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Optimized Sleeping Beauty transposons rapidly generate stable transgenic cell lines

Biotechnology journal, 2015-04, Vol.10 (4), p.647-653

2015

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Image-Based Annotation of Chemogenomic Libraries for Phenotypic Screening

Molecules (Basel, Switzerland), 2022-02, Vol.27 (4), p.1439

2022

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Vaccine-induced COVID-19 mimicry syndrome

eLife, 2022-01, Vol.11

2022

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MLL-AF4 and a murinized pSer-variant thereof are turning on the nucleolar stress pathway

Cell & bioscience, 2022-04, Vol.12 (1), p.47-47, Article 47

2022

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Generation of a Sleeping Beauty Transposon-Based Cellular System for Rapid and Sensitive Screening for Compounds and Cellular Factors Limiting SARS-CoV-2 Replication

Frontiers in microbiology, 2021-07, Vol.12, p.701198-701198

2021

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How chromosomal translocations arise to cause cancer: Gene proximity, trans-splicing, and DNA end joining

iScience, 2023-06, Vol.26 (6), p.106900-106900, Article 106900

2023

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Distinct and targetable role of calcium-sensing receptor in leukaemia

Nature communications, 2023-10, Vol.14 (1), p.6242-6242, Article 6242

2023

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The MLL recombinome of adult CD10-negative B-cell precursor acute lymphoblastic leukemia: results from the GMALL study group

Blood, 2009-04, Vol.113 (17), p.4011-4015

2009

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Do non-genomically encoded fusion transcripts cause recurrent chromosomal translocations?

Cancers, 2012-10, Vol.4 (4), p.1036-1049

2012

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Unraveling the Activation Mechanism of Taspase1 which Controls the Oncogenic AF4–MLL Fusion Protein

EBioMedicine, 2015-05, Vol.2 (5), p.386-395

2015

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The role of reciprocal fusions in MLL-r acute leukemia: studying the chromosomal translocation t(6;11)

Oncogene, 2021-10, Vol.40 (40), p.5902-5912

2021

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Biology of t(6;11) Fusion Proteins and Their Role in MLL-Rearranged Acute Leukemia Lineage Determination

Blood, 2019-11, Vol.134 (Supplement_1), p.5033-5033

2019

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Model System to Analyze RNA-Mediated DNA Repair in Mammalian Cells

CRISPR journal, 2023-06, Vol.6 (3), p.289

2023

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Genomic DNA of leukemic patients: Target for clinical diagnosis of MLL rearrangements

Biotechnology journal, 2006-06, Vol.1 (6), p.656-663

2006

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Functional analysis of the two reciprocal fusion genes MLL-NEBL and NEBL-MLL reveal their oncogenic potential

Cancer letters, 2013-05, Vol.332 (1), p.30-34

2013

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The differential role of the lipid raft-associated protein flotillin 2 for progression of myeloid leukemia

Blood advances, 2022-06, Vol.6 (12), p.3611-3624

2022

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AF4 and AF4N protein complexes: recruitment of P-TEFb kinase, their interactome and potential functions

American journal of blood research, 2015-01, Vol.5 (1), p.10-24

2015

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Premature transcript termination, trans-splicing and DNA repair: a vicious path to cancer

American journal of blood research, 2011-01, Vol.1 (1), p.1-12

2011

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Three-Way Translocations and Insertions Are the Molecular Basis for Complex MLL Rearrangements

Blood, 2007-11, Vol.110 (11), p.979-979

2007

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The MLL Recombinome of Adult ALL - Results from the GMALL Study Group

Blood, 2007-11, Vol.110 (11), p.977-977

2007

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New Insights to the MLL Recombinome Including 8 Novel Partner Genes ACTN4, C2CD3, DCP1A, FLNA, LAMC3, LOC100128568, NRIP3, and TNRC18

Blood, 2008-11, Vol.112 (11), p.2047-2047

2008

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A complex MLL rearrangement identified five years after initial MDS diagnosis results in out-of-frame fusions without progression to acute leukemia

Cancer genetics, 2011-10, Vol.204 (10), p.557-562

2011

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Novel Spliced MLL Fusions Have Been Identified Involving the MLL Partner Genes ELL, EPS15, MLLT3, and SEPT5

Blood, 2007-11, Vol.110 (11), p.978-978

2007

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C/EBP Suppression by Interruption of CUGBP1 Resulting from a Complex Rearrangement of MLL

Blood, 2006-11, Vol.108 (11), p.2080-2080

2006

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Optimized Sleeping Beauty transposons rapidly generate stable transgenic cell lines

Image-Based Annotation of Chemogenomic Libraries for Phenotypic Screening

Vaccine-induced COVID-19 mimicry syndrome

MLL-AF4 and a murinized pSer-variant thereof are turning on the nucleolar stress pathway

Generation of a Sleeping Beauty Transposon-Based Cellular System for Rapid and Sensitive Screening for Compounds and Cellular Factors Limiting SARS-CoV-2 Replication

How chromosomal translocations arise to cause cancer: Gene proximity, trans-splicing, and DNA end joining

Distinct and targetable role of calcium-sensing receptor in leukaemia

The MLL recombinome of adult CD10-negative B-cell precursor acute lymphoblastic leukemia: results from the GMALL study group

Do non-genomically encoded fusion transcripts cause recurrent chromosomal translocations?

Unraveling the Activation Mechanism of Taspase1 which Controls the Oncogenic AF4–MLL Fusion Protein

The role of reciprocal fusions in MLL-r acute leukemia: studying the chromosomal translocation t(6;11)

Biology of t(6;11) Fusion Proteins and Their Role in MLL-Rearranged Acute Leukemia Lineage Determination

Model System to Analyze RNA-Mediated DNA Repair in Mammalian Cells

Genomic DNA of leukemic patients: Target for clinical diagnosis of MLL rearrangements

The insecticides permethrin and chlorpyrifos show limited genotoxicity and no leukemogenic potential in human and murine hematopoietic stem progenitor cells

Functional analysis of the two reciprocal fusion genes MLL-NEBL and NEBL-MLL reveal their oncogenic potential

The differential role of the lipid raft-associated protein flotillin 2 for progression of myeloid leukemia

AF4 and AF4N protein complexes: recruitment of P-TEFb kinase, their interactome and potential functions

Premature transcript termination, trans-splicing and DNA repair: a vicious path to cancer

Three-Way Translocations and Insertions Are the Molecular Basis for Complex MLL Rearrangements

The MLL Recombinome of Adult ALL - Results from the GMALL Study Group

New Insights to the MLL Recombinome Including 8 Novel Partner Genes ACTN4, C2CD3, DCP1A, FLNA, LAMC3, LOC100128568, NRIP3, and TNRC18

A complex MLL rearrangement identified five years after initial MDS diagnosis results in out-of-frame fusions without progression to acute leukemia

Novel Spliced MLL Fusions Have Been Identified Involving the MLL Partner Genes ELL, EPS15, MLLT3, and SEPT5

C/EBP Suppression by Interruption of CUGBP1 Resulting from a Complex Rearrangement of MLL

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