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A new glucose-6-phosphate dehydrogenase variant, G6PD Orissa (44 Ala→Gly), is the major polymorphic variant in tribal populations in India

American journal of human genetics, 1995-12, Vol.57 (6), p.1335-1341

1995

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Three major G6PD‐deficient polymorphic variants identified among the Mauritian population

British journal of haematology, 1999-03, Vol.104 (4), p.849-854

1999

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Clinical and haematological consequences of recurrent G6PD mutations and a single new mutation causing chronic nonspherocytic haemolytic anaemia

British journal of haematology, 1998-06, Vol.101 (4), p.670-675

1998

Volltextzugriff (PDF)

Clinical Decision Making in Chronic Myeloid Leukemia Based on Polymerase Chain Reaction Analysis of Minimal Residual Disease

Blood, 1999-08, Vol.94 (4), p.1484-1486

1999

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Preimplantation genetic diagnosis of β-thalassaemia major

The Lancet (British edition), 1996-06, Vol.347 (9016), p.1696-1696

1996

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New Glucose-6-Phosphate Dehydrogenase Mutations Associated With Chronic Anemia

Blood, 1995-03, Vol.85 (5), p.1377-1380

1995

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Severe hemolytic anemia associated with the homozygous state for an unstable hemoglobin variant (Hb Bushwick)

Blood, 1995-09, Vol.86 (5), p.1977-1982

1995

Volltextzugriff (PDF)

How and why minimal residual disease studies are necessary in leukemia: a review from WP10 and WP12 of the European LeukaemiaNet

Haematologica (Roma), 2009-08, Vol.94 (8), p.1135-1150

2009

Volltextzugriff (PDF)

Management of pregnancy when maternal blood has a very high level of fetal haemoglobin

British journal of haematology, 1994-10, Vol.88 (2), p.432-434

1994

Volltextzugriff (PDF)

Phase I/II trial of adding semisynthetic homoharringtonine in chronic myeloid leukemia patients who have achieved partial or complete cytogenetic response on imatinib

Cancer, 2005-05, Vol.103 (9), p.1850-1855

2005

Volltextzugriff (PDF)

Single-cell analysis based dissection of clonality in myelofibrosis

Nature communications, 2020-01, Vol.11 (1), p.73-73, Article 73

2020

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Rationale for the recommendations for harmonizing current methodology for detecting BCR-ABL transcripts in patients with chronic myeloid leukaemia

Leukemia, 2006-11, Vol.20 (11), p.1925-1930

2006

Volltextzugriff (PDF)

The presence of a BCR-ABL mutant allele in CML does not always explain clinical resistance to imatinib

Leukemia, 2006-04, Vol.20 (4), p.658-663

2006

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Comparison of single-dose and escalating-dose regimens of donor lymphocyte infusion for relapse after allografting for chronic myeloid leukemia

Blood, 2000, Vol.95 (1), p.67-71

2000

Volltextzugriff (PDF)

Durability of responses following donor lymphocyte infusions for patients who relapse after allogeneic stem cell transplantation for chronic myeloid leukemia

Blood, 2000-10, Vol.96 (8), p.2712-2716

2000

Volltextzugriff (PDF)

Early detection of BCR-ABL transcripts by quantitative reverse transcriptase–polymerase chain reaction predicts outcome after allogeneic stem cell transplantation for chronic myeloid leukemia

Blood, 2001-03, Vol.97 (6), p.1560-1565

2001

Volltextzugriff (PDF)

Comparison of single-dose and escalating-dose regimens of donor lymphocyte infusion for relapse after allografting for chronic myeloid leukemia

Blood, 2000-01, Vol.95 (1), p.67-71

2000

Volltextzugriff (PDF)

Monitoring patients in complete cytogenetic remission after treatment of CML in chronic phase with imatinib: patterns of residual leukaemia and prognostic factors for cytogenetic relapse

Leukemia, 2005-04, Vol.19 (4), p.507-512

2005

Volltextzugriff (PDF)

A pilot study of continuous imatinib vs pulsed imatinib with or without G-CSF in CML patients who have achieved a complete cytogenetic response

Leukemia, 2009-06, Vol.23 (6), p.1199-1201

2009

Volltextzugriff (PDF)

Detection of Wilms' Tumor Antigen–Specific CTL in Tumor-Draining Lymph Nodes of Patients with Early Breast Cancer

Clinical cancer research, 2006-01, Vol.12 (1), p.34-42

2006

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Response to donor lymphocyte infusions for chronic myeloid leukemia is dose-dependent: the importance of escalating the cell dose to maximize therapeutic efficacy

Leukemia, 2007-05, Vol.21 (5), p.943-948

2007

Volltextzugriff (PDF)

Fusion of H4/D10S170 to the platelet-derived growth factor receptor β in BCR-ABL-negative myeloproliferative disorders with a t(5;10)(q33;q21)

Cancer research (Chicago, Ill.), 2000-07, Vol.60 (13), p.3592-3598

2000

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A new glucose-6-phosphate dehydrogenase variant, G6PD Orissa (44 Ala→Gly), is the major polymorphic variant in tribal populations in India

Three major G6PD‐deficient polymorphic variants identified among the Mauritian population

Clinical and haematological consequences of recurrent G6PD mutations and a single new mutation causing chronic nonspherocytic haemolytic anaemia

Clinical Decision Making in Chronic Myeloid Leukemia Based on Polymerase Chain Reaction Analysis of Minimal Residual Disease

Preimplantation genetic diagnosis of β-thalassaemia major

New Glucose-6-Phosphate Dehydrogenase Mutations Associated With Chronic Anemia

Severe hemolytic anemia associated with the homozygous state for an unstable hemoglobin variant (Hb Bushwick)

How and why minimal residual disease studies are necessary in leukemia: a review from WP10 and WP12 of the European LeukaemiaNet

Management of pregnancy when maternal blood has a very high level of fetal haemoglobin

Phase I/II trial of adding semisynthetic homoharringtonine in chronic myeloid leukemia patients who have achieved partial or complete cytogenetic response on imatinib

Long-term prognostic significance of early molecular response to imatinib in newly diagnosed chronic myeloid leukemia: an analysis from the International Randomized Study of Interferon and STI571 (IRIS)

Single-cell analysis based dissection of clonality in myelofibrosis

Rationale for the recommendations for harmonizing current methodology for detecting BCR-ABL transcripts in patients with chronic myeloid leukaemia

The presence of a BCR-ABL mutant allele in CML does not always explain clinical resistance to imatinib

Monitoring CML patients responding to treatment with tyrosine kinase inhibitors: review and recommendations for harmonizing current methodology for detecting BCR-ABL transcripts and kinase domain mutations and for expressing results

Comparison of single-dose and escalating-dose regimens of donor lymphocyte infusion for relapse after allografting for chronic myeloid leukemia

Durability of responses following donor lymphocyte infusions for patients who relapse after allogeneic stem cell transplantation for chronic myeloid leukemia

GFI1(36N) as a therapeutic and prognostic marker for myelodysplastic syndrome

Early detection of BCR-ABL transcripts by quantitative reverse transcriptase–polymerase chain reaction predicts outcome after allogeneic stem cell transplantation for chronic myeloid leukemia

Comparison of single-dose and escalating-dose regimens of donor lymphocyte infusion for relapse after allografting for chronic myeloid leukemia

Monitoring patients in complete cytogenetic remission after treatment of CML in chronic phase with imatinib: patterns of residual leukaemia and prognostic factors for cytogenetic relapse

A pilot study of continuous imatinib vs pulsed imatinib with or without G-CSF in CML patients who have achieved a complete cytogenetic response

Detection of Wilms' Tumor Antigen–Specific CTL in Tumor-Draining Lymph Nodes of Patients with Early Breast Cancer

Response to donor lymphocyte infusions for chronic myeloid leukemia is dose-dependent: the importance of escalating the cell dose to maximize therapeutic efficacy

Fusion of H4/D10S170 to the platelet-derived growth factor receptor β in BCR-ABL-negative myeloproliferative disorders with a t(5;10)(q33;q21)

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