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Metabolic energy sensing by mammalian CLC anion/proton exchangers

EMBO reports, 2020-06, Vol.21 (6), p.e47872-n/a

2020

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Multiple Discrete Transitions Underlie Voltage-Dependent Activation in CLC Cl−/H+ Antiporters

Biophysical journal, 2014-09, Vol.107 (6), p.L13-L15

2014

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ClC‑3 Is an Intracellular Chloride/Proton Exchanger with Large Voltage-Dependent Nonlinear Capacitance

ACS chemical neuroscience, 2013-06, Vol.4 (6), p.994-1003

2013

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Glutamate 268 Regulates Transport Probability of the Anion/Proton Exchanger ClC-5

The Journal of biological chemistry, 2012-03, Vol.287 (11), p.8101-8109

2012

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Channel-like slippage modes in the human anion/proton exchanger ClC-4

The Journal of general physiology, 2009-05, Vol.133 (5), p.485-496

2009

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Mutations associated with Dent's disease affect gating and voltage dependence of the human anion/proton exchanger ClC-5

Frontiers in physiology, 2015, Vol.6, p.159-159

Alekov, Alexi K

2015

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Barttin Regulates the Subcellular Localization and Posttranslational Modification of Human Cl - /H + Antiporter ClC-5

Frontiers in physiology, 2018-10, Vol.9, p.1490-1490

2018

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Involvement of ClC-3 chloride/proton exchangers in controlling glutamatergic synaptic strength in cultured hippocampal neurons

Frontiers in cellular neuroscience, 2014-05, Vol.8, p.143-143

2014

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Mutations in CLCN2 encoding a voltage-gated chloride channel are associated with idiopathic generalized epilepsies

Nature genetics, 2003-04, Vol.33 (4), p.527-532

2003

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Anion- and Proton-Dependent Gating of ClC-4 Anion/Proton Transporter under Uncoupling Conditions

Biophysical journal, 2011-03, Vol.100 (5), p.1233-1241

2011

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Barttin modulates trafficking and function of ClC-K channels

Proceedings of the National Academy of Sciences - PNAS, 2006-07, Vol.103 (30), p.11411-11416

2006

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Anion Channels: Regulation of ClC-3 by an Orphan Second Messenger

Current biology, 2008-11, Vol.18 (22), p.R1061-R1064

2008

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Gating of human ClC-2 chloride channels and regulation by carboxy-terminal domains

The Journal of physiology, 2008-11, Vol.586 (22), p.5325-5336

2008

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CLCN2 variants in idiopathic generalized epilepsy

Nature genetics, 2009-09, Vol.41 (9), p.954-955

2009

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C-terminal interaction of KCNQ2 and KCNQ3 K+ channels

The Journal of physiology, 2003-04, Vol.548 (2), p.353-360

2003

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A sodium channel mutation causing epilepsy in man exhibits subtle defects in fast inactivation and activation in vitro

The Journal of physiology, 2000-12, Vol.529 (3), p.533-540

2000

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Mutations in CLCN2 encoding a voltage-gated chloride channel are associated with idiopathic generalized epilepsies

Nature genetics, 2009-09, Vol.41 (9), p.1043

2009

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Cooperative effect of S4–S5 loops in domains D3 and D4 on fast inactivation of the Na+ channel

The Journal of physiology, 2004-11, Vol.561 (1), p.39-51

2004

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Enhanced inactivation and acceleration of activation of the sodium channel associated with epilepsy in man

The European journal of neuroscience, 2001-06, Vol.13 (11), p.2171-2176

2001

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Modeling of Single Noninactivating Na+ Channels: Evidence for Two Open and Several Fast Inactivated States

Biophysical journal, 2006-05, Vol.90 (10), p.3511-3522

2006

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Molecular Basis of Voltage-Dependent Gating in ClC Transporters

Biophysical journal, 2015-01, Vol.108 (2), p.428a-428a

2015

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A novel CLCN5 pathogenic mutation supports Dent disease with normal endosomal acidification

Human mutation, 2018-08, Vol.39 (8), p.1139-1149

2018

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Early cell death induced by Clostridium difficile TcdB: Uptake and Rac1‐glucosylation kinetics are decisive for cell fate

Cellular microbiology, 2018-10, Vol.20 (10), p.e12865-n/a

2018

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Lysine 210 Regulates Voltage-Dependent Gating of the Anion-Proton Exchanger CLC-5

Biophysical journal, 2012-01, Vol.102 (3), p.521a-521a

2012

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Two mutations in the IV/S4-S5 segment of the human skeletal muscle Na + channel disrupt fast and enhance slow inactivation

Neuroscience letters, 2001-06, Vol.306 (3), p.173-176

2001

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Metabolic energy sensing by mammalian CLC anion/proton exchangers

Multiple Discrete Transitions Underlie Voltage-Dependent Activation in CLC Cl−/H+ Antiporters

ClC‑3 Is an Intracellular Chloride/Proton Exchanger with Large Voltage-Dependent Nonlinear Capacitance

Glutamate 268 Regulates Transport Probability of the Anion/Proton Exchanger ClC-5

Channel-like slippage modes in the human anion/proton exchanger ClC-4

Mutations associated with Dent's disease affect gating and voltage dependence of the human anion/proton exchanger ClC-5

Barttin Regulates the Subcellular Localization and Posttranslational Modification of Human Cl - /H + Antiporter ClC-5

Involvement of ClC-3 chloride/proton exchangers in controlling glutamatergic synaptic strength in cultured hippocampal neurons

Mutations in CLCN2 encoding a voltage-gated chloride channel are associated with idiopathic generalized epilepsies

Anion- and Proton-Dependent Gating of ClC-4 Anion/Proton Transporter under Uncoupling Conditions

Barttin modulates trafficking and function of ClC-K channels

Anion Channels: Regulation of ClC-3 by an Orphan Second Messenger

Gating of human ClC-2 chloride channels and regulation by carboxy-terminal domains

CLCN2 variants in idiopathic generalized epilepsy

C-terminal interaction of KCNQ2 and KCNQ3 K+ channels

A sodium channel mutation causing epilepsy in man exhibits subtle defects in fast inactivation and activation in vitro

Mutations in CLCN2 encoding a voltage-gated chloride channel are associated with idiopathic generalized epilepsies

Cooperative effect of S4–S5 loops in domains D3 and D4 on fast inactivation of the Na+ channel

Enhanced inactivation and acceleration of activation of the sodium channel associated with epilepsy in man

Modeling of Single Noninactivating Na+ Channels: Evidence for Two Open and Several Fast Inactivated States

Molecular Basis of Voltage-Dependent Gating in ClC Transporters

A novel CLCN5 pathogenic mutation supports Dent disease with normal endosomal acidification

Early cell death induced by Clostridium difficile TcdB: Uptake and Rac1‐glucosylation kinetics are decisive for cell fate

Lysine 210 Regulates Voltage-Dependent Gating of the Anion-Proton Exchanger CLC-5

Two mutations in the IV/S4-S5 segment of the human skeletal muscle Na + channel disrupt fast and enhance slow inactivation

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