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Inhibiting the system xC−/glutathione axis selectively targets cancers with mutant-p53 accumulation

Nature communications, 2017-03, Vol.8 (1), p.14844-14844, Article 14844

2017

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Oncogenic PIK3CA induces centrosome amplification and tolerance to genome doubling

Nature communications, 2017-11, Vol.8 (1), p.1773-15, Article 1773

2017

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Mutation of the PIK3CA gene in ovarian and breast cancer

Cancer research (Chicago, Ill.), 2004-11, Vol.64 (21), p.7678-7681

2004

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Physiological levels of Pik3ca(H1047R) mutation in the mouse mammary gland results in ductal hyperplasia and formation of ERα-positive tumors

PloS one, 2012, Vol.7 (5), p.e36924-e36924

2012

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Mutation analysis of PIK3CA and PIK3CB in esophageal cancer and Barrett's esophagus

International journal of cancer, 2006-05, Vol.118 (10), p.2644-2646

2006

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An activating Pik3ca mutation coupled with Pten loss is sufficient to initiate ovarian tumorigenesis in mice

The Journal of clinical investigation, 2012-02, Vol.122 (2), p.553-557

2012

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APR-246 potently inhibits tumour growth and overcomes chemoresistance in preclinical models of oesophageal adenocarcinoma

Gut, 2015-10, Vol.64 (10), p.1506-1516

2015

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Identification of Pik3ca Mutation as a Genetic Driver of Prostate Cancer That Cooperates with Pten Loss to Accelerate Progression and Castration-Resistant Growth

Cancer discovery, 2018-06, Vol.8 (6), p.764-779

2018

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Physiological Levels of Pik3caH1047R Mutation in the Mouse Mammary Gland Results in Ductal Hyperplasia and Formation of ERα-Positive Tumors

PloS one, 2012-05, Vol.7 (5), p.e36924

2012

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Heterozygous expression of the oncogenic Pik3ca(H1047R) mutation during murine development results in fatal embryonic and extraembryonic defects

Developmental biology, 2015-08, Vol.404 (1), p.14-26

2015

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Ubiquitous expression of the Pik3caH1047R mutation promotes hypoglycemia, hypoinsulinemia, and organomegaly

The FASEB journal, 2015-04, Vol.29 (4), p.1426-1434

2015

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Physiological expression of the PI3K-activating mutation Pik3ca(H1047R) combines with Apc loss to promote development of invasive intestinal adenocarcinomas in mice

Biochemical journal, 2014-03, Vol.458 (2), p.251-258

2014

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The HER2 I655V Polymorphism and Risk of Breast Cancer in Women < Age 40 Years

Cancer epidemiology, biomarkers & prevention, 2003-10, Vol.12 (10), p.1109-1111

2003

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Novel metastatic models of esophageal adenocarcinoma derived from FLO-1 cells highlight the importance of E-cadherin in cancer metastasis

Oncotarget, 2016-12, Vol.7 (50), p.83342-83358

2016

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The DNMT3B C-->T promoter polymorphism and risk of breast cancer in a British population: a case-control study

Breast cancer research : BCR, 2004-01, Vol.6 (4), p.R390-R394, Article R390

2004

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Novel metastatic models of esophageal adenocarcinoma derived from FLO-1 cells highlight the importance of E-cadherin in cancer metastasis

Oncotarget, 2016-12, Vol.7 (50), p.83342-83358

2016

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Heterozygous expression of the oncogenic Pik3caH1047R mutation during murine development results in fatal embryonic and extraembryonic defects

Developmental biology, 2015-08, Vol.404 (1), p.14-26

2015

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The AIB1 glutamine repeat polymorphism is not associated with risk of breast cancer before age 40 years in Australian women

Breast cancer research : BCR, 2005-01, Vol.7 (3), p.R353-R356, Article R353

2005

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Abstract 4618: A novel Pik3ca -driven mouse model and syngeneic cancer cell line for the preclinical testing of targeted and immune therapies for anal squamous cell carcinoma (ASCC)

Cancer research (Chicago, Ill.), 2019-07, Vol.79 (13_Supplement), p.4618-4618

2019

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Abstract 86: Heterozygous expression of an oncogenic Pik3ca mutation during murine development results in fatal embryonic and extra-embryonic defects

Cancer research (Chicago, Ill.), 2014-10, Vol.74 (19_Supplement), p.86-86

2014

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Abstract 3289: Tissue specific expression of the PI 3-kinase mutation Pik3caH1047R induces hyperplasia and tumorigenesis in a mouse model

Cancer research (Chicago, Ill.), 2012-04, Vol.72 (8_Supplement), p.3289-3289

2012

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Physiological expression of the PI3K-activating mutation Pik3ca H1047R combines with Apc loss to promote development of invasive intestinal adenocarcinomas in mice

Biochemical journal, 2014-03, Vol.458 (2), p.251-258

2014

Volltextzugriff (PDF)

Ubiquitous expression of the Pik3ca H1047R mutation promotes hypoglycemia, hypoinsulinemia, and organomegaly

The FASEB journal, 2015-04, Vol.29 (4), p.1426-1434

2015

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Physiological Levels of Pik3ca.sup.H1047R Mutation in the Mouse Mammary Gland Results in Ductal Hyperplasia and Formation of ER[alpha]-Positive Tumors

PloS one, 2012-05, Vol.7 (5), p.e36924

2012

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Inhibiting the system x C - /glutathione axis selectively targets cancers with mutant-p53 accumulation

Nature communications, 2017-03, Vol.8, p.14844

2017

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Inhibiting the system xC−/glutathione axis selectively targets cancers with mutant-p53 accumulation

Oncogenic PIK3CA induces centrosome amplification and tolerance to genome doubling

Mutation of the PIK3CA gene in ovarian and breast cancer

Physiological levels of Pik3ca(H1047R) mutation in the mouse mammary gland results in ductal hyperplasia and formation of ERα-positive tumors

Mutation analysis of PIK3CA and PIK3CB in esophageal cancer and Barrett's esophagus

An activating Pik3ca mutation coupled with Pten loss is sufficient to initiate ovarian tumorigenesis in mice

APR-246 potently inhibits tumour growth and overcomes chemoresistance in preclinical models of oesophageal adenocarcinoma

Identification of Pik3ca Mutation as a Genetic Driver of Prostate Cancer That Cooperates with Pten Loss to Accelerate Progression and Castration-Resistant Growth

Physiological Levels of Pik3caH1047R Mutation in the Mouse Mammary Gland Results in Ductal Hyperplasia and Formation of ERα-Positive Tumors

Heterozygous expression of the oncogenic Pik3ca(H1047R) mutation during murine development results in fatal embryonic and extraembryonic defects

Ubiquitous expression of the Pik3caH1047R mutation promotes hypoglycemia, hypoinsulinemia, and organomegaly

Physiological expression of the PI3K-activating mutation Pik3ca(H1047R) combines with Apc loss to promote development of invasive intestinal adenocarcinomas in mice

The HER2 I655V Polymorphism and Risk of Breast Cancer in Women < Age 40 Years

Novel metastatic models of esophageal adenocarcinoma derived from FLO-1 cells highlight the importance of E-cadherin in cancer metastasis

The DNMT3B C-->T promoter polymorphism and risk of breast cancer in a British population: a case-control study

Novel metastatic models of esophageal adenocarcinoma derived from FLO-1 cells highlight the importance of E-cadherin in cancer metastasis

Heterozygous expression of the oncogenic Pik3caH1047R mutation during murine development results in fatal embryonic and extraembryonic defects

The AIB1 glutamine repeat polymorphism is not associated with risk of breast cancer before age 40 years in Australian women

Abstract 4618: A novel Pik3ca -driven mouse model and syngeneic cancer cell line for the preclinical testing of targeted and immune therapies for anal squamous cell carcinoma (ASCC)

Abstract 86: Heterozygous expression of an oncogenic Pik3ca mutation during murine development results in fatal embryonic and extra-embryonic defects

Abstract 3289: Tissue specific expression of the PI 3-kinase mutation Pik3caH1047R induces hyperplasia and tumorigenesis in a mouse model

Physiological expression of the PI3K-activating mutation Pik3ca H1047R combines with Apc loss to promote development of invasive intestinal adenocarcinomas in mice

Ubiquitous expression of the Pik3ca H1047R mutation promotes hypoglycemia, hypoinsulinemia, and organomegaly

Physiological Levels of Pik3ca.sup.H1047R Mutation in the Mouse Mammary Gland Results in Ductal Hyperplasia and Formation of ER[alpha]-Positive Tumors

Inhibiting the system x C - /glutathione axis selectively targets cancers with mutant-p53 accumulation

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