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EGF816 Exerts Anticancer Effects in Non-Small Cell Lung Cancer by Irreversibly and Selectively Targeting Primary and Acquired Activating Mutations in the EGF Receptor

Cancer research (Chicago, Ill.), 2016-03, Vol.76 (6), p.1591-1602

2016

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Pharmacology of the Novel Antiangiogenic Peptide ATN-161 (Ac-PHSCN-NH2): Observation of a U-Shaped Dose-Response Curve in Several Preclinical Models of Angiogenesis and Tumor Growth

Clinical cancer research, 2008-04, Vol.14 (7), p.2137-2144

2008

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Overcoming EGFR(T790M) and EGFR(C797S) resistance with mutant-selective allosteric inhibitors

Nature (London), 2016-06, Vol.534 (7605), p.129-132

2016

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HTRF Kinase Assay Development and Methods in Inhibitor Characterization

Methods in molecular biology (Clifton, N.J.), 2016, Vol.1360, p.1

2016

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Superoxide Dismutase 1 (SOD1) Is Essential for H₂O₂-Mediated Oxidation and Inactivation of Phosphatases in Growth Factor Signaling

Proceedings of the National Academy of Sciences - PNAS, 2008-05, Vol.105 (20), p.7147-7152

2008

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Structure-Based Design of Selective LONP1 Inhibitors for Probing In Vitro Biology

Journal of medicinal chemistry, 2021-04, Vol.64 (8), p.4857-4869

2021

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Overcoming EGFR resistance with mutant-selective allosteric inhibitors.(RESEARCH: LETTER)

Nature (London), 2016-06, Vol.534 (7605), p.129

2016

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Overcoming EGFR T790M and C797S resistance with mutant-selective allosteric inhibitors

Nature (London), 2016-05, Vol.534 (7605), p.129-132

2016

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Abstract 1734: In vitro characterization of EGF816, a third-generation mutant-selective EGFR inhibitor

Cancer research (Chicago, Ill.), 2014-10, Vol.74 (19_Supplement), p.1734-1734

2014

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Superoxide dismutase 1 (SOD1) is essential for H^sub 2^O^sub 2^-mediated oxidation and inactivation of phosphatases in growth factor signaling

Proceedings of the National Academy of Sciences - PNAS, 2008-05, Vol.105 (20), p.7147

2008

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Superoxide dismutase 1 (SOD1) is essential for H 2 O 2 -mediated oxidation and inactivation of phosphatases in growth factor signaling

Proceedings of the National Academy of Sciences - PNAS, 2008-05, Vol.105 (20), p.7147-7152

2008

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Superoxide dismutase 1 (SOD1) is essential for H sub(2)O sub(2)-mediated oxidation and inactivation of phosphatases in growth factor signaling

Proceedings of the National Academy of Sciences - PNAS, 2008-05, Vol.105 (20), p.7147-7152

2008

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EGF816 Exerts Anticancer Effects in Non-Small Cell Lung Cancer by Irreversibly and Selectively Targeting Primary and Acquired Activating Mutations in the EGF Receptor

Pharmacology of the Novel Antiangiogenic Peptide ATN-161 (Ac-PHSCN-NH2): Observation of a U-Shaped Dose-Response Curve in Several Preclinical Models of Angiogenesis and Tumor Growth

Overcoming EGFR(T790M) and EGFR(C797S) resistance with mutant-selective allosteric inhibitors

HTRF Kinase Assay Development and Methods in Inhibitor Characterization

Superoxide Dismutase 1 (SOD1) Is Essential for H₂O₂-Mediated Oxidation and Inactivation of Phosphatases in Growth Factor Signaling

Structure-Based Design of Selective LONP1 Inhibitors for Probing In Vitro Biology

Overcoming EGFR resistance with mutant-selective allosteric inhibitors.(RESEARCH: LETTER)

Overcoming EGFR T790M and C797S resistance with mutant-selective allosteric inhibitors

Abstract 1734: In vitro characterization of EGF816, a third-generation mutant-selective EGFR inhibitor

Superoxide dismutase 1 (SOD1) is essential for H^sub 2^O^sub 2^-mediated oxidation and inactivation of phosphatases in growth factor signaling

Superoxide dismutase 1 (SOD1) is essential for H 2 O 2 -mediated oxidation and inactivation of phosphatases in growth factor signaling

Superoxide dismutase 1 (SOD1) is essential for H sub(2)O sub(2)-mediated oxidation and inactivation of phosphatases in growth factor signaling

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