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Human Langerhans cells induce distinct IL-22-producing CD4⁺ T cells lacking IL-17 production

Proceedings of the National Academy of Sciences - PNAS, 2009-12, Vol.106 (51), p.21795-21800

2009

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Increase in TNF-α and Inducible Nitric Oxide Synthase-Expressing Dendritic Cells in Psoriasis and Reduction with Efalizumab (Anti-CD11a)

Proceedings of the National Academy of Sciences - PNAS, 2005-12, Vol.102 (52), p.19057-19062

2005

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Alefacept Reduces Infiltrating T Cells, Activated Dendritic Cells, and Inflammatory Genes in Psoriasis Vulgaris

Proceedings of the National Academy of Sciences - PNAS, 2005-02, Vol.102 (6), p.2075-2080

2005

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Post-therapeutic relapse of psoriasis after CD11a blockade is associated with T cells and inflammatory myeloid DCs

PloS one, 2012-02, Vol.7 (2), p.e30308-e30308

2012

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Atopic dermatitis keratinocytes exhibit normal TH17 cytokine responses

Journal of allergy and clinical immunology, 2010-03, Vol.125 (3), p.744-746.e2

2010

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Interleukin-11 therapy selectively downregulates type I cytokine proinflammatory pathways in psoriasis lesions

The Journal of clinical investigation, 1999-12, Vol.104 (11), p.1527-1537

1999

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Alefacept (anti-CD2) causes a selective reduction in circulating effector memory T cells (Tem) and relative preservation of central memory T cells (Tcm) in psoriasis

Journal of translational medicine, 2007-06, Vol.5 (1), p.27-27, Article 27

2007

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IL-22-producing T22 T cells account for upregulated IL-22 in atopic dermatitis despite reduced IL-17―producing TH17 T cells

Journal of allergy and clinical immunology, 2009-06, Vol.123 (6), p.1244-1252

2009

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Psoriasis Vulgaris Lesions Contain Discrete Populations of Th1 and Th17 T Cells

Journal of investigative dermatology, 2008-05, Vol.128 (5), p.1207-1211

2008

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The Majority of Epidermal T Cells in Psoriasis Vulgaris Lesions can Produce Type 1 Cytokines, Interferon-γ, Interleukin-2, and Tumor Necrosis Factor-α, Defining TC1 (Cytotoxic T Lymphocyte) and TH1 Effector Populations: a Type 1 Differentiation Bias is also Measured in Circulating Blood T Cells in Psoriatic Patients

Journal of investigative dermatology, 1999-11, Vol.113 (5), p.752-759

1999

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Blockade of CD11a by Efalizumab in Psoriasis Patients Induces a Unique State of T-Cell Hyporesponsiveness

Journal of investigative dermatology, 2008-05, Vol.128 (5), p.1182-1191

2008

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Potential role of the chemokine receptors CXCR3, CCR4, and the integrin αEβ7 in the pathogenesis of psoriasis vulgaris

Laboratory investigation, 2001-03, Vol.81 (3), p.335-347

2001

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Blockade of T lymphocyte costimulation with cytotoxic T lymphocyte-associated antigen 4-immunoglobulin (CTLA4Ig) reverses the cellular pathology of psoriatic plaques, including the activation of keratinocytes, dendritic cells, and endothelial cells

The Journal of experimental medicine, 2000-09, Vol.192 (5), p.681-694

2000

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Novel Insight into the Agonistic Mechanism of Alefacept In Vivo: Differentially Expressed Genes May Serve as Biomarkers of Response in Psoriasis Patients

Journal of Immunology, 2007-06, Vol.178 (11), p.7442-7449

2007

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312-nanometer ultraviolet B light (narrow-band UVB) induces apoptosis of T cells within psoriatic lesions

The Journal of experimental medicine, 1999-02, Vol.189 (4), p.711-718

1999

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Atopic dermatitis keratinocytes exhibit normal T(H)17 cytokine responses

Journal of allergy and clinical immunology, 2010-03, Vol.125 (3), p.744-746

2010

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Insights into Gene Modulation by Therapeutic TNF and IFNγ Antibodies: TNF Regulates IFNγ Production by T Cells and TNF-Regulated Genes Linked to Psoriasis Transcriptome

Journal of investigative dermatology, 2008-03, Vol.128 (3), p.655-666

2008

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Cell-Adhesion-Disrupting Action of Interleukin 6 in Human Ductal Breast Carcinoma Cells

Proceedings of the National Academy of Sciences - PNAS, 1994-04, Vol.91 (8), p.3329-3333

1994

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Human Langerhans cells induce distinct IL-22-producing CD4âº T cells lacking IL-17 production

Proceedings of the National Academy of Sciences - PNAS, 2009, Vol.106 (51), p.21795-21800

2009

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Human Langerhans cells induce distinct IL-22-producing CD4 super(+) T cells lacking IL-17 production

Proceedings of the National Academy of Sciences - PNAS, 2009-01, Vol.106 (51), p.21795-21800

2009

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Efalizumab (anti-CD11a)-induced increase in peripheral blood leukocytes in psoriasis patients is preferentially mediated by altered trafficking of memory CD8+ T cells into lesional skin

Clinical immunology (Orlando, Fla.), 2004-10, Vol.113 (1), p.38-46

2004

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IL-22 producing “T22” T-cells account for up-regulated IL-22 in atopic dermatitis (AD), despite reduced IL-17 producing Th17 T-cells

Journal of allergy and clinical immunology, 2009-05, Vol.123 (6), p.1244-52.e2

2009

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Atopic dermatitis keratinocytes exhibit normal T sub(H17 cytokine responses)

Journal of allergy and clinical immunology, 2010-03, Vol.125 (3), p.744-746.e2

2010

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Thalidomide Induces Granuloma Differentiation in Sarcoid Skin Lesions Associated with Disease Improvement

Clinical immunology (Orlando, Fla.), 2002-03, Vol.102 (3), p.225-236

2002

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Atopic dermatitis keratinocytes exhibit normal T H17 cytokine responses

Journal of allergy and clinical immunology, 2010, Vol.125 (3), p.744-746.e2

2010

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Human Langerhans cells induce distinct IL-22-producing CD4⁺ T cells lacking IL-17 production

Increase in TNF-α and Inducible Nitric Oxide Synthase-Expressing Dendritic Cells in Psoriasis and Reduction with Efalizumab (Anti-CD11a)

Alefacept Reduces Infiltrating T Cells, Activated Dendritic Cells, and Inflammatory Genes in Psoriasis Vulgaris

Post-therapeutic relapse of psoriasis after CD11a blockade is associated with T cells and inflammatory myeloid DCs

Atopic dermatitis keratinocytes exhibit normal TH17 cytokine responses

Interleukin-11 therapy selectively downregulates type I cytokine proinflammatory pathways in psoriasis lesions

Alefacept (anti-CD2) causes a selective reduction in circulating effector memory T cells (Tem) and relative preservation of central memory T cells (Tcm) in psoriasis

IL-22-producing T22 T cells account for upregulated IL-22 in atopic dermatitis despite reduced IL-17―producing TH17 T cells

Psoriasis Vulgaris Lesions Contain Discrete Populations of Th1 and Th17 T Cells

Blockade of CD11a by Efalizumab in Psoriasis Patients Induces a Unique State of T-Cell Hyporesponsiveness

Potential role of the chemokine receptors CXCR3, CCR4, and the integrin αEβ7 in the pathogenesis of psoriasis vulgaris

Blockade of T lymphocyte costimulation with cytotoxic T lymphocyte-associated antigen 4-immunoglobulin (CTLA4Ig) reverses the cellular pathology of psoriatic plaques, including the activation of keratinocytes, dendritic cells, and endothelial cells

Novel Insight into the Agonistic Mechanism of Alefacept In Vivo: Differentially Expressed Genes May Serve as Biomarkers of Response in Psoriasis Patients

312-nanometer ultraviolet B light (narrow-band UVB) induces apoptosis of T cells within psoriatic lesions

Atopic dermatitis keratinocytes exhibit normal T(H)17 cytokine responses

Insights into Gene Modulation by Therapeutic TNF and IFNγ Antibodies: TNF Regulates IFNγ Production by T Cells and TNF-Regulated Genes Linked to Psoriasis Transcriptome

Cell-Adhesion-Disrupting Action of Interleukin 6 in Human Ductal Breast Carcinoma Cells

Human Langerhans cells induce distinct IL-22-producing CD4âº T cells lacking IL-17 production

Human Langerhans cells induce distinct IL-22-producing CD4 super(+) T cells lacking IL-17 production

Efalizumab (anti-CD11a)-induced increase in peripheral blood leukocytes in psoriasis patients is preferentially mediated by altered trafficking of memory CD8+ T cells into lesional skin

IL-22 producing “T22” T-cells account for up-regulated IL-22 in atopic dermatitis (AD), despite reduced IL-17 producing Th17 T-cells

Atopic dermatitis keratinocytes exhibit normal T sub(H17 cytokine responses)

Thalidomide Induces Granuloma Differentiation in Sarcoid Skin Lesions Associated with Disease Improvement

Atopic dermatitis keratinocytes exhibit normal T H17 cytokine responses

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