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Neuroinflammatory contributions to pain after SCI: Roles for central glial mechanisms and nociceptor-mediated host defense

Experimental neurology, 2014-08, Vol.258, p.48-61

Walters, Edgar T.

2014

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Nociceptive Biology of Molluscs and Arthropods: Evolutionary Clues About Functions and Mechanisms Potentially Related to Pain

Frontiers in physiology, 2018-08, Vol.9, p.1049-1049

Walters, Edgar T

2018

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Exosomes from Glioma-Associated Mesenchymal Stem Cells Increase the Tumorigenicity of Glioma Stem-like Cells via Transfer of miR-1587

Cancer research (Chicago, Ill.), 2017-11, Vol.77 (21), p.5808-5819

2017

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Nociceptive Sensitization Reduces Predation Risk

Current biology, 2014-05, Vol.24 (10), p.1121-1125

2014

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Zinc Inhibits TRPV1 to Alleviate Chemotherapy-Induced Neuropathic Pain

The Journal of neuroscience, 2018-01, Vol.38 (2), p.474-483

2018

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Nociceptors as chronic drivers of pain and hyperreflexia after spinal cord injury: an adaptive-maladaptive hyperfunctional state hypothesis

Frontiers in physiology, 2012-01, Vol.3, p.309-309

Walters, Edgar T

2012

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Induction of Monocyte Chemoattractant Protein-1 (MCP-1) and Its Receptor CCR2 in Primary Sensory Neurons Contributes to Paclitaxel-Induced Peripheral Neuropathy

The journal of pain, 2013-10, Vol.14 (10), p.1031-1044

2013

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Determinants of TRPV4 activity following selective activation by small molecule agonist GSK1016790A

PloS one, 2011-02, Vol.6 (2), p.e16713-e16713

2011

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TRPV1 channels make major contributions to behavioral hypersensitivity and spontaneous activity in nociceptors after spinal cord injury

Pain (Amsterdam), 2013-10, Vol.154 (10), p.2130-2141

2013

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Adaptive mechanisms driving maladaptive pain: how chronic ongoing activity in primary nociceptors can enhance evolutionary fitness after severe injury

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 2019-11, Vol.374 (1785), p.20190277

Walters, Edgar T

2019

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Retinoids activate the irritant receptor TRPV1 and produce sensory hypersensitivity

The Journal of clinical investigation, 2013-09, Vol.123 (9), p.3941-3951

2013

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Sustained Elevated Adenosine via ADORA2B Promotes Chronic Pain through Neuro-immune Interaction

Cell reports (Cambridge), 2016-06, Vol.16 (1), p.106-119

2016

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Neuroethology: Self-Recognition Helps Octopuses Avoid Entanglement

Current biology, 2014-06, Vol.24 (11), p.R520-R521

2014

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Exaptation and Evolutionary Adaptation in Nociceptor Mechanisms Driving Persistent Pain

Brain, behavior and evolution, 2023, Vol.98 (6), p.314-330

Walters, Edgar T.

2023

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How is chronic pain related to sympathetic dysfunction and autonomic dysreflexia following spinal cord injury?

Autonomic neuroscience, 2018-01, Vol.209, p.79-89

Walters, Edgar T

2018

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Molecular and cellular mechanisms that initiate pain and itch

Cellular and Molecular Life Sciences, 2015-09, Vol.72 (17), p.3201-3223

2015

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EPAC1 and EPAC2 promote nociceptor hyperactivity associated with chronic pain after spinal cord injury

Neurobiology of pain, 2020-01, Vol.7, p.100040-100040, Article 100040

2020

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Evolution of mechanisms and behaviour important for pain

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 2019-11, Vol.374 (1785), p.20190275

2019

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Readiness of nociceptor cell bodies to generate spontaneous activity results from background activity of diverse ion channels and high input resistance

Pain (Amsterdam), 2024-04, Vol.165 (4), p.893-907

2024

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Targeting Pain-evoking Transient Receptor Potential Channels for the Treatment of Pain

Current neuropharmacology, 2013-12, Vol.11 (6), p.652-663

2013

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Interleukin-10 resolves pain hypersensitivity induced by cisplatin by reversing sensory neuron hyperexcitability

Pain (Amsterdam), 2020-10, Vol.161 (10), p.2344-2352

2020

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Strong inferences about pain in invertebrates require stronger evidence

Animal sentience, 2022-01, Vol.7 (32), p.1

Walters, Edgar T

2022

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Depolarization-Dependent C-Raf Signaling Promotes Hyperexcitability and Reduces Opioid Sensitivity of Isolated Nociceptors after Spinal Cord Injury

The Journal of neuroscience, 2020-08, Vol.40 (34), p.6522-6535

2020

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Persistent nociceptor hyperactivity as a painful evolutionary adaptation

Trends in neurosciences (Regular ed.), 2023-03, Vol.46 (3), p.211-227

2023

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Oral Dimethyl Fumarate Reduces Peripheral Neuropathic Pain in Rodents via NFE2L2 Antioxidant Signaling

Anesthesiology (Philadelphia), 2020-02, Vol.132 (2), p.343-356

2020

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Neuroinflammatory contributions to pain after SCI: Roles for central glial mechanisms and nociceptor-mediated host defense

Nociceptive Biology of Molluscs and Arthropods: Evolutionary Clues About Functions and Mechanisms Potentially Related to Pain

Exosomes from Glioma-Associated Mesenchymal Stem Cells Increase the Tumorigenicity of Glioma Stem-like Cells via Transfer of miR-1587

Nociceptive Sensitization Reduces Predation Risk

Zinc Inhibits TRPV1 to Alleviate Chemotherapy-Induced Neuropathic Pain

Nociceptors as chronic drivers of pain and hyperreflexia after spinal cord injury: an adaptive-maladaptive hyperfunctional state hypothesis

Induction of Monocyte Chemoattractant Protein-1 (MCP-1) and Its Receptor CCR2 in Primary Sensory Neurons Contributes to Paclitaxel-Induced Peripheral Neuropathy

Determinants of TRPV4 activity following selective activation by small molecule agonist GSK1016790A

TRPV1 channels make major contributions to behavioral hypersensitivity and spontaneous activity in nociceptors after spinal cord injury

Adaptive mechanisms driving maladaptive pain: how chronic ongoing activity in primary nociceptors can enhance evolutionary fitness after severe injury

Retinoids activate the irritant receptor TRPV1 and produce sensory hypersensitivity

Sustained Elevated Adenosine via ADORA2B Promotes Chronic Pain through Neuro-immune Interaction

Neuroethology: Self-Recognition Helps Octopuses Avoid Entanglement

Exaptation and Evolutionary Adaptation in Nociceptor Mechanisms Driving Persistent Pain

How is chronic pain related to sympathetic dysfunction and autonomic dysreflexia following spinal cord injury?

Molecular and cellular mechanisms that initiate pain and itch

EPAC1 and EPAC2 promote nociceptor hyperactivity associated with chronic pain after spinal cord injury

Evolution of mechanisms and behaviour important for pain

Readiness of nociceptor cell bodies to generate spontaneous activity results from background activity of diverse ion channels and high input resistance

Targeting Pain-evoking Transient Receptor Potential Channels for the Treatment of Pain

Interleukin-10 resolves pain hypersensitivity induced by cisplatin by reversing sensory neuron hyperexcitability

Strong inferences about pain in invertebrates require stronger evidence

Depolarization-Dependent C-Raf Signaling Promotes Hyperexcitability and Reduces Opioid Sensitivity of Isolated Nociceptors after Spinal Cord Injury

Persistent nociceptor hyperactivity as a painful evolutionary adaptation

Oral Dimethyl Fumarate Reduces Peripheral Neuropathic Pain in Rodents via NFE2L2 Antioxidant Signaling

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