UB Paderborn / Katalog / Suche / in CDI

Sortiert nach

Sie befinden Sich nicht im Netzwerk der Universität Paderborn. Der Zugriff auf elektronische Ressourcen ist gegebenenfalls nur via VPN oder Shibboleth (DFN-AAI) möglich. mehr Informationen...

A computational model predicts adjunctive pharmacotherapy for cardiac safety via selective inhibition of the late cardiac Na current

Journal of molecular and cellular cardiology, 2016-10, Vol.99, p.151-161

2016

Link zum Volltext

Verfügbarkeit prüfen

FK506 activates BMPR2, rescues endothelial dysfunction, and reverses pulmonary hypertension

The Journal of clinical investigation, 2013-08, Vol.123 (8), p.3600-3613

2013

Link zum Volltext

Verfügbarkeit prüfen

A novel, potent, and selective inhibitor of cardiac late sodium current suppresses experimental arrhythmias

The Journal of pharmacology and experimental therapeutics, 2013-01, Vol.344 (1), p.23

2013

Link zum Volltext

Verfügbarkeit prüfen

The novel late Na+ current inhibitor, GS‐6615 (eleclazine) and its anti‐arrhythmic effects in rabbit isolated heart preparations

British journal of pharmacology, 2016-11, Vol.173 (21), p.3088-3098

2016

Link zum Volltext

Verfügbarkeit prüfen

Modulation of myosin by cardiac myosin binding protein-C peptides improves cardiac contractility in ex-vivo experimental heart failure models

Scientific reports, 2022-03, Vol.12 (1), p.4337-4337, Article 4337

2022

Link zum Volltext

Verfügbarkeit prüfen

Intracellular Na+ overload causes oxidation of CaMKII and leads to Ca2+ mishandling in isolated ventricular myocytes

Journal of molecular and cellular cardiology, 2014-11, Vol.76, p.247-256

2014

Link zum Volltext

Verfügbarkeit prüfen

Use-dependent block of cardiac late Na+ current by ranolazine

Heart rhythm, 2009-11, Vol.6 (11), p.1625-1631

2009

Link zum Volltext

Verfügbarkeit prüfen

Ranolazine block of human Nav1.4 sodium channels and paramyotonia congenita mutants

Channels (Austin, Tex.), 2011-03, Vol.5 (2), p.161-172

2011

Link zum Volltext

Verfügbarkeit prüfen

Eleclazine exhibits enhanced selectivity for long QT syndrome type 3–associated late Na+ current

Heart rhythm, 2018-02, Vol.15 (2), p.277-286

2018

Link zum Volltext

Verfügbarkeit prüfen

Selective inhibition of physiological late Na + current stabilizes ventricular repolarization

American journal of physiology. Heart and circulatory physiology, 2018-02, Vol.314 (2), p.H236-H245

2018

Link zum Volltext

Verfügbarkeit prüfen

Dysregulated Smooth Muscle Cell BMPR2-ARRB2 Axis Causes Pulmonary Hypertension

Circulation research, 2023-03, Vol.132 (5), p.545-564

2023

Link zum Volltext

Verfügbarkeit prüfen

Abstract 13402: FHL-1 Contributes to and Colocalizes With Titin in Cardiac Hypertrophy

Circulation (New York, N.Y.), 2020-11, Vol.142 (Suppl_3 Suppl 3), p.A13402-A13402

2020

Link zum Volltext

Verfügbarkeit prüfen

Abstract 17193: Inhibiting Late Sodium Current Attenuates Isoproterenol-Induced Transient Inward Current and Delayed Afterdepolarizations in Ventricular Myocytes

Circulation (New York, N.Y.), 2015-11, Vol.132 (Suppl_3 Suppl 3), p.A17193-A17193

2015

Link zum Volltext

Verfügbarkeit prüfen

Contribution of the late sodium current to intracellular sodium and calcium overload in rabbit ventricular myocytes treated by anemone toxin

American journal of physiology. Heart and circulatory physiology, 2016-02, Vol.310 (3), p.H426-H435

2016

Link zum Volltext

Verfügbarkeit prüfen

Block of Na+ currents and suppression of action potentials in embryonic rat dorsal root ganglion neurons by ranolazine

Neuropharmacology, 2012-06, Vol.62 (7), p.2250-2259

2012

Link zum Volltext

Verfügbarkeit prüfen

The novel late Na super(+) current inhibitor, GS-6615 (eleclazine) and its anti-arrhythmic effects in rabbit isolated heart preparations

British journal of pharmacology, 2016-11, Vol.173 (21), p.3088-3098

2016

Link zum Volltext

Verfügbarkeit prüfen

Post-junctional mechanisms involved in the potentiation of cardiac adrenergic responses by cocaine

European journal of pharmacology, 2000-05, Vol.397 (1), p.139-150

2000

Link zum Volltext

Verfügbarkeit prüfen

Selective Inhibition of Late Na+ Current Reduces Arrhythmic Activity in Spontaneously Hypertensive Rat Myocytes

Biophysical journal, 2014-01, Vol.106 (2), p.328a-328a

2014

Link zum Volltext

Verfügbarkeit prüfen

Abstract 16820: CaMKIIδc Transgenic Mice Develop Spontaneous Ventricular Arrhythmias Which Can Be Suppressed by Inhibition of Late Na+ Current

Circulation (New York, N.Y.), 2014-11, Vol.130 (suppl_2)

2014

Link zum Volltext

Verfügbarkeit prüfen

Discovery of Dihydrobenzoxazepinone (GS-6615) Late Sodium Current Inhibitor (Late I Nai), a Phase II Agent with Demonstrated Preclinical Anti-Ischemic and Antiarrhythmic Properties

Journal of medicinal chemistry, 2016-10, Vol.59 (19), p.9005-9017

2016

Link zum Volltext

Verfügbarkeit prüfen

SM22α-targeted deletion of bone morphogenetic protein receptor 1A in mice impairs cardiac and vascular development, and influences organogenesis

Development (Cambridge), 2008-09, Vol.135 (17), p.2981-2991

2008

Link zum Volltext

Verfügbarkeit prüfen

Inhibition of the late sodium current slows t-tubule disruption during the progression of hypertensive heart disease in the rat

American journal of physiology. Heart and circulatory physiology, 2013-10, Vol.305 (7), p.H1068-H1079

2013

Link zum Volltext

Verfügbarkeit prüfen

Smooth Muscle Protein 22α–Mediated Patchy Deletion of Bmpr1a Impairs Cardiac Contractility but Protects Against Pulmonary Vascular Remodeling

Circulation research, 2008-02, Vol.102 (3), p.380-388

2008

Link zum Volltext

Verfügbarkeit prüfen

Patchy deletion of Bmpr1a potentiates proximal pulmonary artery remodeling in mice exposed to chronic hypoxia

Biomechanics and modeling in mechanobiology, 2013, Vol.12 (1), p.33-42

2013

Link zum Volltext

Verfügbarkeit prüfen

Intracellular Na+ overload causes oxidation of CaMKII and leads to Ca 2 + mishandling in isolated ventricular myocytes

Journal of molecular and cellular cardiology, 2014, Vol.76, p.247-256

2014

Link zum Volltext

Verfügbarkeit prüfen

1 – 25 von 43

Suche als RSS-Feed

Suchergebnisse filtern

Filter anzeigen

Nur zeigen

Geprüft (Peer-Review)
(43)
Mit Volltextzugriff
(39)

Publikationsform

Artikel
(43)

Sprache

Englisch
(43)
Japanisch
(3)

Erscheinungsjahr

n.n

Menü

Weitere Dienste

Einstellungen

A computational model predicts adjunctive pharmacotherapy for cardiac safety via selective inhibition of the late cardiac Na current

FK506 activates BMPR2, rescues endothelial dysfunction, and reverses pulmonary hypertension

A novel, potent, and selective inhibitor of cardiac late sodium current suppresses experimental arrhythmias

The novel late Na+ current inhibitor, GS‐6615 (eleclazine) and its anti‐arrhythmic effects in rabbit isolated heart preparations

Modulation of myosin by cardiac myosin binding protein-C peptides improves cardiac contractility in ex-vivo experimental heart failure models

Intracellular Na+ overload causes oxidation of CaMKII and leads to Ca2+ mishandling in isolated ventricular myocytes

Use-dependent block of cardiac late Na+ current by ranolazine

Ranolazine block of human Nav1.4 sodium channels and paramyotonia congenita mutants

Eleclazine exhibits enhanced selectivity for long QT syndrome type 3–associated late Na+ current

Selective inhibition of physiological late Na + current stabilizes ventricular repolarization

Dysregulated Smooth Muscle Cell BMPR2-ARRB2 Axis Causes Pulmonary Hypertension

Abstract 13402: FHL-1 Contributes to and Colocalizes With Titin in Cardiac Hypertrophy

Abstract 17193: Inhibiting Late Sodium Current Attenuates Isoproterenol-Induced Transient Inward Current and Delayed Afterdepolarizations in Ventricular Myocytes

Contribution of the late sodium current to intracellular sodium and calcium overload in rabbit ventricular myocytes treated by anemone toxin

Block of Na+ currents and suppression of action potentials in embryonic rat dorsal root ganglion neurons by ranolazine

The novel late Na super(+) current inhibitor, GS-6615 (eleclazine) and its anti-arrhythmic effects in rabbit isolated heart preparations

Post-junctional mechanisms involved in the potentiation of cardiac adrenergic responses by cocaine

Selective Inhibition of Late Na+ Current Reduces Arrhythmic Activity in Spontaneously Hypertensive Rat Myocytes

Abstract 16820: CaMKIIδc Transgenic Mice Develop Spontaneous Ventricular Arrhythmias Which Can Be Suppressed by Inhibition of Late Na+ Current

Discovery of Dihydrobenzoxazepinone (GS-6615) Late Sodium Current Inhibitor (Late I Nai), a Phase II Agent with Demonstrated Preclinical Anti-Ischemic and Antiarrhythmic Properties

SM22α-targeted deletion of bone morphogenetic protein receptor 1A in mice impairs cardiac and vascular development, and influences organogenesis

Inhibition of the late sodium current slows t-tubule disruption during the progression of hypertensive heart disease in the rat

Smooth Muscle Protein 22α–Mediated Patchy Deletion of Bmpr1a Impairs Cardiac Contractility but Protects Against Pulmonary Vascular Remodeling

Patchy deletion of Bmpr1a potentiates proximal pulmonary artery remodeling in mice exposed to chronic hypoxia

Intracellular Na+ overload causes oxidation of CaMKII and leads to Ca 2 + mishandling in isolated ventricular myocytes

Suchergebnisse filtern