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Mycobacterial Cell Wall: A Source of Successful Targets for Old and New Drugs

Applied sciences, 2020-04, Vol.10 (7), p.2278

Vilchèze, Catherine

2020

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Mycobacterium tuberculosis is extraordinarily sensitive to killing by a vitamin C-induced Fenton reaction

Nature communications, 2013, Vol.4 (1), p.1881-1881, Article 1881

2013

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Auranofin exerts broad-spectrum bactericidal activities by targeting thiol-redox homeostasis

Proceedings of the National Academy of Sciences - PNAS, 2015-04, Vol.112 (14), p.4453-4458

2015

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Vitamin C Potentiates the Killing of Mycobacterium tuberculosis by the First-Line Tuberculosis Drugs Isoniazid and Rifampin in Mice

Antimicrobial agents and chemotherapy, 2018-03, Vol.62 (3)

2018

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Separable roles for Mycobacterium tuberculosis ESX-3 effectors in iron acquisition and virulence

Proceedings of the National Academy of Sciences - PNAS, 2016-01, Vol.113 (3), p.E348-E357

2016

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The mechanism of isoniazid killing : Clarity through the scope of genetics

Annual review of microbiology, 2007-01, Vol.61 (1), p.35-50

2007

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The Combination of Sulfamethoxazole, Trimethoprim, and Isoniazid or Rifampin Is Bactericidal and Prevents the Emergence of Drug Resistance in Mycobacterium tuberculosis

Antimicrobial Agents and Chemotherapy, 2012-10, Vol.56 (10), p.5142-5148

2012

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Altered NADH/NAD+ Ratio Mediates Coresistance to Isoniazid and Ethionamide in Mycobacteria

Antimicrobial Agents and Chemotherapy, 2005-02, Vol.49 (2), p.708-720

2005

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Energetics of Respiration and Oxidative Phosphorylation in Mycobacteria

Microbiology spectrum, 2014-06, Vol.2 (3)

2014

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Mycobacterium tuberculosis Dihydrofolate Reductase Is Not a Target Relevant to the Antitubercular Activity of Isoniazid

Antimicrobial Agents and Chemotherapy, 2010-09, Vol.54 (9), p.3776-3782

2010

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Transfer of a point mutation in Mycobacterium tuberculosis inhA resolves the target of isoniazid

Nature medicine, 2006-09, Vol.12 (9), p.1027-1029

2006

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Sterilization by Adaptive Immunity of a Conditionally Persistent Mutant of Mycobacterium tuberculosis

mBio, 2021-01, Vol.12 (1)

2021

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Keto-mycolic acid-dependent pellicle formation confers tolerance to drug-sensitive Mycobacterium tuberculosis

mBio, , Vol.4 (3), p.e00222

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Phosphorylation of KasB regulates virulence and acid-fastness in Mycobacterium tuberculosis

PLoS pathogens, 2014-05, Vol.10 (5), p.e1004115

2014

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Mycothiol biosynthesis is essential for ethionamide susceptibility in Mycobacterium tuberculosis

Molecular microbiology, 2008-09, Vol.69 (5), p.1316-1329

2008

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Novel Inhibitors of InhA Efficiently Kill Mycobacterium tuberculosis under Aerobic and Anaerobic Conditions

Antimicrobial Agents and Chemotherapy, 2011-08, Vol.55 (8), p.3889-3898

2011

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Loss-of-function mutations in ndh do not confer delamanid, ethionamide, isoniazid, or pretomanid resistance in Mycobacterium tuberculosis

Antimicrobial agents and chemotherapy, 2024-01, Vol.68 (1), p.e0109623

2024

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The Isoniazid Paradigm of Killing, Resistance, and Persistence in Mycobacterium tuberculosis

Journal of molecular biology, 2019-08, Vol.431 (18), p.3450-3461

2019

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Inactivation of the inhA-Encoded Fatty Acid Synthase II (FASII) Enoyl-Acyl Carrier Protein Reductase Induces Accumulation of the FASI End Products and Cell Lysis of Mycobacterium smegmatis

Journal of Bacteriology, 2000-07, Vol.182 (14), p.4059-4067

2000

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Phosphorylation of InhA inhibits mycolic acid biosynthesis and growth of Mycobacterium tuberculosis

Molecular microbiology, 2010-12, Vol.78 (6), p.1591-1605

2010

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Determinants of the Inhibition of DprE1 and CYP2C9 by Antitubercular Thiophenes

Angewandte Chemie (International ed.), 2017-10, Vol.56 (42), p.13011-13015

International ed. in English, 2017

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NAD⁺ auxotrophy is bacteriocidal for the tubercle bacilli

Molecular microbiology, 2010-04, Vol.76 (2), p.365-377

2010

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The Complete Genome Sequence of the Emerging Pathogen Mycobacterium haemophilum Explains Its Unique Culture Requirements

mBio, 2015-11, Vol.6 (6), p.e01313-e01315

2015

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Mycobacterium bovis Strain Ravenel Is Attenuated in Cattle

Pathogens (Basel), 2022-11, Vol.11 (11), p.1330

2022

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Mycobacterial Cell Wall: A Source of Successful Targets for Old and New Drugs

Mycobacterium tuberculosis is extraordinarily sensitive to killing by a vitamin C-induced Fenton reaction

Auranofin exerts broad-spectrum bactericidal activities by targeting thiol-redox homeostasis

Vitamin C Potentiates the Killing of Mycobacterium tuberculosis by the First-Line Tuberculosis Drugs Isoniazid and Rifampin in Mice

Separable roles for Mycobacterium tuberculosis ESX-3 effectors in iron acquisition and virulence

The mechanism of isoniazid killing : Clarity through the scope of genetics

The Combination of Sulfamethoxazole, Trimethoprim, and Isoniazid or Rifampin Is Bactericidal and Prevents the Emergence of Drug Resistance in Mycobacterium tuberculosis

Altered NADH/NAD+ Ratio Mediates Coresistance to Isoniazid and Ethionamide in Mycobacteria

Energetics of Respiration and Oxidative Phosphorylation in Mycobacteria

Mycobacterium tuberculosis Dihydrofolate Reductase Is Not a Target Relevant to the Antitubercular Activity of Isoniazid

Transfer of a point mutation in Mycobacterium tuberculosis inhA resolves the target of isoniazid

Sterilization by Adaptive Immunity of a Conditionally Persistent Mutant of Mycobacterium tuberculosis

Keto-mycolic acid-dependent pellicle formation confers tolerance to drug-sensitive Mycobacterium tuberculosis

Phosphorylation of KasB regulates virulence and acid-fastness in Mycobacterium tuberculosis

Mycothiol biosynthesis is essential for ethionamide susceptibility in Mycobacterium tuberculosis

Novel Inhibitors of InhA Efficiently Kill Mycobacterium tuberculosis under Aerobic and Anaerobic Conditions

Targeting Mycobacterium tuberculosis Tumor Necrosis Factor Alpha-Downregulating Genes for the Development of Antituberculous Vaccines

Loss-of-function mutations in ndh do not confer delamanid, ethionamide, isoniazid, or pretomanid resistance in Mycobacterium tuberculosis

The Isoniazid Paradigm of Killing, Resistance, and Persistence in Mycobacterium tuberculosis

Inactivation of the inhA-Encoded Fatty Acid Synthase II (FASII) Enoyl-Acyl Carrier Protein Reductase Induces Accumulation of the FASI End Products and Cell Lysis of Mycobacterium smegmatis

Phosphorylation of InhA inhibits mycolic acid biosynthesis and growth of Mycobacterium tuberculosis

Determinants of the Inhibition of DprE1 and CYP2C9 by Antitubercular Thiophenes

NAD⁺ auxotrophy is bacteriocidal for the tubercle bacilli

The Complete Genome Sequence of the Emerging Pathogen Mycobacterium haemophilum Explains Its Unique Culture Requirements

Mycobacterium bovis Strain Ravenel Is Attenuated in Cattle

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