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LOTUS as an endogenous protein converting the adult central nervous system environment from nonpermissive to permissive for axonal regrowth after brain injury

Neuropathology, 2020-02, Vol.40 (1), p.14-20

Takei, Kohtaro

2020

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Nogo receptor antagonist LOTUS exerts suppression on axonal growth‐inhibiting receptor PIR‐B

Journal of neurochemistry, 2020-11, Vol.155 (3), p.285-299

2020

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LOTUS, an endogenous Nogo receptor antagonist, is involved in synapse and memory formation

Scientific reports, 2021-03, Vol.11 (1), p.5085-5085, Article 5085

2021

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Blockade of chondroitin sulfate proteoglycans-induced axonal growth inhibition by LOTUS

Neuroscience, 2017-07, Vol.356, p.265-274

2017

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LOTUS suppresses amyloid β-induced dendritic spine elimination through the blockade of amyloid β binding to PirB

Molecular medicine (Cambridge, Mass.), 2022-12, Vol.28 (1), p.154-154

2022

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The soluble form of LOTUS inhibits Nogo receptor type 1-mediated signaling induced by B lymphocyte stimulator and chondroitin sulfate proteoglycans

Neuroscience letters, 2018-09, Vol.683, p.61-68

2018

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LOTUS overexpression accelerates neuronal plasticity after focal brain ischemia in mice

PloS one, 2017-09, Vol.12 (9), p.e0184258-e0184258

2017

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Sex-Specific Protection of Osteoarthritis by Deleting Cartilage Acid Protein 1

PloS one, 2016-07, Vol.11 (7), p.e0159157

2016

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Axonal regeneration and functional recovery driven by endogenous Nogo receptor antagonist LOTUS in a rat model of unilateral pyramidotomy

Experimental neurology, 2020-01, Vol.323, p.113068-113068, Article 113068

2020

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Crmp4 deletion promotes recovery from spinal cord injury by neuroprotection and limited scar formation

Scientific reports, 2015-02, Vol.5 (1), p.8269, Article 8269

2015

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Lateral olfactory tract usher substance (LOTUS) protein, an endogenous Nogo receptor antagonist, converts a non-permissive to permissive brain environment for axonal regrowth

Neural regeneration research, 2018-07, Vol.13 (7), p.1193-1194

2018

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LOTUS suppresses axon growth inhibition by blocking interaction between Nogo receptor-1 and all four types of its ligand

Molecular and cellular neuroscience, 2014-07, Vol.61, p.211-218

2014

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Regulation of axonal regeneration by the level of function of the endogenous Nogo receptor antagonist LOTUS

Scientific reports, 2017-09, Vol.7 (1), p.12119-9, Article 12119

2017

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Cartilage Acidic Protein—1B (LOTUS), an Endogenous Nogo Receptor Antagonist for Axon Tract Formation

Science (American Association for the Advancement of Science), 2011-08, Vol.333 (6043), p.769-773

2011

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Localized role of CRMP1 and CRMP2 in neurite outgrowth and growth cone steering

Developmental neurobiology (Hoboken, N.J.), 2012-12, Vol.72 (12), p.1528-1540

2012

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Axonal branching in lateral olfactory tract is promoted by Nogo signaling

Scientific reports, 2016-12, Vol.6 (1), p.39586-39586, Article 39586

2016

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LOTUS, a potent blocker of Nogo receptor-1 causing inhibition of axonal growth

Neural regeneration research, 2015, Vol.10 (1), p.46-48

2015

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Semaphorin3A Alleviates Skin Lesions and Scratching Behavior in NC/Nga Mice, an Atopic Dermatitis Model

Journal of investigative dermatology, 2008-12, Vol.128 (12), p.2842-2849

2008

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Regulation of Dendritic Branching and Spine Maturation by Semaphorin3A-Fyn Signaling

The Journal of neuroscience, 2006-03, Vol.26 (11), p.2971-2980

2006

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Regulation of Spine Development by Semaphorin3A through Cyclin-Dependent Kinase 5 Phosphorylation of Collapsin Response Mediator Protein 1

The Journal of neuroscience, 2007-11, Vol.27 (46), p.12546-12554

2007

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Lateral olfactory tract usher substance (LOTUS), an endogenous Nogo receptor antagonist, ameliorates disease progression in amyotrophic lateral sclerosis model mice

Cell death discovery, 2023-12, Vol.9 (1), p.454-454, Article 454

2023

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Cerebrospinal fluid level of Nogo receptor 1 antagonist lateral olfactory tract usher substance (LOTUS) correlates inversely with the extent of neuroinflammation

Journal of neuroinflammation, 2018-02, Vol.15 (1), p.46-46, Article 46

2018

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LOTUS Inhibits Neuronal Apoptosis and Promotes Tract Regeneration in Contusive Spinal Cord Injury Model Mice

eNeuro, 2018-09, Vol.5 (5), p.ENEURO.0303-18.2018

2018

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The carboxyl-terminal region of Crtac1B/LOTUS acts as a functional domain in endogenous antagonism to Nogo receptor-1

Biochemical and biophysical research communications, 2012-02, Vol.418 (2), p.390-395

2012

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Collapsin Response Mediator Protein 1 Mediates Reelin Signaling in Cortical Neuronal Migration

The Journal of neuroscience, 2006-12, Vol.26 (51), p.13357-13362

2006

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LOTUS as an endogenous protein converting the adult central nervous system environment from nonpermissive to permissive for axonal regrowth after brain injury

Nogo receptor antagonist LOTUS exerts suppression on axonal growth‐inhibiting receptor PIR‐B

LOTUS, an endogenous Nogo receptor antagonist, is involved in synapse and memory formation

Blockade of chondroitin sulfate proteoglycans-induced axonal growth inhibition by LOTUS

LOTUS suppresses amyloid β-induced dendritic spine elimination through the blockade of amyloid β binding to PirB

The soluble form of LOTUS inhibits Nogo receptor type 1-mediated signaling induced by B lymphocyte stimulator and chondroitin sulfate proteoglycans

LOTUS overexpression accelerates neuronal plasticity after focal brain ischemia in mice

Sex-Specific Protection of Osteoarthritis by Deleting Cartilage Acid Protein 1

Axonal regeneration and functional recovery driven by endogenous Nogo receptor antagonist LOTUS in a rat model of unilateral pyramidotomy

Crmp4 deletion promotes recovery from spinal cord injury by neuroprotection and limited scar formation

Lateral olfactory tract usher substance (LOTUS) protein, an endogenous Nogo receptor antagonist, converts a non-permissive to permissive brain environment for axonal regrowth

LOTUS suppresses axon growth inhibition by blocking interaction between Nogo receptor-1 and all four types of its ligand

Regulation of axonal regeneration by the level of function of the endogenous Nogo receptor antagonist LOTUS

Cartilage Acidic Protein—1B (LOTUS), an Endogenous Nogo Receptor Antagonist for Axon Tract Formation

Localized role of CRMP1 and CRMP2 in neurite outgrowth and growth cone steering

Axonal branching in lateral olfactory tract is promoted by Nogo signaling

LOTUS, a potent blocker of Nogo receptor-1 causing inhibition of axonal growth

Semaphorin3A Alleviates Skin Lesions and Scratching Behavior in NC/Nga Mice, an Atopic Dermatitis Model

Regulation of Dendritic Branching and Spine Maturation by Semaphorin3A-Fyn Signaling

Regulation of Spine Development by Semaphorin3A through Cyclin-Dependent Kinase 5 Phosphorylation of Collapsin Response Mediator Protein 1

Lateral olfactory tract usher substance (LOTUS), an endogenous Nogo receptor antagonist, ameliorates disease progression in amyotrophic lateral sclerosis model mice

Cerebrospinal fluid level of Nogo receptor 1 antagonist lateral olfactory tract usher substance (LOTUS) correlates inversely with the extent of neuroinflammation

LOTUS Inhibits Neuronal Apoptosis and Promotes Tract Regeneration in Contusive Spinal Cord Injury Model Mice

The carboxyl-terminal region of Crtac1B/LOTUS acts as a functional domain in endogenous antagonism to Nogo receptor-1

Collapsin Response Mediator Protein 1 Mediates Reelin Signaling in Cortical Neuronal Migration

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