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Nucleation-Dependent Polymerization Is an Essential Component of Amyloid-Mediated Neuronal Cell Death

The Journal of neuroscience, 2005-02, Vol.25 (5), p.1071-1080

2005

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Amyloid Deposition in the Hippocampus and Entorhinal Cortex: Quantitative Analysis of a Transgenic Mouse Model

Proceedings of the National Academy of Sciences - PNAS, 2003-04, Vol.100 (8), p.4837-4842

2003

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Selective Vulnerability of Dentate Granule Cells Prior to Amyloid Deposition in PDAPP Mice: Digital Morphometric Analyses

Proceedings of the National Academy of Sciences - PNAS, 2004-05, Vol.101 (18), p.7141-7146

2004

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The Oligomerization of Amyloid β-Protein Begins Intracellularly in Cells Derived from Human Brain

Biochemistry (Easton), 2000-09, Vol.39 (35), p.10831-10839

2000

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Mechanism of plasminogen activator inhibitor‐1 regulation by oncostatin M and interleukin‐1 in human astrocytes

Journal of neurochemistry, 2002-11, Vol.83 (3), p.696-703

2002

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The Plasmin System Is Induced by and Degrades Amyloid-beta Aggregates

The Journal of neuroscience, 2000-06, Vol.20 (11), p.3937-3946

2000

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α2β1 and αVβ1 integrin signaling pathways mediate amyloid-β-induced neurotoxicity

Neurobiology of aging, 2007-02, Vol.28 (2), p.226-237

2007

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Aggregated Amyloid-beta Protein Induces Cortical Neuronal Apoptosis and Concomitant "Apoptotic" Pattern of Gene Induction

The Journal of neuroscience, 1997-10, Vol.17 (20), p.7736-7745

1997

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Tissue Plasminogen Activator Requires Plasminogen to Modulate Amyloid‐β Neurotoxicity and Deposition

Journal of neurochemistry, 2000-11, Vol.75 (5), p.2172-2177

2000

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Long-term soluble Aβ1–40 activates CaM kinase II in organotypic hippocampal cultures

Neurobiology of aging, 2007-09, Vol.28 (9), p.1388-1395

2007

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Amyloid β Peptide Potentiates Cytokine Secretion by Interleukin-1 β-Activated Human Astrocytoma Cells

Proceedings of the National Academy of Sciences - PNAS, 1995-11, Vol.92 (23), p.10738-10741

1995

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Oncostatin M and the Interleukin-6 and Soluble Interleukin-6 Receptor Complex Regulate α1-Antichymotrypsin Expression in Human Cortical Astrocytes

The Journal of biological chemistry, 1998-02, Vol.273 (7), p.4112-4118

1998

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Astrocyte‐ and hepatocyte‐specific expression of genes from the distal serpin subcluster at 14q32.1 associates with tissue‐specific chromatin structures

Journal of neurochemistry, 2005-08, Vol.94 (3), p.763-773

2005

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Duration of α1‐antichymotrypsin gene activation by interleukin‐1 is determined by efficiency of inhibitor of nuclear factor κBα resynthesis in primary human astrocytes

Journal of neurochemistry, 2005-02, Vol.92 (4), p.730-738

2005

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Amyloid ox-tox transducers

Nature (London), 1996-08, Vol.382 (6593), p.674-675

1996

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c‐Jun Contributes to Amyloid β‐Induced Neuronal Apoptosis but Is Not Necessary for Amyloid β‐Induced c‐jun Induction

Journal of neurochemistry, 1999-12, Vol.73 (6), p.2609-2612

1999

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Therapeutic Approaches Related to Amyloid-.beta. Peptide and Alzheimer's Disease

Journal of medicinal chemistry, 1995-10, Vol.38 (21), p.4141-4154

1995

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Duration of alpha 1-antichymotrypsin gene activation by interleukin-1 is determined by efficiency of inhibitor of nuclear factor kappa B alpha resynthesis in primary human astrocytes

Journal of neurochemistry, 2005-02, Vol.92 (4), p.730-738

2005

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beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity

The Journal of neuroscience, 1992-02, Vol.12 (2), p.376-389

1992

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Mechanism of Interleukin-1- and Tumor Necrosis Factor alpha -Dependent Regulation of the alpha 1-Antichymotrypsin Gene in Human Astrocytes

The Journal of neuroscience, 2000-10, Vol.20 (20), p.7510-7516

2000

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Increased Activity‐Regulating and Neuroprotective Efficacy of α‐Secretase‐Derived Secreted Amyloid Precursor Protein Conferred by a C‐Terminal Heparin‐Binding Domain

Journal of neurochemistry, 1996-11, Vol.67 (5), p.1882-1896

1996

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Evidence for excitoprotective and intraneuronal calcium-regulating roles for secreted forms of the β-amyloid precursor protein

Neuron (Cambridge, Mass.), 1993-02, Vol.10 (2), p.243-254

1993

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β-Amyloid precursor protein metabolites and loss of neuronal Ca2+ homeostasis in Alzheimer's disease

Trends in neurosciences (Regular ed.), 1993-10, Vol.16 (10), p.409-414

1993

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Calcium-destabilizing and neurodegenerative effects of aggregated β-amyloid peptide are attenuated by basic FGF

Brain research, 1993-09, Vol.621 (1), p.35-49

1993

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Secondary structure of amyloid beta peptide correlates with neurotoxic activity in vitro

Molecular pharmacology, 1994-03, Vol.45 (3), p.373-379

1994

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Nucleation-Dependent Polymerization Is an Essential Component of Amyloid-Mediated Neuronal Cell Death

Amyloid Deposition in the Hippocampus and Entorhinal Cortex: Quantitative Analysis of a Transgenic Mouse Model

Selective Vulnerability of Dentate Granule Cells Prior to Amyloid Deposition in PDAPP Mice: Digital Morphometric Analyses

The Oligomerization of Amyloid β-Protein Begins Intracellularly in Cells Derived from Human Brain

Mechanism of plasminogen activator inhibitor‐1 regulation by oncostatin M and interleukin‐1 in human astrocytes

The Plasmin System Is Induced by and Degrades Amyloid-beta Aggregates

α2β1 and αVβ1 integrin signaling pathways mediate amyloid-β-induced neurotoxicity

Aggregated Amyloid-beta Protein Induces Cortical Neuronal Apoptosis and Concomitant "Apoptotic" Pattern of Gene Induction

Tissue Plasminogen Activator Requires Plasminogen to Modulate Amyloid‐β Neurotoxicity and Deposition

Long-term soluble Aβ1–40 activates CaM kinase II in organotypic hippocampal cultures

Amyloid β Peptide Potentiates Cytokine Secretion by Interleukin-1 β-Activated Human Astrocytoma Cells

Oncostatin M and the Interleukin-6 and Soluble Interleukin-6 Receptor Complex Regulate α1-Antichymotrypsin Expression in Human Cortical Astrocytes

Astrocyte‐ and hepatocyte‐specific expression of genes from the distal serpin subcluster at 14q32.1 associates with tissue‐specific chromatin structures

Duration of α1‐antichymotrypsin gene activation by interleukin‐1 is determined by efficiency of inhibitor of nuclear factor κBα resynthesis in primary human astrocytes

Amyloid ox-tox transducers

c‐Jun Contributes to Amyloid β‐Induced Neuronal Apoptosis but Is Not Necessary for Amyloid β‐Induced c‐jun Induction

Therapeutic Approaches Related to Amyloid-.beta. Peptide and Alzheimer's Disease

Duration of alpha 1-antichymotrypsin gene activation by interleukin-1 is determined by efficiency of inhibitor of nuclear factor kappa B alpha resynthesis in primary human astrocytes

beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity

Mechanism of Interleukin-1- and Tumor Necrosis Factor alpha -Dependent Regulation of the alpha 1-Antichymotrypsin Gene in Human Astrocytes

Increased Activity‐Regulating and Neuroprotective Efficacy of α‐Secretase‐Derived Secreted Amyloid Precursor Protein Conferred by a C‐Terminal Heparin‐Binding Domain

Evidence for excitoprotective and intraneuronal calcium-regulating roles for secreted forms of the β-amyloid precursor protein

β-Amyloid precursor protein metabolites and loss of neuronal Ca2+ homeostasis in Alzheimer's disease

Calcium-destabilizing and neurodegenerative effects of aggregated β-amyloid peptide are attenuated by basic FGF

Secondary structure of amyloid beta peptide correlates with neurotoxic activity in vitro

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