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Hypoxia-inducible factor-1α signaling in aquaporin upregulation after traumatic brain injury

Neuroscience letters, 2009-03, Vol.453 (1), p.68-72

2009

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Brain ischemia and reperfusion: molecular mechanisms of neuronal injury

Journal of the Neurological Sciences, 2000-10, Vol.179 (1), p.1-33

2000

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Current brain endothelin research: a pathway to novel approaches impacting clinical trials

Neurological research (New York), 2011-03, Vol.33 (2), p.115-118

2011

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Reduced apoptosis by combining normobaric oxygenation with ethanol in transient ischemic stroke

Brain research, 2013-09, Vol.1531, p.17-24

2013

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Retraction notice to “Synapse loss regulated by matrix metalloproteinases in traumatic brain injury is associated with hypoxia-inducible factor-1α expression” [Brain Res. 1268 (2009) 125–134]

Brain research, 2018-09, Vol.1694, p.150-150

2018

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Polyadenylated mRNA staining reveals distinct neuronal phenotypes following endothelin-1, focal brain ischemia, and global brain ischemia/reperfusion

Neurological research (New York), 2011-03, Vol.33 (2), p.145-161

2011

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Neuronal damage and functional deficits are ameliorated by inhibition of aquaporin and HIF1α after traumatic brain injury (TBI)

Journal of the neurological sciences, 2012-12, Vol.323 (1), p.134-140

2012

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Cerebral angiogenesis and expression of angiogenic factors in aging rats after exercise

Current neurovascular research, 2006-02, Vol.3 (1), p.15-23

2006

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Endothelin receptors A and B are expressed in distinct cellular compartments of rat hippocampus following global ischemia: an immunocytochemical study

Neurological research (New York), 2011-03, Vol.33 (2), p.162-168

2011

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Exacerbation of Brain Injury by Post-Stroke Exercise Is Contingent Upon Exercise Initiation Timing

Frontiers in cellular neuroscience, 2017-10, Vol.11, p.311-311

2017

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Neovascularization following traumatic brain injury: possible evidence for both angiogenesis and vasculogenesis

Neurological research (New York), 2007-06, Vol.29 (4), p.375-381

2007

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Cerebral ischemia-induced apoptosis and necrosis in normal and diabetic rats: Effects of insulin and C-peptide

Brain research, 2006-06, Vol.1096 (1), p.204-212

2006

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Synapse loss regulated by matrix metalloproteinases in traumatic brain injury is associated with hypoxia inducible factor-1α expression

Brain research, 2009-05, Vol.1268, p.125-134

2009

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Increased astrocyte proliferation in rats after running exercise

Neuroscience letters, 2005-10, Vol.386 (3), p.160-164

2005

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Exercise pre-conditioning strengthens brain microvascular integrity in a rat stroke model

Neurological research (New York), 2006-03, Vol.28 (2), p.184-189

2006

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Exercise preconditioning ameliorates inflammatory injury in ischemic rats during reperfusion

Acta neuropathologica, 2005-04, Vol.109 (3), p.237-246

2005

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Pericyte Migration from the Vascular Wall in Response to Traumatic Brain Injury

Microvascular research, 2000-07, Vol.60 (1), p.55-69

2000

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Differential expression of capillary VEGF isoforms following traumatic brain injury

Neurological research (New York), 2007-06, Vol.29 (4), p.395-403

2007

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Prereperfusion saline infusion into ischemic territory reduces inflammatory injury after transient middle cerebral artery occlusion in rats

Stroke (1970), 2002-10, Vol.33 (10), p.2492-2498

2002

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Alterations in cerebral cortex microvessels and the microcirculation in a rat model of traumatic brain injury: a correlative EM and laser Doppler flowmetry study

Neurological research (New York), 2007-06, Vol.29 (4), p.339-347

2007

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Exercise-induced overexpression of angiogenic factors and reduction of ischemia/reperfusion injury in stroke

Current neurovascular research, 2004-12, Vol.1 (5), p.411-420

2004

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Combining Normobaric Oxygen with Ethanol or Hypothermia Prevents Brain Damage from Thromboembolic Stroke via PKC-Akt-NOX Modulation

Molecular neurobiology, 2017-03, Vol.54 (2), p.1263-1277

2017

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Extent of nerve cell injury in Marmarou's model compared to other brain trauma models

Neurological research (New York), 2007-06, Vol.29 (4), p.348-355

2007

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Inflammation in Diabetic Encephalopathy is Prevented by C-Peptide

The review of diabetic studies, 2009, Vol.6 (1), p.37-42

2009

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Editorial: microvascular and neuronal responses in a model of diffuse brain injury: therapeutic implications

Neurological research (New York), 2007-06, Vol.29 (4), p.337-338

Rafols, José A

2007

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Hypoxia-inducible factor-1α signaling in aquaporin upregulation after traumatic brain injury

Brain ischemia and reperfusion: molecular mechanisms of neuronal injury

Current brain endothelin research: a pathway to novel approaches impacting clinical trials

Reduced apoptosis by combining normobaric oxygenation with ethanol in transient ischemic stroke

Retraction notice to “Synapse loss regulated by matrix metalloproteinases in traumatic brain injury is associated with hypoxia-inducible factor-1α expression” [Brain Res. 1268 (2009) 125–134]

Polyadenylated mRNA staining reveals distinct neuronal phenotypes following endothelin-1, focal brain ischemia, and global brain ischemia/reperfusion

Neuronal damage and functional deficits are ameliorated by inhibition of aquaporin and HIF1α after traumatic brain injury (TBI)

Cerebral angiogenesis and expression of angiogenic factors in aging rats after exercise

Endothelin receptors A and B are expressed in distinct cellular compartments of rat hippocampus following global ischemia: an immunocytochemical study

Exacerbation of Brain Injury by Post-Stroke Exercise Is Contingent Upon Exercise Initiation Timing

Neovascularization following traumatic brain injury: possible evidence for both angiogenesis and vasculogenesis

Cerebral ischemia-induced apoptosis and necrosis in normal and diabetic rats: Effects of insulin and C-peptide

Synapse loss regulated by matrix metalloproteinases in traumatic brain injury is associated with hypoxia inducible factor-1α expression

Increased astrocyte proliferation in rats after running exercise

Exercise pre-conditioning strengthens brain microvascular integrity in a rat stroke model

Exercise preconditioning ameliorates inflammatory injury in ischemic rats during reperfusion

Pericyte Migration from the Vascular Wall in Response to Traumatic Brain Injury

Differential expression of capillary VEGF isoforms following traumatic brain injury

Prereperfusion saline infusion into ischemic territory reduces inflammatory injury after transient middle cerebral artery occlusion in rats

Alterations in cerebral cortex microvessels and the microcirculation in a rat model of traumatic brain injury: a correlative EM and laser Doppler flowmetry study

Exercise-induced overexpression of angiogenic factors and reduction of ischemia/reperfusion injury in stroke

Combining Normobaric Oxygen with Ethanol or Hypothermia Prevents Brain Damage from Thromboembolic Stroke via PKC-Akt-NOX Modulation

Extent of nerve cell injury in Marmarou's model compared to other brain trauma models

Inflammation in Diabetic Encephalopathy is Prevented by C-Peptide

Editorial: microvascular and neuronal responses in a model of diffuse brain injury: therapeutic implications

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