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Interleukin-6 Signaling Pathway and Its Role in Kidney Disease: An Update

Frontiers in immunology, 2017-04, Vol.8, p.405-405

2017

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Selenoprotein T protects against cisplatin‐induced acute kidney injury through suppression of oxidative stress and apoptosis

The FASEB journal, 2020-09, Vol.34 (9), p.11983-11996

2020

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Subcellular trafficking of tubular MDM2 implicates in acute kidney injury to chronic kidney disease transition during multiple low‐dose cisplatin exposure

The FASEB journal, 2020-01, Vol.34 (1), p.1620-1636

2020

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The classic signalling and trans‐signalling of interleukin‐6 are both injurious in podocyte under high glucose exposure

Journal of cellular and molecular medicine, 2018-01, Vol.22 (1), p.251-260

2018

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Inhibition of SIRT2 Alleviates Fibroblast Activation and Renal Tubulointerstitial Fibrosis via MDM2

Cellular physiology and biochemistry, 2018-01, Vol.46 (2), p.451-460

2018

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Risk factors for the mortality of hemodialysis patients with COVID‐19: A multicenter study from the overall hemodialysis population in Wuhan

Seminars in dialysis, 2022-01, Vol.35 (1), p.71-80

2022

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MDM2 Contributes to High Glucose-Induced Glomerular Mesangial Cell Proliferation and Extracellular Matrix Accumulation via Notch1

Scientific reports, 2017-09, Vol.7 (1), p.10393-12, Article 10393

2017

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MDM2 is implicated in high‐glucose‐induced podocyte mitotic catastrophe via Notch1 signalling

Journal of cellular and molecular medicine, 2017-12, Vol.21 (12), p.3435-3444

2017

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IL‐6 increases podocyte motility via MLC‐mediated focal adhesion impairment and cytoskeleton disassembly

Journal of cellular physiology, 2018-09, Vol.233 (9), p.7173-7181

2018

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MAD2B promotes podocyte injury through regulating Numb-dependent Notch 1 pathway in diabetic nephropathy

International journal of biological sciences, 2022-01, Vol.18 (5), p.1896-1911

2022

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NADPH Oxidase-Induced NALP3 Inflammasome Activation Is Driven by Thioredoxin-Interacting Protein Which Contributes to Podocyte Injury in Hyperglycemia

Journal of diabetes research, 2015-01, Vol.2015, p.504761-12

2015

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PKC-α Triggers EGFR Ubiquitination, Endocytosis and ERK Activation in Podocytes Stimulated with High Glucose

Cellular physiology and biochemistry, 2017-01, Vol.42 (1), p.281-294

2017

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MAD2B-mediated cell cycle reentry of podocytes is involved in the pathogenesis of FSGS

International journal of biological sciences, 2021-01, Vol.17 (15), p.4396-4408

2021

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Clinical Characteristics of and Medical Interventions for COVID-19 in Hemodialysis Patients in Wuhan, China

Journal of the American Society of Nephrology, 2020-07, Vol.31 (7), p.1387-1397

2020

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MAD2B contributes to parietal epithelial cell activation and crescentic glomerulonephritis via Skp2

American journal of physiology. Renal physiology, 2020-10, Vol.319 (4), p.F636-F646

2020

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A glimpse into the psychological status of E.N.T inpatients in China: A cross-sectional survey of three hospitals in different regions

World Journal of Otorhinolaryngology-Head and Neck Surgery, 2019-06, Vol.5 (2), p.95-104

2019

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Rapid assessment of avoidable blindness cataract in country Anyue in Sichuan Province

Zhong hua yi xue za zhi, 2013-12, Vol.93 (47), p.3741-3743

2013

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Role and Association of Inflammatory and Apoptotic Caspases in Renal Tubulointerstitial Fibrosis

Kidney & blood pressure research, 2016-01, Vol.41 (5), p.643-653

2016

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Serologic Detection of SARS-CoV-2 Infections in Hemodialysis Centers: A Multicenter Retrospective Study in Wuhan, China

American journal of kidney diseases, 2020-10, Vol.76 (4), p.490-499.e1

2020

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MDM2 mediates fibroblast activation and renal tubulointerstitial fibrosis via a p53-independent pathway

American journal of physiology. Renal physiology, 2017-04, Vol.312 (4), p.F760-F768

2017

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Effects of travoprost on actin cytoskeleton and β-catenin in the human trabecular meshwork cells treated with Dexamethasone

Chung-hua yen k'o tsa chih, 2011-04, Vol.47 (4), p.336

2011

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MAD2B promotes tubular epithelial-to-mesenchymal transition and renal tubulointerstitial fibrosis via Skp2

Journal of molecular medicine (Berlin, Germany), 2016-11, Vol.94 (11), p.1297-1307

2016

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Erratum to “NADPH Oxidase-Induced NALP3 Inflammasome Activation Is Driven by Thioredoxin-Interacting Protein Which Contributes to Podocyte Injury in Hyperglycemia”

Journal of diabetes research, 2016-01, Vol.2016, p.1213892-2

2016

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MAD2B-mediated SnoN downregulation is implicated in fibroblast activation and tubulointerstitial fibrosis

American journal of physiology. Renal physiology, 2016-07, Vol.311 (1), p.F207-F216

2016

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MDM 2 is implicated in high‐glucose‐induced podocyte mitotic catastrophe via Notch1 signalling

Journal of cellular and molecular medicine, 2017-12, Vol.21 (12), p.3435-3444

2017

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Interleukin-6 Signaling Pathway and Its Role in Kidney Disease: An Update

Selenoprotein T protects against cisplatin‐induced acute kidney injury through suppression of oxidative stress and apoptosis

Subcellular trafficking of tubular MDM2 implicates in acute kidney injury to chronic kidney disease transition during multiple low‐dose cisplatin exposure

The classic signalling and trans‐signalling of interleukin‐6 are both injurious in podocyte under high glucose exposure

Inhibition of SIRT2 Alleviates Fibroblast Activation and Renal Tubulointerstitial Fibrosis via MDM2

Risk factors for the mortality of hemodialysis patients with COVID‐19: A multicenter study from the overall hemodialysis population in Wuhan

MDM2 Contributes to High Glucose-Induced Glomerular Mesangial Cell Proliferation and Extracellular Matrix Accumulation via Notch1

MDM2 is implicated in high‐glucose‐induced podocyte mitotic catastrophe via Notch1 signalling

IL‐6 increases podocyte motility via MLC‐mediated focal adhesion impairment and cytoskeleton disassembly

MAD2B promotes podocyte injury through regulating Numb-dependent Notch 1 pathway in diabetic nephropathy

NADPH Oxidase-Induced NALP3 Inflammasome Activation Is Driven by Thioredoxin-Interacting Protein Which Contributes to Podocyte Injury in Hyperglycemia

PKC-α Triggers EGFR Ubiquitination, Endocytosis and ERK Activation in Podocytes Stimulated with High Glucose

MAD2B-mediated cell cycle reentry of podocytes is involved in the pathogenesis of FSGS

Clinical Characteristics of and Medical Interventions for COVID-19 in Hemodialysis Patients in Wuhan, China

MAD2B contributes to parietal epithelial cell activation and crescentic glomerulonephritis via Skp2

A glimpse into the psychological status of E.N.T inpatients in China: A cross-sectional survey of three hospitals in different regions

Rapid assessment of avoidable blindness cataract in country Anyue in Sichuan Province

Role and Association of Inflammatory and Apoptotic Caspases in Renal Tubulointerstitial Fibrosis

Serologic Detection of SARS-CoV-2 Infections in Hemodialysis Centers: A Multicenter Retrospective Study in Wuhan, China

MDM2 mediates fibroblast activation and renal tubulointerstitial fibrosis via a p53-independent pathway

Effects of travoprost on actin cytoskeleton and β-catenin in the human trabecular meshwork cells treated with Dexamethasone

MAD2B promotes tubular epithelial-to-mesenchymal transition and renal tubulointerstitial fibrosis via Skp2

Erratum to “NADPH Oxidase-Induced NALP3 Inflammasome Activation Is Driven by Thioredoxin-Interacting Protein Which Contributes to Podocyte Injury in Hyperglycemia”

MAD2B-mediated SnoN downregulation is implicated in fibroblast activation and tubulointerstitial fibrosis

MDM 2 is implicated in high‐glucose‐induced podocyte mitotic catastrophe via Notch1 signalling

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