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DNA–protein crosslinks from environmental exposure: Mechanisms of formation and repair

Environmental and molecular mutagenesis, 2020-08, Vol.61 (7), p.716-729

2020

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FAM111A protects replication forks from protein obstacles via its trypsin-like domain

Nature communications, 2020-03, Vol.11 (1), p.1318-1318, Article 1318

2020

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The Deubiquitinating Enzyme BAP1 Regulates Cell Growth via Interaction with HCF-1

The Journal of biological chemistry, 2009-12, Vol.284 (49), p.34179-34188

2009

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The APC/C inhibitor, Emi1, is essential for prevention of rereplication

Genes & development, 2007-01, Vol.21 (2), p.184-194

2007

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Spartan/C1orf124 is important to prevent UV-induced mutagenesis

Cell cycle (Georgetown, Tex.), 2012-09, Vol.11 (18), p.3395-3402

2012

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Dimerization-dependent serine protease activity of FAM111A prevents replication fork stalling at topoisomerase 1 cleavage complexes

Nature communications, 2024-03, Vol.15 (1), p.2064-2064

2024

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Spartan deficiency causes genomic instability and progeroid phenotypes

Nature communications, 2014-12, Vol.5 (1), p.5744-5744, Article 5744

2014

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Functions and evolution of FAM111 serine proteases

Frontiers in molecular biosciences, 2022-12, Vol.9, p.1081166-1081166

2022

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Right Place, Right Time, and Only Once: Replication Initiation in Metazoans

Cell, 2005-10, Vol.123 (1), p.13-24

2005

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MYC regulates ribosome biogenesis and mitochondrial gene expression programs through its interaction with host cell factor-1

eLife, 2021-01, Vol.10

2021

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UBE2T Is the E2 in the Fanconi Anemia Pathway and Undergoes Negative Autoregulation

Molecular cell, 2006-08, Vol.23 (4), p.589-596

2006

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Regulation of error-prone translesion synthesis by Spartan/C1orf124

Nucleic acids research, 2013-02, Vol.41 (3), p.1661

2013

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UBE2T, the Fanconi Anemia Core Complex, and FANCD2 Are Recruited Independently to Chromatin: a Basis for the Regulation of FANCD2 Monoubiquitination

Molecular and Cellular Biology, 2007-12, Vol.27 (24), p.8421-8430

2007

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BRCA1-associated Protein 1 (BAP1) Deubiquitinase Antagonizes the Ubiquitin-mediated Activation of FoxK2 Target Genes

The Journal of biological chemistry, 2015-01, Vol.290 (3), p.1580-1591

2015

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Human KIAA1018/FAN1 localizes to stalled replication forks via its ubiquitin-binding domain

Cell cycle (Georgetown, Tex.), 2010-10, Vol.9 (19), p.3977-3983

2010

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Acute Reduction of an Origin Recognition Complex (ORC) Subunit in Human Cells Reveals a Requirement of ORC for Cdk2 Activation

The Journal of biological chemistry, 2005-07, Vol.280 (30), p.27624-27630

2005

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Spartan deficiency causes accumulation of Topoisomerase 1 cleavage complexes and tumorigenesis

Nucleic acids research, 2017-05, Vol.45 (8), p.4564

2017

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A mechanism for the tissue specificity in BAP1 cancer syndrome

Translational cancer research, 2019-12, Vol.8 (Suppl 6), p.S621-S624

Machida, Yuichi J

2019

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Tyrosine Phosphorylation of Mitochondrial Creatine Kinase 1 Enhances a Druggable Tumor Energy Shuttle Pathway

Cell metabolism, 2018-12, Vol.28 (6), p.833-847.e8

2018

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Intra-S phase checkpoint kinase Chk1 dissociates replication proteins Treslin and TopBP1 through multiple mechanisms during replication stress

The Journal of biological chemistry, 2022-04, Vol.298 (4), p.101777-101777, Article 101777

2022

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Cellular Checkpoint Mechanisms Monitoring Proper Initiation of DNA Replication

The Journal of biological chemistry, 2005-02, Vol.280 (8), p.6253-6256

2005

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APC/C--the master controller of origin licensing?

Cell division, 2007-02, Vol.2 (1), p.8-8, Article 8

2007

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Proliferating Human Cells Hypomorphic for Origin Recognition Complex 2 and Pre-replicative Complex Formation Have a Defect in p53 Activation and Cdk2 Kinase Activation

The Journal of biological chemistry, 2006-03, Vol.281 (10), p.6253-6260

2006

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Targeted comparative RNA interference analysis reveals differential requirement of genes essential for cell proliferation

Molecular biology of the cell, 2006-11, Vol.17 (11), p.4837-4845

2006

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The APC/C inhibitor, Emi1, is essential for prevention of re‐replication

The FASEB journal, 2007-04, Vol.21 (5), p.A657-A657

2007

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DNA–protein crosslinks from environmental exposure: Mechanisms of formation and repair

FAM111A protects replication forks from protein obstacles via its trypsin-like domain

The Deubiquitinating Enzyme BAP1 Regulates Cell Growth via Interaction with HCF-1

The APC/C inhibitor, Emi1, is essential for prevention of rereplication

Spartan/C1orf124 is important to prevent UV-induced mutagenesis

Dimerization-dependent serine protease activity of FAM111A prevents replication fork stalling at topoisomerase 1 cleavage complexes

Spartan deficiency causes genomic instability and progeroid phenotypes

Functions and evolution of FAM111 serine proteases

Right Place, Right Time, and Only Once: Replication Initiation in Metazoans

MYC regulates ribosome biogenesis and mitochondrial gene expression programs through its interaction with host cell factor-1

UBE2T Is the E2 in the Fanconi Anemia Pathway and Undergoes Negative Autoregulation

Regulation of error-prone translesion synthesis by Spartan/C1orf124

UBE2T, the Fanconi Anemia Core Complex, and FANCD2 Are Recruited Independently to Chromatin: a Basis for the Regulation of FANCD2 Monoubiquitination

BRCA1-associated Protein 1 (BAP1) Deubiquitinase Antagonizes the Ubiquitin-mediated Activation of FoxK2 Target Genes

Human KIAA1018/FAN1 localizes to stalled replication forks via its ubiquitin-binding domain

Acute Reduction of an Origin Recognition Complex (ORC) Subunit in Human Cells Reveals a Requirement of ORC for Cdk2 Activation

Spartan deficiency causes accumulation of Topoisomerase 1 cleavage complexes and tumorigenesis

A mechanism for the tissue specificity in BAP1 cancer syndrome

Tyrosine Phosphorylation of Mitochondrial Creatine Kinase 1 Enhances a Druggable Tumor Energy Shuttle Pathway

Intra-S phase checkpoint kinase Chk1 dissociates replication proteins Treslin and TopBP1 through multiple mechanisms during replication stress

Cellular Checkpoint Mechanisms Monitoring Proper Initiation of DNA Replication

APC/C--the master controller of origin licensing?

Proliferating Human Cells Hypomorphic for Origin Recognition Complex 2 and Pre-replicative Complex Formation Have a Defect in p53 Activation and Cdk2 Kinase Activation

Targeted comparative RNA interference analysis reveals differential requirement of genes essential for cell proliferation

The APC/C inhibitor, Emi1, is essential for prevention of re‐replication

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