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Mitochondrial dysfunction accounts for the stochastic heterogeneity in telomere-dependent senescence

PLoS biology, 2007-05, Vol.5 (5), p.e110-e110

2007

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On the Programmed/Non-Programmed Nature of Ageing within the Life History

Current biology, 2011-09, Vol.21 (18), p.R701-R707

2011

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Deciphering death: a commentary on Gompertz (1825) 'On the nature of the function expressive of the law of human mortality, and on a new mode of determining the value of life contingencies'

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 2015-04, Vol.370 (1666)

Kirkwood, Thomas B L

2015

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On the evolution of cellular senescence

Aging cell, 2020-12, Vol.19 (12), p.e13270-n/a

2020

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Can aging be programmed? A critical literature review

Aging cell, 2016-12, Vol.15 (6), p.986-998

2016

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Grip strength and mortality: a biomarker of ageing?

The Lancet (British edition), 2015-07, Vol.386 (9990), p.226-227

2015

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Grip Strength Decline and Its Determinants in the Very Old: Longitudinal Findings from the Newcastle 85+ Study

PloS one, 2016-09, Vol.11 (9), p.e0163183-e0163183

2016

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Age-related frailty and its association with biological markers of ageing

BMC medicine, 2015-07, Vol.13 (1), p.161-161, Article 161

2015

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Mitochondria in the signaling pathways that control longevity and health span

Ageing research reviews, 2019-09, Vol.54, p.100940-100940, Article 100940

2019

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Dynamic modelling of pathways to cellular senescence reveals strategies for targeted interventions

PLoS computational biology, 2014-08, Vol.10 (8), p.e1003728-e1003728

2014

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A systematic look at an old problem

Nature (London), 2008-02, Vol.451 (7179), p.644-647

Kirkwood, Thomas B. L

2008

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Grip strength across the life course: normative data from twelve British studies

PloS one, 2014-12, Vol.9 (12), p.e113637-e113637

2014

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Systems biology of ageing and longevity

Philosophical transactions of the Royal Society of London. Series B. Biological sciences, 2011-01, Vol.366 (1561), p.64

Kirkwood, Thomas B L

2011

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Clonal expansion of early to mid-life mitochondrial DNA point mutations drives mitochondrial dysfunction during human ageing

PLoS genetics, 2014-09, Vol.10 (9), p.e1004620-e1004620

2014

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Understanding the Odd Science of Aging

Cell, 2005-02, Vol.120 (4), p.437-447

Kirkwood, Thomas B.L.

2005

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Frailty and the role of inflammation, immunosenescence and cellular ageing in the very old: Cross-sectional findings from the Newcastle 85+ Study

Mechanisms of ageing and development, 2012-06, Vol.133 (6), p.456-466

2012

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A comparison of subjective and objective measures of physical activity from the Newcastle 85+ study

Age and ageing, 2015-07, Vol.44 (4), p.691-694

2015

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Why and how are we living longer?

Experimental physiology, 2017-09, Vol.102 (9), p.1067-1074

Kirkwood, Thomas B. L.

2017

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The free-radical theory of ageing--older, wiser and still alive: modelling positional effects of the primary targets of ROS reveals new support

BioEssays, 2012-08, Vol.34 (8), p.692-700

2012

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Prevalence and incidence of sarcopenia in the very old: findings from the Newcastle 85+ Study

Journal of cachexia, sarcopenia and muscle, 2017-04, Vol.8 (2), p.229-237

2017

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The place of genetics in ageing research

Nature reviews. Genetics, 2012-08, Vol.13 (8), p.589

2012

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Senolytics and the compression of late-life mortality

Experimental gerontology, 2021-11, Vol.155, p.111588-111588, Article 111588

2021

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Integrated Stochastic Model of DNA Damage Repair by Non-homologous End Joining and p53/p21-Mediated Early Senescence Signalling

PLoS computational biology, 2015-05, Vol.11 (5), p.e1004246-e1004246

2015

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Why do we age?

Nature (London), 2000-11, Vol.408 (6809), p.233-238

2000

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Comparison of mitochondrial mutation spectra in ageing human colonic epithelium and disease: absence of evidence for purifying selection in somatic mitochondrial DNA point mutations

PLoS genetics, 2012-11, Vol.8 (11), p.e1003082-e1003082

2012

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Mitochondrial dysfunction accounts for the stochastic heterogeneity in telomere-dependent senescence

On the Programmed/Non-Programmed Nature of Ageing within the Life History

Deciphering death: a commentary on Gompertz (1825) 'On the nature of the function expressive of the law of human mortality, and on a new mode of determining the value of life contingencies'

On the evolution of cellular senescence

Can aging be programmed? A critical literature review

Grip strength and mortality: a biomarker of ageing?

Grip Strength Decline and Its Determinants in the Very Old: Longitudinal Findings from the Newcastle 85+ Study

Age-related frailty and its association with biological markers of ageing

Mitochondria in the signaling pathways that control longevity and health span

Dynamic modelling of pathways to cellular senescence reveals strategies for targeted interventions

A systematic look at an old problem

Grip strength across the life course: normative data from twelve British studies

Systems biology of ageing and longevity

Clonal expansion of early to mid-life mitochondrial DNA point mutations drives mitochondrial dysfunction during human ageing

Understanding the Odd Science of Aging

Frailty and the role of inflammation, immunosenescence and cellular ageing in the very old: Cross-sectional findings from the Newcastle 85+ Study

A comparison of subjective and objective measures of physical activity from the Newcastle 85+ study

Why and how are we living longer?

The free-radical theory of ageing--older, wiser and still alive: modelling positional effects of the primary targets of ROS reveals new support

Prevalence and incidence of sarcopenia in the very old: findings from the Newcastle 85+ Study

The place of genetics in ageing research

Senolytics and the compression of late-life mortality

Integrated Stochastic Model of DNA Damage Repair by Non-homologous End Joining and p53/p21-Mediated Early Senescence Signalling

Why do we age?

Comparison of mitochondrial mutation spectra in ageing human colonic epithelium and disease: absence of evidence for purifying selection in somatic mitochondrial DNA point mutations

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