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Cardiac late Na+ current: Proarrhythmic effects, roles in long QT syndromes, and pathological relationship to CaMKII and oxidative stress

Heart rhythm, 2015-02, Vol.12 (2), p.440-448

2015

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The arrhythmogenic consequences of increasing late INa in the cardiomyocyte

Cardiovascular research, 2013-09, Vol.99 (4), p.600-611

2013

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Pathophysiology and pharmacology of the cardiac "late sodium current."

Pharmacology & therapeutics (Oxford), 2008-09, Vol.119 (3), p.326-339

2008

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An increase of late sodium current induces delayed afterdepolarizations and sustained triggered activity in atrial myocytes

American journal of physiology. Heart and circulatory physiology, 2008-05, Vol.294 (5), p.H2031-H2039

2008

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A novel, potent, and selective inhibitor of cardiac late sodium current suppresses experimental arrhythmias

The Journal of pharmacology and experimental therapeutics, 2013-01, Vol.344 (1), p.23

2013

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Blocking late sodium current reduces hydrogen peroxide-induced arrhythmogenic activity and contractile dysfunction

The Journal of pharmacology and experimental therapeutics, 2006-07, Vol.318 (1), p.214

2006

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Late sodium current contributes to the reverse rate-dependent effect of IKr inhibition on ventricular repolarization

Circulation (New York, N.Y.), 2011-04, Vol.123 (16), p.1713-1720

2011

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Intracellular Na+ overload causes oxidation of CaMKII and leads to Ca2+ mishandling in isolated ventricular myocytes

Journal of molecular and cellular cardiology, 2014-11, Vol.76, p.247-256

2014

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Adenosine and Adenosine Receptors in the Cardiovascular System: Biochemistry, Physiology, and Pharmacology

The American journal of cardiology, 1997-06, Vol.79 (12), p.2-10

1997

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Late sodium current inhibition as a new cardioprotective approach

Journal of molecular and cellular cardiology, 2008-06, Vol.44 (6), p.954-967

2008

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A slowly inactivating sodium current contributes to spontaneous diastolic depolarization of atrial myocytes

American journal of physiology. Heart and circulatory physiology, 2009-10, Vol.297 (4), p.H1254-H1262

2009

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Use-dependent block of cardiac late Na+ current by ranolazine

Heart rhythm, 2009-11, Vol.6 (11), p.1625-1631

2009

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Antagonism by Ranolazine of the Pro-Arrhythmic Effects of Increasing Late INa in Guinea Pig Ventricular Myocytes

Journal of cardiovascular pharmacology, 2004-08, Vol.44 (2), p.192-199

2004

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Antiarrhythmic effects of ranolazine in a guinea pig in vitro model of long-QT syndrome

The Journal of pharmacology and experimental therapeutics, 2004-08, Vol.310 (2), p.599

2004

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Rapid Kinetic Interactions of Ranolazine With HERG K+ Current

Journal of cardiovascular pharmacology, 2008-06, Vol.51 (6), p.581-589

2008

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Pharmacology and therapeutic applications of A1 adenosine receptor ligands

Current topics in medicinal chemistry, 2003-02, Vol.3 (4), p.369

2003

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Reduction of repolarization reserve unmasks the proarrhythmic role of endogenous late Na+ current in the heart

American journal of physiology. Heart and circulatory physiology, 2009-09, Vol.297 (3), p.H1048-H1057

2009

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Calmodulin kinase II and protein kinase C mediate the effect of increased intracellular calcium to augment late sodium current in rabbit ventricular myocytes

American Journal of Physiology: Cell Physiology, 2012-04, Vol.302 (8), p.C1141-C1151

2012

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An increase in late sodium current potentiates the proarrhythmic activities of low-risk QT-prolonging drugs in female rabbit hearts

The Journal of pharmacology and experimental therapeutics, 2006-02, Vol.316 (2), p.718

2006

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Role of late sodium current in modulating the proarrhythmic and antiarrhythmic effects of quinidine

Heart rhythm, 2008-12, Vol.5 (12), p.1726-1734

2008

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The late Na+ current (INa) inhibitor ranolazine attenuates effects of palmitoyl-L-carnitine to increase late INa and cause ventricular diastolic dysfunction

The Journal of pharmacology and experimental therapeutics, 2009-08, Vol.330 (2), p.550

2009

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Ranolazine, an antianginal agent, markedly reduces ventricular arrhythmias induced by ischemia and ischemia-reperfusion

American journal of physiology. Heart and circulatory physiology, 2009-11, Vol.297 (5), p.H1923-H1929

2009

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Block of Na+ currents and suppression of action potentials in embryonic rat dorsal root ganglion neurons by ranolazine

Neuropharmacology, 2012-06, Vol.62 (7), p.2250-2259

2012

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Augmentation of late sodium current unmasks the proarrhythmic effects of amiodarone

Cardiovascular research, 2008-02, Vol.77 (3), p.481-488

2008

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Larger late sodium current density as well as greater sensitivities to ATX II and ranolazine in rabbit left atrial than left ventricular myocytes

American journal of physiology. Heart and circulatory physiology, 2014-02, Vol.306 (3), p.H455-H461

2014

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Cardiac late Na+ current: Proarrhythmic effects, roles in long QT syndromes, and pathological relationship to CaMKII and oxidative stress

The arrhythmogenic consequences of increasing late INa in the cardiomyocyte

Pathophysiology and pharmacology of the cardiac "late sodium current."

An increase of late sodium current induces delayed afterdepolarizations and sustained triggered activity in atrial myocytes

A novel, potent, and selective inhibitor of cardiac late sodium current suppresses experimental arrhythmias

Blocking late sodium current reduces hydrogen peroxide-induced arrhythmogenic activity and contractile dysfunction

Late sodium current contributes to the reverse rate-dependent effect of IKr inhibition on ventricular repolarization

Intracellular Na+ overload causes oxidation of CaMKII and leads to Ca2+ mishandling in isolated ventricular myocytes

Adenosine and Adenosine Receptors in the Cardiovascular System: Biochemistry, Physiology, and Pharmacology

Late sodium current inhibition as a new cardioprotective approach

A slowly inactivating sodium current contributes to spontaneous diastolic depolarization of atrial myocytes

Use-dependent block of cardiac late Na+ current by ranolazine

Antagonism by Ranolazine of the Pro-Arrhythmic Effects of Increasing Late INa in Guinea Pig Ventricular Myocytes

Antiarrhythmic effects of ranolazine in a guinea pig in vitro model of long-QT syndrome

Rapid Kinetic Interactions of Ranolazine With HERG K+ Current

Pharmacology and therapeutic applications of A1 adenosine receptor ligands

Reduction of repolarization reserve unmasks the proarrhythmic role of endogenous late Na+ current in the heart

Calmodulin kinase II and protein kinase C mediate the effect of increased intracellular calcium to augment late sodium current in rabbit ventricular myocytes

An increase in late sodium current potentiates the proarrhythmic activities of low-risk QT-prolonging drugs in female rabbit hearts

Role of late sodium current in modulating the proarrhythmic and antiarrhythmic effects of quinidine

The late Na+ current (INa) inhibitor ranolazine attenuates effects of palmitoyl-L-carnitine to increase late INa and cause ventricular diastolic dysfunction

Ranolazine, an antianginal agent, markedly reduces ventricular arrhythmias induced by ischemia and ischemia-reperfusion

Block of Na+ currents and suppression of action potentials in embryonic rat dorsal root ganglion neurons by ranolazine

Augmentation of late sodium current unmasks the proarrhythmic effects of amiodarone

Larger late sodium current density as well as greater sensitivities to ATX II and ranolazine in rabbit left atrial than left ventricular myocytes

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