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Inhibition of overactivated p38 MAPK can restore hematopoiesis in myelodysplastic syndrome progenitors

Blood, 2006-12, Vol.108 (13), p.4170-4177

2006

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Meta-analysis of microarray studies reveals a novel hematopoietic progenitor cell signature and demonstrates feasibility of inter-platform data integration

PloS one, 2008-08, Vol.3 (8), p.e2965-e2965

2008

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Inhibition of the TGF-β receptor I kinase promotes hematopoiesis in MDS

Blood, 2008-10, Vol.112 (8), p.3434-3443

2008

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Transforming growth factor-beta signaling in normal and malignant hematopoiesis

Journal of interferon & cytokine research, 2007-07, Vol.27 (7), p.543

2007

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Inhibition of p38α MAPK disrupts the pathological loop of proinflammatory factor production in the myelodysplastic syndrome bone marrow microenvironment

Leukemia & lymphoma, 2008-01, Vol.49 (10), p.1963-1975

2008

Volltextzugriff (PDF)

Design and Synthesis of Novel Derivatives of ATRA Demonstrate the Combined Importance of Acid Moiety and Conjugated Double Bonds in Its Binding to PMLRAR-Alpha Oncogene in Acute Promyelocytic Leukemia

Blood, 2008-11, Vol.112 (11), p.5036-5036

2008

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Integrative Genomic Analysis Reveals Aberrant Epigenetic Marks in MDS That Can Be Seen in Peripheral Blood Leucocytes

Blood, 2008-11, Vol.112 (11), p.595-595

2008

Volltextzugriff (PDF)

A High Resolution Epigenomic Map of Myelofibrosis Reveals Multiple Chromosomal Deletions and Amplifications Accompanied by a High Level of Functionally Important Methylation

Blood, 2006-11, Vol.108 (11), p.2684-2684

2006

Volltextzugriff (PDF)

Meta-Transcriptome of Bone Marrow Stem Cells Demonstrates Platform and Lab Dependant Variability in Gene Expression and Reveals a Novel Set of Enriched Genes

Blood, 2006-11, Vol.108 (11), p.4189-4189

2006

Volltextzugriff (PDF)

High Resolution Epigenomic Mapping of Myelodysplastic Syndrome Reveals a High Level of Functionally Important Methylation

Blood, 2006-11, Vol.108 (11), p.2637-2637

2006

Volltextzugriff (PDF)

Myelosuppresive Effects of Interferon [slpha] and Transforming Growth Factor β Can Be Reversed by Novel p38 MAPK Inhibitor SD-282 through Inhibition of Cell Cycle Arrest

Blood, 2004-11, Vol.104 (11), p.4229-4229

2004

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SD-208, a Novel Transforming Growth Factor Beta Receptor I Kinase Inhibitor, Can Stimulate Hematopoiesis in Myelodysplastic Syndrome Progenitors

Blood, 2005-11, Vol.106 (11), p.3448-3448

2005

Volltextzugriff (PDF)

Novel P38 MAP Kinase Inhibitor and Anti-P38 RNA Interference as Potential Therapeutic Approaches in Myelodysplastic Syndromes

Blood, 2004-11, Vol.104 (11), p.470-470

2004

Volltextzugriff (PDF)

Inhibition of p38alpha MAPK disrupts the pathological loop of proinflammatory factor production in the myelodysplastic syndrome bone marrow microenvironment

Leukemia & lymphoma, 2008-10, Vol.49 (10), p.1963-1975

2008

Volltextzugriff (PDF)

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Inhibition of overactivated p38 MAPK can restore hematopoiesis in myelodysplastic syndrome progenitors

Meta-analysis of microarray studies reveals a novel hematopoietic progenitor cell signature and demonstrates feasibility of inter-platform data integration

Inhibition of the TGF-β receptor I kinase promotes hematopoiesis in MDS

Transforming growth factor-beta signaling in normal and malignant hematopoiesis

Inhibition of p38α MAPK disrupts the pathological loop of proinflammatory factor production in the myelodysplastic syndrome bone marrow microenvironment

Design and Synthesis of Novel Derivatives of ATRA Demonstrate the Combined Importance of Acid Moiety and Conjugated Double Bonds in Its Binding to PMLRAR-Alpha Oncogene in Acute Promyelocytic Leukemia

Integrative Genomic Analysis Reveals Aberrant Epigenetic Marks in MDS That Can Be Seen in Peripheral Blood Leucocytes

A High Resolution Epigenomic Map of Myelofibrosis Reveals Multiple Chromosomal Deletions and Amplifications Accompanied by a High Level of Functionally Important Methylation

Meta-Transcriptome of Bone Marrow Stem Cells Demonstrates Platform and Lab Dependant Variability in Gene Expression and Reveals a Novel Set of Enriched Genes

High Resolution Epigenomic Mapping of Myelodysplastic Syndrome Reveals a High Level of Functionally Important Methylation

Myelosuppresive Effects of Interferon [slpha] and Transforming Growth Factor β Can Be Reversed by Novel p38 MAPK Inhibitor SD-282 through Inhibition of Cell Cycle Arrest

SD-208, a Novel Transforming Growth Factor Beta Receptor I Kinase Inhibitor, Can Stimulate Hematopoiesis in Myelodysplastic Syndrome Progenitors

Novel P38 MAP Kinase Inhibitor and Anti-P38 RNA Interference as Potential Therapeutic Approaches in Myelodysplastic Syndromes

Inhibition of the TGF-β receptor I kinase promotes hematopoiesis in MDS. Commentary

Inhibition of p38alpha MAPK disrupts the pathological loop of proinflammatory factor production in the myelodysplastic syndrome bone marrow microenvironment

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