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Drosophila myosin mutants model the disparate severity of type 1 and type 2B distal arthrogryposis and indicate an enhanced actin affinity mechanism

Skeletal muscle, 2020-08, Vol.10 (1), p.1-24, Article 24

2020

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The NADPH metabolic network regulates human αB-crystallin cardiomyopathy and reductive stress in Drosophila melanogaster

PLoS genetics, 2013-06, Vol.9 (6), p.e1003544-e1003544

2013

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A Drosophila model of dominant inclusion body myopathy type 3 shows diminished myosin kinetics that reduce muscle power and yield myofibrillar defects

Disease models & mechanisms, 2017-06, Vol.10 (6), p.761-771

2017

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Expression of the inclusion body myopathy 3 mutation in Drosophila depresses myosin function and stability and recapitulates muscle inclusions and weakness

Molecular biology of the cell, 2012-06, Vol.23 (11), p.2057-2065

2012

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Aging‐affiliated post‐translational modifications of skeletal muscle myosin affect biochemical properties, myofibril structure, muscle function, and proteostasis

Aging cell, 2024-06, Vol.23 (6), p.e14134-n/a

2024

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X-ray diffraction from flight muscle with a headless myosin mutation: implications for interpreting reflection patterns

Frontiers in physiology, 2014-10, Vol.5, p.416-416

2014

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Alternative S2 Hinge Regions of the Myosin Rod Differentially Affect Muscle Function, Myofibril Dimensions and Myosin Tail Length

Journal of molecular biology, 2007-04, Vol.367 (5), p.1312-1329

2007

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Suppression of myopathic lamin mutations by muscle-specific activation of AMPK and modulation of downstream signaling

Human molecular genetics, 2019-02, Vol.28 (3), p.351-371

2019

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A Restrictive Cardiomyopathy Mutation in an Invariant Proline at the Myosin Head/Rod Junction Enhances Head Flexibility and Function, Yielding Muscle Defects in Drosophila

Journal of molecular biology, 2016-06, Vol.428 (11), p.2446-2461

2016

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Drosophila UNC-45 accumulates in embryonic blastoderm and in muscles, and is essential for muscle myosin stability

Journal of cell science, 2011-03, Vol.124 (5), p.699-705

2011

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Variable N-terminal Regions of Muscle Myosin Heavy Chain Modulate ATPase Rate and Actin Sliding Velocity

The Journal of biological chemistry, 2003-05, Vol.278 (19), p.17475-17482

2003

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The myosin converter domain modulates muscle performance

Nature cell biology, 2002-04, Vol.4 (4), p.312-317

2002

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Manipulating Levels of Stress‐Response Proteins in a Drosophila Model of Myosin‐Based Inclusion Body Myopathy 3 Worsens Muscle Dysfunction

The FASEB journal, 2020-04, Vol.34 (S1), p.1-1

2020

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An Alternative Domain Near the Nucleotide-binding Site of Drosophila Muscle Myosin Affects ATPase Kinetics

Journal of molecular biology, 2005-10, Vol.353 (1), p.14-25

2005

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Assembly of thick filaments and myofibrils occurs in the absence of the myosin head

The EMBO journal, 1999-04, Vol.18 (7), p.1793-1804

1999

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A Drosophila Model of Myosin-Based Inclusion Body Myopathy Type 3: Effects on Muscle Structure, Muscle Function and Aggregated Protein Profiles

Biophysical journal, 2015-01, Vol.108 (2), p.304a-304a

2015

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Alleviation of Skeletal Muscle Defects Induced by Huntington's Disease-causing Amyloid by Modulating TOR Pathway in a Drosophila Model

Biophysical journal, 2013-01, Vol.104 (2), p.483a-483a

2013

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A Drosophila model of dominant inclusion body myopathy 3 shows diminished myosin kinetics that reduce muscle power and yield myofibrillar defects

Disease models & mechanisms, 2017-01

2017

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Exploration and Suppression of Tau-Induced Cardiac and Skeletal Muscle Defects in a Drosophila Model

Biophysical journal, 2013-01, Vol.104 (2), p.486a-486a

2013

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The NADPH Metabolic Network Regulates Human [alpha]B-crystallin Cardiomyopathy and Reductive Stress in Drosophila melanogaster: e1003544

PLoS genetics, 2013-06, Vol.9 (6)

2013

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The E706K IBM3 Myosin Mutation Depresses the Chemomechanical Properties and Increases the Lability of the Molecular Motor

Biophysical journal, 2011-02, Vol.100 (3), p.129a-129a

2011

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Drosophila UNC-45 accumulates in embryonic blastoderm and in muscles, and is essential for muscle myosin stability

Development (Cambridge), 2011-03, Vol.138 (6), p.e1-e1

2011

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The NADPH Metabolic Network Regulates Human alpha B-crystallin Cardiomyopathy and Reductive Stress in Drosophila melanogaster. e1003544

PLoS genetics, 2013-06, Vol.9 (6)

2013

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Guidelines and Resources for Conducting an Environmental Crime Investigation in the United States

Environmental forensics, 2002-06, Vol.3 (2), p.91-113

2002

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ACCREDITATIION AT THE US EPA--NEIC

Quality assurance (San Diego, Calif.), 2002-01, Vol.9 (1), p.31-41

2002

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Drosophila myosin mutants model the disparate severity of type 1 and type 2B distal arthrogryposis and indicate an enhanced actin affinity mechanism

The NADPH metabolic network regulates human αB-crystallin cardiomyopathy and reductive stress in Drosophila melanogaster

A Drosophila model of dominant inclusion body myopathy type 3 shows diminished myosin kinetics that reduce muscle power and yield myofibrillar defects

Expression of the inclusion body myopathy 3 mutation in Drosophila depresses myosin function and stability and recapitulates muscle inclusions and weakness

Aging‐affiliated post‐translational modifications of skeletal muscle myosin affect biochemical properties, myofibril structure, muscle function, and proteostasis

X-ray diffraction from flight muscle with a headless myosin mutation: implications for interpreting reflection patterns

Alternative S2 Hinge Regions of the Myosin Rod Differentially Affect Muscle Function, Myofibril Dimensions and Myosin Tail Length

Suppression of myopathic lamin mutations by muscle-specific activation of AMPK and modulation of downstream signaling

A Restrictive Cardiomyopathy Mutation in an Invariant Proline at the Myosin Head/Rod Junction Enhances Head Flexibility and Function, Yielding Muscle Defects in Drosophila

Drosophila UNC-45 accumulates in embryonic blastoderm and in muscles, and is essential for muscle myosin stability

Variable N-terminal Regions of Muscle Myosin Heavy Chain Modulate ATPase Rate and Actin Sliding Velocity

The myosin converter domain modulates muscle performance

Manipulating Levels of Stress‐Response Proteins in a Drosophila Model of Myosin‐Based Inclusion Body Myopathy 3 Worsens Muscle Dysfunction

An Alternative Domain Near the Nucleotide-binding Site of Drosophila Muscle Myosin Affects ATPase Kinetics

Assembly of thick filaments and myofibrils occurs in the absence of the myosin head

A Drosophila Model of Myosin-Based Inclusion Body Myopathy Type 3: Effects on Muscle Structure, Muscle Function and Aggregated Protein Profiles

Alleviation of Skeletal Muscle Defects Induced by Huntington's Disease-causing Amyloid by Modulating TOR Pathway in a Drosophila Model

A Drosophila model of dominant inclusion body myopathy 3 shows diminished myosin kinetics that reduce muscle power and yield myofibrillar defects

Exploration and Suppression of Tau-Induced Cardiac and Skeletal Muscle Defects in a Drosophila Model

The NADPH Metabolic Network Regulates Human [alpha]B-crystallin Cardiomyopathy and Reductive Stress in Drosophila melanogaster: e1003544

The E706K IBM3 Myosin Mutation Depresses the Chemomechanical Properties and Increases the Lability of the Molecular Motor

Drosophila UNC-45 accumulates in embryonic blastoderm and in muscles, and is essential for muscle myosin stability

The NADPH Metabolic Network Regulates Human alpha B-crystallin Cardiomyopathy and Reductive Stress in Drosophila melanogaster. e1003544

Guidelines and Resources for Conducting an Environmental Crime Investigation in the United States

ACCREDITATIION AT THE US EPA--NEIC

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