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Diabetic cardiomyopathy: pathophysiology and clinical features

Heart failure reviews, 2013-03, Vol.18 (2), p.149-166

2013

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GSK-3β, a Therapeutic Target for Cardiomyocyte Protection

Circulation Journal, 2009, Vol.73(7), pp.1184-1192

2009

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Effects of diabetes on myocardial infarct size and cardioprotection by preconditioning and postconditioning

Cardiovascular diabetology, 2012-06, Vol.11 (1), p.67-67

2012

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Empagliflozin attenuates acute kidney injury after myocardial infarction in diabetic rats

Scientific reports, 2020-04, Vol.10 (1), p.7238-7238, Article 7238

2020

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Intracellular artificial supramolecules based on de novo designed Y15 peptides

Nature communications, 2021-06, Vol.12 (1), p.3412-3412, Article 3412

2021

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Empagliflozin normalizes the size and number of mitochondria and prevents reduction in mitochondrial size after myocardial infarction in diabetic hearts

Physiological reports, 2018-06, Vol.6 (12), p.e13741-n/a

2018

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mTORC1 inhibition attenuates necroptosis through RIP1 inhibition-mediated TFEB activation

Biochimica et biophysica acta. Molecular basis of disease, 2019-12, Vol.1865 (12), p.165552-165552, Article 165552

2019

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Impact of the COVID-19 Pandemic on Glycemic Control and Blood Pressure Control in Patients with Diabetes in Japan

Internal Medicine, 2022/01/01, Vol.61(1), pp.37-48

2022

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Canagliflozin, a sodium–glucose cotransporter 2 inhibitor, normalizes renal susceptibility to type 1 cardiorenal syndrome through reduction of renal oxidative stress in diabetic rats

Journal of diabetes investigation, 2019-07, Vol.10 (4), p.933-946

2019

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Ligand-Directed Acyl Imidazole Chemistry for Labeling of Membrane-Bound Proteins on Live Cells

Journal of the American Chemical Society, 2012-03, Vol.134 (9), p.3961-3964

2012

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Involvement of necroptosis in contrast-induced nephropathy in a rat CKD model

Clinical and experimental nephrology, 2021-07, Vol.25 (7), p.708-717

2021

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Diabetes increases the susceptibility to acute kidney injury after myocardial infarction through augmented activation of renal Toll-like receptors in rats

American journal of physiology. Heart and circulatory physiology, 2017-12, Vol.313 (6), p.H1130-H1142

2017

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Suppression of autophagic flux contributes to cardiomyocyte death by activation of necroptotic pathways

Journal of molecular and cellular cardiology, 2017-07, Vol.108, p.203-213

2017

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Reduction in GLP-1 secretory capacity may be a novel independent risk factor of coronary artery stenosis

Scientific reports, 2021-08, Vol.11 (1), p.15578-15578, Article 15578

2021

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Accuracy of flash glucose monitoring in insulin‐treated patients with type 2 diabetes

Journal of diabetes investigation, 2019-05, Vol.10 (3), p.846-850

2019

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A conditional proteomics approach to identify proteins involved in zinc homeostasis

Nature methods, 2016-11, Vol.13 (11), p.931

2016

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Fluorophore Labeling of Native FKBP12 by Ligand-Directed Tosyl Chemistry Allows Detection of Its Molecular Interactions in Vitro and in Living Cells

Journal of the American Chemical Society, 2013-05, Vol.135 (18), p.6782-6785

2013

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Xanthine oxidoreductase-mediated injury is amplified by upregulated AMP deaminase in type 2 diabetic rat hearts under the condition of pressure overload

Journal of molecular and cellular cardiology, 2021-05, Vol.154, p.21-31

2021

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Endoplasmic Reticulum Stress in Diabetic Hearts Abolishes Erythropoietin-Induced Myocardial Protection by Impairment of Phospho–Glycogen Synthase Kinase-3β–Mediated Suppression of Mitochondrial Permeability Transition

Diabetes (New York, N.Y.), 2009-12, Vol.58 (12), p.2863-2872

2009

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Role of the gap junction in ischemic preconditioning in the heart

American journal of physiology. Heart and circulatory physiology, 2010-04, Vol.298 (4), p.H1115-H1125

2010

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Suppressed autophagic response underlies augmentation of renal ischemia/reperfusion injury by type 2 diabetes

Scientific reports, 2017-07, Vol.7 (1), p.5311-13, Article 5311

2017

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Does p53 Inhibition Suppress Myocardial Ischemia–Reperfusion Injury?

Journal of cardiovascular pharmacology and therapeutics, 2018-07, Vol.23 (4), p.350-357

2018

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Drug Development Targeting the Glycogen Synthase Kinase-3β (GSK-3β)-Mediated Signal Transduction Pathway: Role of GSK-3β in Myocardial Protection Against Ischemia/Reperfusion Injury

Journal of Pharmacological Sciences, 2009, Vol.109(2), pp.162-167

2009

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Accelerated recovery of mitochondrial membrane potential by GSK-3β inactivation affords cardiomyocytes protection from oxidant-induced necrosis

PloS one, 2014-11, Vol.9 (11), p.e112529-e112529

2014

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Distinct impacts of sleep-disordered breathing on glycemic variability in patients with and without diabetes mellitus

PloS one, 2017-12, Vol.12 (12), p.e0188689-e0188689

2017

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Diabetic cardiomyopathy: pathophysiology and clinical features

GSK-3β, a Therapeutic Target for Cardiomyocyte Protection

Effects of diabetes on myocardial infarct size and cardioprotection by preconditioning and postconditioning

Empagliflozin attenuates acute kidney injury after myocardial infarction in diabetic rats

Intracellular artificial supramolecules based on de novo designed Y15 peptides

Empagliflozin normalizes the size and number of mitochondria and prevents reduction in mitochondrial size after myocardial infarction in diabetic hearts

mTORC1 inhibition attenuates necroptosis through RIP1 inhibition-mediated TFEB activation

Impact of the COVID-19 Pandemic on Glycemic Control and Blood Pressure Control in Patients with Diabetes in Japan

Canagliflozin, a sodium–glucose cotransporter 2 inhibitor, normalizes renal susceptibility to type 1 cardiorenal syndrome through reduction of renal oxidative stress in diabetic rats

Ligand-Directed Acyl Imidazole Chemistry for Labeling of Membrane-Bound Proteins on Live Cells

Involvement of necroptosis in contrast-induced nephropathy in a rat CKD model

Diabetes increases the susceptibility to acute kidney injury after myocardial infarction through augmented activation of renal Toll-like receptors in rats

Suppression of autophagic flux contributes to cardiomyocyte death by activation of necroptotic pathways

Reduction in GLP-1 secretory capacity may be a novel independent risk factor of coronary artery stenosis

Accuracy of flash glucose monitoring in insulin‐treated patients with type 2 diabetes

A conditional proteomics approach to identify proteins involved in zinc homeostasis

Fluorophore Labeling of Native FKBP12 by Ligand-Directed Tosyl Chemistry Allows Detection of Its Molecular Interactions in Vitro and in Living Cells

Xanthine oxidoreductase-mediated injury is amplified by upregulated AMP deaminase in type 2 diabetic rat hearts under the condition of pressure overload

Endoplasmic Reticulum Stress in Diabetic Hearts Abolishes Erythropoietin-Induced Myocardial Protection by Impairment of Phospho–Glycogen Synthase Kinase-3β–Mediated Suppression of Mitochondrial Permeability Transition

Role of the gap junction in ischemic preconditioning in the heart

Suppressed autophagic response underlies augmentation of renal ischemia/reperfusion injury by type 2 diabetes

Does p53 Inhibition Suppress Myocardial Ischemia–Reperfusion Injury?

Drug Development Targeting the Glycogen Synthase Kinase-3β (GSK-3β)-Mediated Signal Transduction Pathway: Role of GSK-3β in Myocardial Protection Against Ischemia/Reperfusion Injury

Accelerated recovery of mitochondrial membrane potential by GSK-3β inactivation affords cardiomyocytes protection from oxidant-induced necrosis

Distinct impacts of sleep-disordered breathing on glycemic variability in patients with and without diabetes mellitus

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