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NFAT1 Directly Regulates IL8 and MMP3 to Promote Melanoma Tumor Growth and Metastasis

Cancer research (Chicago, Ill.), 2016-06, Vol.76 (11), p.3145-3155

2016

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Transcriptional control of melanoma metastasis: The importance of the tumor microenvironment

Seminars in cancer biology, 2011-04, Vol.21 (2), p.83-88

2011

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Protease activated receptor-1 inhibits the Maspin tumor-suppressor gene to determine the melanoma metastatic phenotype

Proceedings of the National Academy of Sciences - PNAS, 2011-01, Vol.108 (2), p.626-631

2011

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Silencing cAMP-response Element-binding Protein (CREB) Identifies CYR61 as a Tumor Suppressor Gene in Melanoma

The Journal of biological chemistry, 2009-09, Vol.284 (38), p.26194-26206

2009

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Crosstalk between Protease-activated Receptor 1 and Platelet-activating Factor Receptor Regulates Melanoma Cell Adhesion Molecule (MCAM/MUC18) Expression and Melanoma Metastasis

The Journal of biological chemistry, 2009-10, Vol.284 (42), p.28845-28855

2009

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Overexpression of Protease-Activated Receptor-1 Contributes to Melanoma Metastasis via Regulation of Connexin 43

Cancer research (Chicago, Ill.), 2009-08, Vol.69 (16), p.6730-6737

2009

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Expression of Id-1 Is Regulated by MCAM/MUC18: A Missing Link in Melanoma Progression

Cancer research (Chicago, Ill.), 2011-05, Vol.71 (10), p.3494-3504

2011

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Carbon nanotube capsules enhance the in vivo efficacy of cisplatin

Acta biomaterialia, 2017-08, Vol.58, p.466-478

2017

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PAR-1 and Thrombin: The Ties That Bind the Microenvironment to Melanoma Metastasis

Cancer research (Chicago, Ill.), 2011-11, Vol.71 (21), p.6561-6566

2011

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Mutations Within the Kinase Domain and Truncations of the Epidermal Growth Factor Receptor Are Rare Events in Bladder Cancer: Implications for Therapy

Clinical cancer research, 2006-08, Vol.12 (15), p.4671-4677

2006

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Phosphorylated Epidermal Growth Factor Receptor on Tumor-Associated Endothelial Cells Is a Primary Target for Therapy with Tyrosine Kinase Inhibitors

Neoplasia (New York, N.Y.), 2008-05, Vol.10 (5), p.489-500

2008

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Increased Levels of Stress Induces a More Metastatic Phenotype in Human Melanoma Cells

The FASEB journal, 2022-05, Vol.36 (S1), p.n/a

2022

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Targeting melanoma growth and metastasis with systemic delivery of liposome-incorporated protease-activated receptor-1 small interfering RNA

Cancer research (Chicago, Ill.), 2008-11, Vol.68 (21), p.9078-9086

2008

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CREB Inhibits AP-2α Expression to Regulate the Malignant Phenotype of Melanoma

PloS one, 2010-08, Vol.5 (8), p.e12452

2010

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Protease-Activated Receptors and other G-Protein-Coupled Receptors: the Melanoma Connection

Frontiers in genetics, 2016-06, Vol.7, p.112-112

2016

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The emerging role of the thrombin receptor (PAR-1) in melanoma metastasis--a possible therapeutic target

Oncotarget, 2011-01, Vol.2 (1-2), p.8-17

2011

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Tumor Immunotherapy in Melanoma: Strategies for Overcoming Mechanisms of Resistance and Escape

American journal of clinical dermatology, 2008-01, Vol.9 (5), p.307-311

2008

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Emerging Roles of PAR-1 and PAFR in Melanoma Metastasis

Cancer microenvironment, 2008-12, Vol.1 (1), p.103-111

2008

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Stress Promotes a More Invasive Phenotype in Human Melanoma Cells

The FASEB journal, 2020-04, Vol.34 (S1), p.1-1

2020

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CREB inhibits AP-2alpha expression to regulate the malignant phenotype of melanoma

PloS one, 2010-08, Vol.5 (8), p.e12452-e12452

2010

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NFAT1 directly regulates IL-8 and MMP-3 to promote melanoma tumor growth and metastasis

Cancer research (Chicago, Ill.), 2016-03, Vol.76 (11), p.3145-3155

2016

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CREB Inhibits AP-2[alpha] Expression to Regulate the Malignant Phenotype of Melanoma

PloS one, 2010-08, Vol.5 (8), p.e12452

2010

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NFAT1 Directly Regulates IL8 and MMP3 to Promote Melanoma Tumor Growth and Metastasis

Transcriptional control of melanoma metastasis: The importance of the tumor microenvironment

Protease activated receptor-1 inhibits the Maspin tumor-suppressor gene to determine the melanoma metastatic phenotype

Silencing cAMP-response Element-binding Protein (CREB) Identifies CYR61 as a Tumor Suppressor Gene in Melanoma

Crosstalk between Protease-activated Receptor 1 and Platelet-activating Factor Receptor Regulates Melanoma Cell Adhesion Molecule (MCAM/MUC18) Expression and Melanoma Metastasis

Overexpression of Protease-Activated Receptor-1 Contributes to Melanoma Metastasis via Regulation of Connexin 43

Expression of Id-1 Is Regulated by MCAM/MUC18: A Missing Link in Melanoma Progression

Carbon nanotube capsules enhance the in vivo efficacy of cisplatin

PAR-1 and Thrombin: The Ties That Bind the Microenvironment to Melanoma Metastasis

Mutations Within the Kinase Domain and Truncations of the Epidermal Growth Factor Receptor Are Rare Events in Bladder Cancer: Implications for Therapy

Src activation by [beta]-adrenoreceptors is a key switch for tumour metastasis

Phosphorylated Epidermal Growth Factor Receptor on Tumor-Associated Endothelial Cells Is a Primary Target for Therapy with Tyrosine Kinase Inhibitors

Increased Levels of Stress Induces a More Metastatic Phenotype in Human Melanoma Cells

Src activation by β-adrenoreceptors is a key switch for tumour metastasis

Targeting melanoma growth and metastasis with systemic delivery of liposome-incorporated protease-activated receptor-1 small interfering RNA

CREB Inhibits AP-2α Expression to Regulate the Malignant Phenotype of Melanoma

Protease-Activated Receptors and other G-Protein-Coupled Receptors: the Melanoma Connection

The emerging role of the thrombin receptor (PAR-1) in melanoma metastasis--a possible therapeutic target

Tumor Immunotherapy in Melanoma: Strategies for Overcoming Mechanisms of Resistance and Escape

Emerging Roles of PAR-1 and PAFR in Melanoma Metastasis

Stress Promotes a More Invasive Phenotype in Human Melanoma Cells

CREB inhibits AP-2alpha expression to regulate the malignant phenotype of melanoma

NFAT1 directly regulates IL-8 and MMP-3 to promote melanoma tumor growth and metastasis

Correction: Corrigendum: Src activation by β-adrenoreceptors is a key switch for tumour metastasis

CREB Inhibits AP-2[alpha] Expression to Regulate the Malignant Phenotype of Melanoma

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