physiology and pharmacology, 2009-01, Vol.87 (1), p.69

2009

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Modulation of late sodium current by Ca2+–calmodulin‐dependent protein kinase II, protein kinase C and Ca2+ during hypoxia in rabbit ventricular myocytes

Experimental physiology, 2017-07, Vol.102 (7), p.818-834

2017

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Selective attenuation of isoproterenol-stimulated arrhythmic activity by a partial agonist of adenosine A1 receptor

Circulation (New York, N.Y.), 2002, Vol.105 (1), p.118-123

2002

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A computational modelling approach combined with cellular electrophysiology data provides insights into the therapeutic benefit of targeting the late Na+ current

The Journal of physiology, 2015-03, Vol.593 (6), p.1429-1442

2015

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Modulation of late sodium current by Ca 2+ -calmodulin-dependent protein kinase II, protein kinase C and Ca 2+ during hypoxia in rabbit ventricular myocytes

Experimental physiology, 2017-07, Vol.102 (7), p.818-834

2017

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Larger late sodium current density as well as greater sensitivities to ATX II and ranolazine in rabbit left atrial than left ventricular myocytes

American journal of physiology. Heart and circulatory physiology, 2014-02, Vol.306 (3), p.H455-H461

2014

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Protein kinase C and Ca(2+) -calmodulin-dependent protein kinase II mediate the enlarged reverse INCX induced by ouabain-increased late sodium current in rabbit ventricular myocytes

Experimental physiology, 2015-04, Vol.100 (4), p.399

2015

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Reducing the late sodium current improves cardiac function during sodium pump inhibition by ouabain

The Journal of pharmacology and experimental therapeutics, 2011-05, Vol.337 (2), p.513

2011

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Selective attenuation by adenosine of arrhythmogenic action of isoproterenol on ventricular myocytes

American journal of physiology. Heart and circulatory physiology, 2001-06, Vol.49 (6), p.H2789-H2795

2001

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The arrhythmogenic consequences of increasing late INa in the cardiomyocyte

An increase of late sodium current induces delayed afterdepolarizations and sustained triggered activity in atrial myocytes

Basal late sodium current is a significant contributor to the duration of action potential of guinea pig ventricular myocytes

Blocking late sodium current reduces hydrogen peroxide-induced arrhythmogenic activity and contractile dysfunction

A slowly inactivating sodium current contributes to spontaneous diastolic depolarization of atrial myocytes

Antagonism by Ranolazine of the Pro-Arrhythmic Effects of Increasing Late INa in Guinea Pig Ventricular Myocytes

The late Na+ current (INa) inhibitor ranolazine attenuates effects of palmitoyl-L-carnitine to increase late INa and cause ventricular diastolic dysfunction

Antiarrhythmic effects of ranolazine in a guinea pig in vitro model of long-QT syndrome

Enhanced basal late sodium current appears to underlie the age-related prolongation of action potential duration in guinea pig ventricular myocytes

An increase in late sodium current potentiates the proarrhythmic activities of low-risk QT-prolonging drugs in female rabbit hearts

Ranolazine Attenuates Hypoxia- and Hydrogen Peroxide-induced Increases in Sodium Channel Late Openings in Ventricular Myocytes

Probing an intelligent predictive maintenance approach with deep learning and augmented reality for machine tools in IoT-enabled manufacturing

The role of late I Na in development of cardiac arrhythmias

Use of preclinical assays to predict risk of drug-induced torsades de pointes

Abstract 17251: Enhanced Late Sodium Current Predisposes Cardiomyocytes From Aged Guinea Pigs to the Arrhythmogenic Effects of Hydrogen Peroxide

Abstract 17193: Inhibiting Late Sodium Current Attenuates Isoproterenol-Induced Transient Inward Current and Delayed Afterdepolarizations in Ventricular Myocytes

Contribution of frequency-augmented inward Ca2+ current to myocardial contractility

Modulation of late sodium current by Ca2+–calmodulin‐dependent protein kinase II, protein kinase C and Ca2+ during hypoxia in rabbit ventricular myocytes

Selective attenuation of isoproterenol-stimulated arrhythmic activity by a partial agonist of adenosine A1 receptor

A computational modelling approach combined with cellular electrophysiology data provides insights into the therapeutic benefit of targeting the late Na+ current

Modulation of late sodium current by Ca 2+ -calmodulin-dependent protein kinase II, protein kinase C and Ca 2+ during hypoxia in rabbit ventricular myocytes

Larger late sodium current density as well as greater sensitivities to ATX II and ranolazine in rabbit left atrial than left ventricular myocytes

Protein kinase C and Ca(2+) -calmodulin-dependent protein kinase II mediate the enlarged reverse INCX induced by ouabain-increased late sodium current in rabbit ventricular myocytes

Reducing the late sodium current improves cardiac function during sodium pump inhibition by ouabain

Selective attenuation by adenosine of arrhythmogenic action of isoproterenol on ventricular myocytes

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