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Proteostasis in Cerebral Small Vessel Disease

Frontiers in neuroscience, 2019-11, Vol.13, p.1142-1142

Haffner, Christof

2019

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Cerebral small vessel disease-related protease HtrA1 processes latent TGF-β binding protein 1 and facilitates TGF-β signaling

Proceedings of the National Academy of Sciences - PNAS, 2014-11, Vol.111 (46), p.16496-16501

2014

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Histone Deacetylase 9 Activates IKK to Regulate Atherosclerotic Plaque Vulnerability

Circulation research, 2020-08, Vol.127 (6), p.811-823

2020

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Whole-exome sequencing reveals a role of HTRA1 and EGFL8 in brain white matter hyperintensities

Brain (London, England : 1878), 2021-10, Vol.144 (9), p.2670-2682

2021

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Inhaled nitric oxide suppresses neuroinflammation in experimental ischemic stroke

Journal of neuroinflammation, 2023-12, Vol.20 (1), p.301-301, Article 301

2023

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Co-aggregate formation of CADASIL-mutant NOTCH3: a single-particle analysis

Human molecular genetics, 2011-08, Vol.20 (16), p.3256-3265

2011

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Proteomic profiling in cerebral amyloid angiopathy reveals an overlap with CADASIL highlighting accumulation of HTRA1 and its substrates

Acta neuropathologica communications, 2022-01, Vol.10 (1), p.6-6, Article 6

2022

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The emerging role of the HTRA1 protease in brain microvascular disease

Frontiers in dementia, 2023-04, Vol.2, p.1146055

Haffner, Christof

2023

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A novel HTRA1 exon 2 mutation causes loss of protease activity in a Pakistani CARASIL patient

Journal of neurology, 2015-05, Vol.262 (5), p.1369-1372

2015

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Reply to Liu et al: Loss of TGF-β signaling in CARASIL pathogenesis

Proceedings of the National Academy of Sciences - PNAS, 2015-04, Vol.112 (14), p.E1694-E1694

2015

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Transmembrane Protein 147 (TMEM147) Is a Novel Component of the Nicalin-NOMO Protein Complex

The Journal of biological chemistry, 2010-08, Vol.285 (34), p.26174-26181

2010

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Differential Localization and Identification of a Critical Aspartate Suggest Non-redundant Proteolytic Functions of the Presenilin Homologues SPPL2b and SPPL3

The Journal of biological chemistry, 2005-11, Vol.280 (47), p.39515-39523

2005

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Genetic factors in cerebral small vessel disease and their impact on stroke and dementia

Journal of Cerebral Blood Flow & Metabolism, 2016-01, Vol.36 (1), p.158-171

2016

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The Nicastrin-like Protein Nicalin Regulates Assembly and Stability of the Nicalin-Nodal Modulator (NOMO) Membrane Protein Complex

The Journal of biological chemistry, 2007-04, Vol.282 (14), p.10632-10638

2007

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The Function of Plakophilin 1 in Desmosome Assembly and Actin Filament Organization

The Journal of cell biology, 2000-04, Vol.149 (1), p.209-222

2000

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Nicalin and its binding partner Nomo are novel Nodal signaling antagonists

The EMBO journal, 2004-08, Vol.23 (15), p.3041-3050

2004

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The choroid plexus is a key cerebral invasion route for T cells after stroke

Acta neuropathologica, 2017-12, Vol.134 (6), p.851-868

2017

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CADASIL, CARASIL, CARASAL: The linguistic subtleties of cerebral small vessel disease

Neurology, 2016-10, Vol.87 (17), p.1752-1753

2016

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Heterozygous HTRA1 mutations are associated with autosomal dominant cerebral small vessel disease

Brain (London, England : 1878), 2015-08, Vol.138 (Pt 8), p.2347-2358

2015

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Synaptojanin 2, a Novel Synaptojanin Isoform with a Distinct Targeting Domain and Expression Pattern

The Journal of biological chemistry, 1997-12, Vol.272 (49), p.30817-30821

1997

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The Atherosclerosis Risk Variant rs2107595 Mediates Allele-Specific Transcriptional Regulation of HDAC9 via E2F3 and Rb1

Stroke (1970), 2019-10, Vol.50 (10), p.2651-2660

2019

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CADASIL brain vessels show a HTRA1 loss-of-function profile

Acta neuropathologica, 2018-07, Vol.136 (1), p.111-125

2018

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Response to Letter Regarding Article, “Cysteine-Sparing CADASIL Mutations in NOTCH3 Show Proaggregatory Properties In Vitro”

Stroke (1970), 2015-06, Vol.46 (6), p.e154-e154

2015

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Deficiency of the Stroke Relevant HDAC9 Gene Attenuates Atherosclerosis in Accord With Allele-Specific Effects at 7p21.1

Stroke (1970), 2015-01, Vol.46 (1), p.197-202

2015

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Brain-released alarmins and stress response synergize in accelerating atherosclerosis progression after stroke

Science translational medicine, 2018-03, Vol.10 (432)

2018

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Proteostasis in Cerebral Small Vessel Disease

Cerebral small vessel disease-related protease HtrA1 processes latent TGF-β binding protein 1 and facilitates TGF-β signaling

Histone Deacetylase 9 Activates IKK to Regulate Atherosclerotic Plaque Vulnerability

Whole-exome sequencing reveals a role of HTRA1 and EGFL8 in brain white matter hyperintensities

Inhaled nitric oxide suppresses neuroinflammation in experimental ischemic stroke

Co-aggregate formation of CADASIL-mutant NOTCH3: a single-particle analysis

Proteomic profiling in cerebral amyloid angiopathy reveals an overlap with CADASIL highlighting accumulation of HTRA1 and its substrates

The emerging role of the HTRA1 protease in brain microvascular disease

A novel HTRA1 exon 2 mutation causes loss of protease activity in a Pakistani CARASIL patient

Reply to Liu et al: Loss of TGF-β signaling in CARASIL pathogenesis

Transmembrane Protein 147 (TMEM147) Is a Novel Component of the Nicalin-NOMO Protein Complex

Differential Localization and Identification of a Critical Aspartate Suggest Non-redundant Proteolytic Functions of the Presenilin Homologues SPPL2b and SPPL3

Genetic factors in cerebral small vessel disease and their impact on stroke and dementia

The Nicastrin-like Protein Nicalin Regulates Assembly and Stability of the Nicalin-Nodal Modulator (NOMO) Membrane Protein Complex

The Function of Plakophilin 1 in Desmosome Assembly and Actin Filament Organization

Nicalin and its binding partner Nomo are novel Nodal signaling antagonists

The choroid plexus is a key cerebral invasion route for T cells after stroke

CADASIL, CARASIL, CARASAL: The linguistic subtleties of cerebral small vessel disease

Heterozygous HTRA1 mutations are associated with autosomal dominant cerebral small vessel disease

Synaptojanin 2, a Novel Synaptojanin Isoform with a Distinct Targeting Domain and Expression Pattern

The Atherosclerosis Risk Variant rs2107595 Mediates Allele-Specific Transcriptional Regulation of HDAC9 via E2F3 and Rb1

CADASIL brain vessels show a HTRA1 loss-of-function profile

Response to Letter Regarding Article, “Cysteine-Sparing CADASIL Mutations in NOTCH3 Show Proaggregatory Properties In Vitro”

Deficiency of the Stroke Relevant HDAC9 Gene Attenuates Atherosclerosis in Accord With Allele-Specific Effects at 7p21.1

Brain-released alarmins and stress response synergize in accelerating atherosclerosis progression after stroke

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