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The molecular pathogenesis of the myelodysplastic syndromes

European journal of haematology, 2015-07, Vol.95 (1), p.3-15

2015

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U2AF1 mutations induce oncogenic IRAK4 isoforms and activate innate immune pathways in myeloid malignancies

Nature cell biology, 2019-05, Vol.21 (5), p.640

2019

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Splicing factor gene mutations in the myelodysplastic syndromes: impact on disease phenotype and therapeutic applications

Advances in biological regulation, 2017-01, Vol.63, p.59-70

2017

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IL8-CXCR2 pathway inhibition as a therapeutic strategy against MDS and AML stem cells

Blood, 2015-05, Vol.125 (20), p.3144-3152

2015

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Application of CRISPR/Cas9 genome editing to the study and treatment of disease

Archives of toxicology, 2015-07, Vol.89 (7), p.1023-1034

2015

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Gene expression and risk of leukemic transformation in myelodysplasia

Blood, 2017-12, Vol.130 (24), p.2642-2653

2017

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Cell-specific proteome analyses of human bone marrow reveal molecular features of age-dependent functional decline

Nature communications, 2018-10, Vol.9 (1), p.4004-18, Article 4004

2018

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ASXL1 mutations are associated with distinct epigenomic alterations that lead to sensitivity to venetoclax and azacytidine

Blood cancer journal (New York), 2021-09, Vol.11 (9), p.157-157, Article 157

2021

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Identification of a BRCA1-mRNA Splicing Complex Required for Efficient DNA Repair and Maintenance of Genomic Stability

Molecular cell, 2014-05, Vol.54 (3), p.445-459

2014

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Advances in the 5q− syndrome

Blood, 2010-12, Vol.116 (26), p.5803-5811

2010

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Recent Advances in the 5q- Syndrome

Mediterranean journal of hematology and infectious diseases, 2015-05, Vol.7 (1), p.e2015037-e2015037

2015

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Lenalidomide inhibits the malignant clone and up-regulates the SPARC gene mapping to the commonly deleted region in 5q- syndrome patients

Proceedings of the National Academy of Sciences - PNAS, 2007-07, Vol.104 (27), p.11406-11411

2007

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Phospholipase C beta1 (PI‐PLCbeta1)/Cyclin D3/protein kinase C (PKC) alpha signaling modulation during iron‐induced oxidative stress in myelodysplastic syndromes (MDS)

The FASEB journal, 2020-11, Vol.34 (11), p.15400-15416

2020

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Impact of Splicing Factor Mutations on Pre-mRNA Splicing in the Myelodysplastic Syndromes

Current pharmaceutical design, 2016-05, Vol.22 (16), p.2333

2016

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The impact of spliceosome mutations in MDS

HemaSphere, 2019-06, Vol.3 (Suppl), p.132-134

2019

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ASXL1 mutation correction by CRISPR/Cas9 restores gene function in leukemia cells and increases survival in mouse xenografts

Oncotarget, 2015-12, Vol.6 (42), p.44061-44071

2015

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Splicing factor mutations in the myelodysplastic syndromes: target genes and therapeutic approaches

Advances in biological regulation, 2018-01, Vol.67, p.13-29

2018

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Clonal heterogeneity in the 5q- syndrome: p53 expressing progenitors prevail during lenalidomide treatment and expand at disease progression

Haematologica (Roma), 2009-12, Vol.94 (12), p.1762

2009

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Aberrant overexpression of CD14 on granulocytes sensitizes the innate immune response in mDia1 heterozygous del(5q) MDS

Blood, 2014-07, Vol.124 (5), p.780-790

2014

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The molecular pathogenesis of the myelodysplastic syndromes

U2AF1 mutations induce oncogenic IRAK4 isoforms and activate innate immune pathways in myeloid malignancies

Splicing factor gene mutations in the myelodysplastic syndromes: impact on disease phenotype and therapeutic applications

IL8-CXCR2 pathway inhibition as a therapeutic strategy against MDS and AML stem cells

Application of CRISPR/Cas9 genome editing to the study and treatment of disease

SF3B1 mutations induce R-loop accumulation and DNA damage in MDS and leukemia cells with therapeutic implications

Gene expression and risk of leukemic transformation in myelodysplasia

Cell-specific proteome analyses of human bone marrow reveal molecular features of age-dependent functional decline

ASXL1 mutations are associated with distinct epigenomic alterations that lead to sensitivity to venetoclax and azacytidine

SIRT1 Activation Disrupts Maintenance of Myelodysplastic Syndrome Stem and Progenitor Cells by Restoring TET2 Function

Identification of a BRCA1-mRNA Splicing Complex Required for Efficient DNA Repair and Maintenance of Genomic Stability

Advances in the 5q− syndrome

Stem and progenitor cells in myelodysplastic syndromes show aberrant stage-specific expansion and harbor genetic and epigenetic alterations

Recent Advances in the 5q- Syndrome

Response of high-risk MDS to azacitidine and lenalidomide is impacted by baseline and acquired mutations in a cluster of three inositide-specific genes

Overexpression of IL-1 receptor accessory protein in stem and progenitor cells and outcome correlation in AML and MDS

Lenalidomide inhibits the malignant clone and up-regulates the SPARC gene mapping to the commonly deleted region in 5q- syndrome patients

Phospholipase C beta1 (PI‐PLCbeta1)/Cyclin D3/protein kinase C (PKC) alpha signaling modulation during iron‐induced oxidative stress in myelodysplastic syndromes (MDS)

Impact of Splicing Factor Mutations on Pre-mRNA Splicing in the Myelodysplastic Syndromes

The impact of spliceosome mutations in MDS

ASXL1 mutation correction by CRISPR/Cas9 restores gene function in leukemia cells and increases survival in mouse xenografts

Loss of heterozygosity in 7q myeloid disorders: clinical associations and genomic pathogenesis

Splicing factor mutations in the myelodysplastic syndromes: target genes and therapeutic approaches

Clonal heterogeneity in the 5q- syndrome: p53 expressing progenitors prevail during lenalidomide treatment and expand at disease progression

Aberrant overexpression of CD14 on granulocytes sensitizes the innate immune response in mDia1 heterozygous del(5q) MDS

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