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NLRP3 is activated in Alzheimer’s disease and contributes to pathology in APP/PS1 mice

Nature (London), 2013-01, Vol.493 (7434), p.674-678

2013

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Microglial PD‐1 stimulation by astrocytic PD‐L1 suppresses neuroinflammation and Alzheimer’s disease pathology

The EMBO journal, 2021-12, Vol.40 (24), p.e108662-n/a

2021

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Distinct and non-redundant roles of microglia and myeloid subsets in mouse models of Alzheimer's disease

The Journal of neuroscience, 2011-08, Vol.31 (31), p.11159-11171

2011

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Nitration of Tyrosine 10 Critically Enhances Amyloid β Aggregation and Plaque Formation

Neuron (Cambridge, Mass.), 2011-09, Vol.71 (5), p.833-844

2011

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Locus ceruleus controls Alzheimer's disease pathology by modulating microglial functions through norepinephrine

Proceedings of the National Academy of Sciences - PNAS, 2010-03, Vol.107 (13), p.6058-6063

2010

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Innate immune activation in neurodegenerative disease

Nature reviews. Immunology, 2014-07, Vol.14 (7), p.463-477

2014

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Sepsis causes neuroinflammation and concomitant decrease of cerebral metabolism

Journal of neuroinflammation, 2008-09, Vol.5 (1), p.38-38, Article 38

2008

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Impact and Therapeutic Potential of PPARs in Alzheimers Disease

Current neuropharmacology, 2011-12, Vol.9 (4), p.643-650

2011

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Characterization and clinical use of inflammatory cerebrospinal fluid protein markers in Alzheimer's disease

Alzheimer's research & therapy, 2018-02, Vol.10 (1), p.25-25, Article 25

2018

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Selective Loss of Noradrenaline Exacerbates Early Cognitive Dysfunction and Synaptic Deficits in APP/PS1 Mice

Biological psychiatry (1969), 2013-03, Vol.73 (5), p.454-463

2013

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Induced LC degeneration in APP/PS1 transgenic mice accelerates early cerebral amyloidosis and cognitive deficits

Neurochemistry international, 2010-11, Vol.57 (4), p.375-382

2010

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PPARγ/RXRα-induced and CD36-mediated microglial amyloid-β phagocytosis results in cognitive improvement in amyloid precursor protein/presenilin 1 mice

The Journal of neuroscience, 2012-11, Vol.32 (48), p.17321-17331

2012

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Microglia-derived ASC specks cross-seed amyloid-β in Alzheimer’s disease

Nature (London), 2017-12, Vol.552 (7685), p.355-361

2017

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Imaging microglial activation and glucose consumption in a mouse model of Alzheimer's disease

Neurobiology of aging, 2013, Vol.34 (1), p.351-354

2013

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Truncated and modified amyloid-beta species

Alzheimer's research & therapy, 2014-05, Vol.6 (3), p.28-28, Article 28

2014

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PPARs in Alzheimer's Disease

PPAR Research, 2008-01, Vol.2008 (2008), p.581-588-052

2008

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Dysregulation of TLR5 and TAM Ligands in the Alzheimer’s Brain as Contributors to Disease Progression

Molecular neurobiology, 2019-09, Vol.56 (9), p.6539-6550

2019

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CXCR3 promotes plaque formation and behavioral deficits in an Alzheimer's disease model

The Journal of clinical investigation, 2015-01, Vol.125 (1), p.365-378

2015

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SFRP1 modulates astrocyte‐to‐microglia crosstalk in acute and chronic neuroinflammation

EMBO reports, 2021-11, Vol.22 (11), p.e51696-n/a

2021

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Critical role of astroglial apolipoprotein E and liver X receptor-α expression for microglial Aβ phagocytosis

The Journal of neuroscience, 2011-05, Vol.31 (19), p.7049-7059

2011

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Nitric Oxide Decreases the Enzymatic Activity of Insulin Degrading Enzyme in APP/PS1 Mice

Journal of neuroimmune pharmacology, 2012-03, Vol.7 (1), p.165-172

2012

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Postoperative Ileus Involves Interleukin-1 Receptor Signaling in Enteric Glia

Gastroenterology (New York, N.Y. 1943), 2014, Vol.146 (1), p.176-187.e1

2014

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Mrp14 deficiency ameliorates amyloid β burden by increasing microglial phagocytosis and modulation of amyloid precursor protein processing

The Journal of neuroscience, 2012-12, Vol.32 (49), p.17824-17829

2012

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Variability and temporal dynamics of novel object recognition in aging male C57BL/6 mice

Behavioural processes, 2018-12, Vol.157, p.711-716

2018

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NLRP3 is activated in Alzheimer’s disease and contributes to pathology in APP/PS1 mice

Microglial PD‐1 stimulation by astrocytic PD‐L1 suppresses neuroinflammation and Alzheimer’s disease pathology

Distinct and non-redundant roles of microglia and myeloid subsets in mouse models of Alzheimer's disease

Nitration of Tyrosine 10 Critically Enhances Amyloid β Aggregation and Plaque Formation

Locus ceruleus controls Alzheimer's disease pathology by modulating microglial functions through norepinephrine

Innate immune activation in neurodegenerative disease

Sepsis causes neuroinflammation and concomitant decrease of cerebral metabolism

Impact and Therapeutic Potential of PPARs in Alzheimers Disease

Characterization and clinical use of inflammatory cerebrospinal fluid protein markers in Alzheimer's disease

Selective Loss of Noradrenaline Exacerbates Early Cognitive Dysfunction and Synaptic Deficits in APP/PS1 Mice

Induced LC degeneration in APP/PS1 transgenic mice accelerates early cerebral amyloidosis and cognitive deficits

PPARγ/RXRα-induced and CD36-mediated microglial amyloid-β phagocytosis results in cognitive improvement in amyloid precursor protein/presenilin 1 mice

Microglia-derived ASC specks cross-seed amyloid-β in Alzheimer’s disease

Imaging microglial activation and glucose consumption in a mouse model of Alzheimer's disease

Truncated and modified amyloid-beta species

PPARs in Alzheimer's Disease

Dysregulation of TLR5 and TAM Ligands in the Alzheimer’s Brain as Contributors to Disease Progression

CXCR3 promotes plaque formation and behavioral deficits in an Alzheimer's disease model

SFRP1 modulates astrocyte‐to‐microglia crosstalk in acute and chronic neuroinflammation

Critical role of astroglial apolipoprotein E and liver X receptor-α expression for microglial Aβ phagocytosis

Nitric Oxide Decreases the Enzymatic Activity of Insulin Degrading Enzyme in APP/PS1 Mice

Postoperative Ileus Involves Interleukin-1 Receptor Signaling in Enteric Glia

Mrp14 deficiency ameliorates amyloid β burden by increasing microglial phagocytosis and modulation of amyloid precursor protein processing

Variability and temporal dynamics of novel object recognition in aging male C57BL/6 mice

Landscape of brain myeloid cell transcriptome along the spatiotemporal progression of Alzheimer’s disease reveals distinct sequential responses to Aβ and tau

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