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Combination Therapy With Histone Deacetylase Inhibitors (HDACi) for the Treatment of Cancer: Achieving the Full Therapeutic Potential of HDACi

Frontiers in oncology, 2018-03, Vol.8, p.92-92

2018

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PARP Inhibitors: Clinical Relevance, Mechanisms of Action and Tumor Resistance

Frontiers in cell and developmental biology, 2020-09, Vol.8, p.564601-564601

2020

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Identification of a BRCA1-mRNA Splicing Complex Required for Efficient DNA Repair and Maintenance of Genomic Stability

Molecular cell, 2014-05, Vol.54 (3), p.445-459

2014

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Nucleophosmin: from structure and function to disease development

BMC molecular biology, 2016-08, Vol.17 (1), p.19-19, Article 19

2016

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Functions and therapeutic roles of exosomes in cancer

Frontiers in oncology, 2014-01, Vol.4, p.127

2014

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Assessing Molecular Docking Tools to Guide Targeted Drug Discovery of CD38 Inhibitors

International journal of molecular sciences, 2020-07, Vol.21 (15), p.5183

2020

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Barrier-to-autointegration factor 1 (Banf1) regulates poly [ADP-ribose] polymerase 1 (PARP1) activity following oxidative DNA damage

Nature communications, 2019-12, Vol.10 (1), p.5501-12, Article 5501

2019

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Deep Learning-Based Pan-Cancer Classification Model Reveals Tissue-of-Origin Specific Gene Expression Signatures

Cancers, 2022-02, Vol.14 (5), p.1185

2022

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The fructose-bisphosphate, Aldolase A (ALDOA), facilitates DNA-PKcs and ATM kinase activity to regulate DNA double-strand break repair

Scientific reports, 2023-09, Vol.13 (1), p.15171-14, Article 15171

2023

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Recent Advances in Cancer Therapy Targeting Proteins Involved in DNA Double-Strand Break Repair

Clinical cancer research, 2009-10, Vol.15 (20), p.6314-6320

2009

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Drug Discovery Approaches Utilizing Three-Dimensional Cell Culture

Assay and drug development technologies, 2016-02, Vol.14 (1), p.19-28

2016

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Epidermal Growth Factor Receptor (EGFR)-Mutated Non-Small-Cell Lung Cancer (NSCLC)

Pharmaceuticals (Basel, Switzerland), 2020-09, Vol.13 (10), p.273

2020

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Cell Metabolism and DNA Repair Pathways: Implications for Cancer Therapy

Frontiers in cell and developmental biology, 2021-03, Vol.9, p.633305

2021

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The Therapeutic Potential of DNA Damage Repair Pathways and Genomic Stability in Lung Cancer

Frontiers in oncology, 2020-07, Vol.10, p.1256-1256

2020

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BRCA1 Deficiency Exacerbates Estrogen-Induced DNA Damage and Genomic Instability

Cancer research (Chicago, Ill.), 2014-05, Vol.74 (10), p.2773-2784

2014

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Phosphorylation of Exo1 modulates homologous recombination repair of DNA double-strand breaks

Nucleic acids research, 2010-04, Vol.38 (6), p.1821-1831

2010

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Epithelial-to-Mesenchymal Transition Enhances Cancer Cell Sensitivity to Cytotoxic Effects of Cold Atmospheric Plasmas in Breast and Bladder Cancer Systems

Cancers, 2021-06, Vol.13 (12), p.2889

2021

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Chemotherapeutic compounds targeting the DNA double-strand break repair pathways: the good, the bad, and the promising

Frontiers in oncology, 2014-01, Vol.4, p.86-86

2014

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The expression and role of the Lem-D proteins Ankle2, Emerin, Lemd2, and TMPO in triple-negative breast cancer cell growth

Frontiers in oncology, 2024-04, Vol.14, p.1222698-1222698

2024

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Redox Regulation in the Base Excision Repair Pathway: Old and New Players as Cancer Therapeutic Targets

Current medicinal chemistry, 2020-01, Vol.27 (12), p.1901-1921

2020

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Tumor Hypoxia Drives Genomic Instability

Frontiers in cell and developmental biology, 2021-03, Vol.9, p.626229-626229

2021

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Human single-stranded DNA binding proteins are essential for maintaining genomic stability

BMC molecular biology, 2013-04, Vol.14 (1), p.9-9, Article 9

2013

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hSSB2 (NABP1) is required for the recruitment of RPA during the cellular response to DNA UV damage

Scientific reports, 2021-10, Vol.11 (1), p.20256-12, Article 20256

2021

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hSSB1 (NABP2/ OBFC2B) is required for the repair of 8-oxo-guanine by the hOGG1-mediated base excision repair pathway

Nucleic acids research, 2015-10, Vol.43 (18), p.8817-8829

2015

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Multiple human single-stranded DNA binding proteins function in genome maintenance: structural, biochemical and functional analysis

Critical reviews in biochemistry and molecular biology, 2009-06, Vol.44 (2-3), p.98-116

2009

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Combination Therapy With Histone Deacetylase Inhibitors (HDACi) for the Treatment of Cancer: Achieving the Full Therapeutic Potential of HDACi

PARP Inhibitors: Clinical Relevance, Mechanisms of Action and Tumor Resistance

Identification of a BRCA1-mRNA Splicing Complex Required for Efficient DNA Repair and Maintenance of Genomic Stability

Nucleophosmin: from structure and function to disease development

Functions and therapeutic roles of exosomes in cancer

Assessing Molecular Docking Tools to Guide Targeted Drug Discovery of CD38 Inhibitors

Barrier-to-autointegration factor 1 (Banf1) regulates poly [ADP-ribose] polymerase 1 (PARP1) activity following oxidative DNA damage

Deep Learning-Based Pan-Cancer Classification Model Reveals Tissue-of-Origin Specific Gene Expression Signatures

The fructose-bisphosphate, Aldolase A (ALDOA), facilitates DNA-PKcs and ATM kinase activity to regulate DNA double-strand break repair

Recent Advances in Cancer Therapy Targeting Proteins Involved in DNA Double-Strand Break Repair

Drug Discovery Approaches Utilizing Three-Dimensional Cell Culture

Epidermal Growth Factor Receptor (EGFR)-Mutated Non-Small-Cell Lung Cancer (NSCLC)

Cell Metabolism and DNA Repair Pathways: Implications for Cancer Therapy

The Therapeutic Potential of DNA Damage Repair Pathways and Genomic Stability in Lung Cancer

BRCA1 Deficiency Exacerbates Estrogen-Induced DNA Damage and Genomic Instability

Phosphorylation of Exo1 modulates homologous recombination repair of DNA double-strand breaks

Epithelial-to-Mesenchymal Transition Enhances Cancer Cell Sensitivity to Cytotoxic Effects of Cold Atmospheric Plasmas in Breast and Bladder Cancer Systems

Chemotherapeutic compounds targeting the DNA double-strand break repair pathways: the good, the bad, and the promising

The expression and role of the Lem-D proteins Ankle2, Emerin, Lemd2, and TMPO in triple-negative breast cancer cell growth

Redox Regulation in the Base Excision Repair Pathway: Old and New Players as Cancer Therapeutic Targets

Tumor Hypoxia Drives Genomic Instability

Human single-stranded DNA binding proteins are essential for maintaining genomic stability

hSSB2 (NABP1) is required for the recruitment of RPA during the cellular response to DNA UV damage

hSSB1 (NABP2/ OBFC2B) is required for the repair of 8-oxo-guanine by the hOGG1-mediated base excision repair pathway

Multiple human single-stranded DNA binding proteins function in genome maintenance: structural, biochemical and functional analysis

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