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Anti-Tumor Effects of Exosomes Derived from Drug-Incubated Permanently Growing Human MSC

International journal of molecular sciences, 2020-10, Vol.21 (19), p.7311

2020

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Altered Tumor Plasticity after Different Cancer Cell Fusions with MSC

International journal of molecular sciences, 2020-11, Vol.21 (21), p.8347

2020

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Potential Role of MSC/Cancer Cell Fusion and EMT for Breast Cancer Stem Cell Formation

Cancers, 2019-09, Vol.11 (10), p.1432

2019

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Acquisition of new tumor cell properties by MSC-derived exosomes

International journal of oncology, 2015-07, Vol.47 (1), p.244-252

2015

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Spontaneous Fusion of MSC with Breast Cancer Cells Can Generate Tumor Dormancy

International journal of molecular sciences, 2021-05, Vol.22 (11), p.5930

2021

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Effect of SSRI exposure on the proliferation rate and glucose uptake in breast and ovary cancer cell lines

Scientific reports, 2021-01, Vol.11 (1), p.1250-1250, Article 1250

2021

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Cancer stem cell niche models and contribution by mesenchymal stroma/stem cells

Molecular cancer, 2017-02, Vol.16 (1), p.28-28, Article 28

2017

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Impact of the Tumor Microenvironment on Tumor Heterogeneity and Consequences for Cancer Cell Plasticity and Stemness

Cancers, 2020-12, Vol.12 (12), p.3716

2020

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In Vitro Fusion of Normal and Neoplastic Breast Epithelial Cells with Human Mesenchymal Stroma/Stem Cells Partially Involves Tumor Necrosis Factor Receptor Signaling

Stem cells (Dayton, Ohio), 2018-07, Vol.36 (7), p.977-989

2018

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MSC-Derived Extracellular Vesicles in Tumors and Therapy

Cancers, 2021-10, Vol.13 (20), p.5212

2021

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Taxol-Loaded MSC-Derived Exosomes Provide a Therapeutic Vehicle to Target Metastatic Breast Cancer and Other Carcinoma Cells

Cancers, 2019-06, Vol.11 (6), p.798

2019

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Enhanced metastatic capacity of breast cancer cells after interaction and hybrid formation with mesenchymal stroma/stem cells (MSC)

Cell communication and signaling, 2018-01, Vol.16 (1), p.2-2, Article 2

2018

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The Intimate Relationship Among EMT, MET and TME: A T(ransdifferentiation) E(nhancing) M(ix) to Be Exploited for Therapeutic Purposes

Cancers, 2020-12, Vol.12 (12), p.3674

2020

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MSC stimulate ovarian tumor growth during intercellular communication but reduce tumorigenicity after fusion with ovarian cancer cells

Cell communication and signaling, 2018-10, Vol.16 (1), p.67-67, Article 67

2018

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In Vivo Cell Fusion between Mesenchymal Stroma/Stem-Like Cells and Breast Cancer Cells

Cancers, 2019-02, Vol.11 (2), p.185

2019

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Involvement of Actin Cytoskeletal Components in Breast Cancer Cell Fusion with Human Mesenchymal Stroma/Stem-Like Cells

International journal of molecular sciences, 2019-02, Vol.20 (4), p.876

2019

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Cancer Cell Fusion and Post-Hybrid Selection Process (PHSP)

Cancers, 2021-09, Vol.13 (18), p.4636

2021

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Hybrid Formation and Fusion of Cancer Cells In Vitro and In Vivo

Cancers, 2021-09, Vol.13 (17), p.4496

2021

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Human MSC-Derived Exosomes Reduce Cellular Senescence in Renal Epithelial Cells

International journal of molecular sciences, 2021-12, Vol.22 (24), p.13562

2021

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Breast Carcinoma: From Initial Tumor Cell Detachment to Settlement at Secondary Sites

BioMed research international, 2017-01, Vol.2017 (2017), p.1-11

2017

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The role of TGF-β and its crosstalk with RAC1/RAC1b signaling in breast and pancreas carcinoma

Cell communication and signaling, 2017-05, Vol.15 (1), p.19-19, Article 19

2017

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TGF-β-Dependent Growth Arrest and Cell Migration in Benign and Malignant Breast Epithelial Cells Are Antagonistically Controlled by Rac1 and Rac1b

International journal of molecular sciences, 2017-07, Vol.18 (7), p.1574

2017

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Reversible Growth-Arrest of a Spontaneously-Derived Human MSC-Like Cell Line

International journal of molecular sciences, 2020-07, Vol.21 (13), p.4752

2020

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Changes in uPA, PAI-1, and TGF-β Production during Breast Cancer Cell Interaction with Human Mesenchymal Stroma/Stem-Like Cells (MSC)

International journal of molecular sciences, 2019-05, Vol.20 (11), p.2630

2019

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SCCOHT tumors acquire chemoresistance and protection by interacting mesenchymal stroma/stem cells within the tumor microenvironment

International journal of oncology, 2016-12, Vol.49 (6), p.2453-2463

2016

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Anti-Tumor Effects of Exosomes Derived from Drug-Incubated Permanently Growing Human MSC

Altered Tumor Plasticity after Different Cancer Cell Fusions with MSC

Potential Role of MSC/Cancer Cell Fusion and EMT for Breast Cancer Stem Cell Formation

Acquisition of new tumor cell properties by MSC-derived exosomes

Spontaneous Fusion of MSC with Breast Cancer Cells Can Generate Tumor Dormancy

Effect of SSRI exposure on the proliferation rate and glucose uptake in breast and ovary cancer cell lines

Cancer stem cell niche models and contribution by mesenchymal stroma/stem cells

Impact of the Tumor Microenvironment on Tumor Heterogeneity and Consequences for Cancer Cell Plasticity and Stemness

In Vitro Fusion of Normal and Neoplastic Breast Epithelial Cells with Human Mesenchymal Stroma/Stem Cells Partially Involves Tumor Necrosis Factor Receptor Signaling

MSC-Derived Extracellular Vesicles in Tumors and Therapy

Taxol-Loaded MSC-Derived Exosomes Provide a Therapeutic Vehicle to Target Metastatic Breast Cancer and Other Carcinoma Cells

Enhanced metastatic capacity of breast cancer cells after interaction and hybrid formation with mesenchymal stroma/stem cells (MSC)

The Intimate Relationship Among EMT, MET and TME: A T(ransdifferentiation) E(nhancing) M(ix) to Be Exploited for Therapeutic Purposes

MSC stimulate ovarian tumor growth during intercellular communication but reduce tumorigenicity after fusion with ovarian cancer cells

In Vivo Cell Fusion between Mesenchymal Stroma/Stem-Like Cells and Breast Cancer Cells

Involvement of Actin Cytoskeletal Components in Breast Cancer Cell Fusion with Human Mesenchymal Stroma/Stem-Like Cells

Cancer Cell Fusion and Post-Hybrid Selection Process (PHSP)

Hybrid Formation and Fusion of Cancer Cells In Vitro and In Vivo

Human MSC-Derived Exosomes Reduce Cellular Senescence in Renal Epithelial Cells

Breast Carcinoma: From Initial Tumor Cell Detachment to Settlement at Secondary Sites

The role of TGF-β and its crosstalk with RAC1/RAC1b signaling in breast and pancreas carcinoma

TGF-β-Dependent Growth Arrest and Cell Migration in Benign and Malignant Breast Epithelial Cells Are Antagonistically Controlled by Rac1 and Rac1b

Reversible Growth-Arrest of a Spontaneously-Derived Human MSC-Like Cell Line

Changes in uPA, PAI-1, and TGF-β Production during Breast Cancer Cell Interaction with Human Mesenchymal Stroma/Stem-Like Cells (MSC)

SCCOHT tumors acquire chemoresistance and protection by interacting mesenchymal stroma/stem cells within the tumor microenvironment

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