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Conscience, Law, and Politics

Religions (Basel, Switzerland ), 2022-12, Vol.13 (12), p.1211

Hannon, Patrick

2022

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Daily exposure to Di(2-ethylhexyl) phthalate alters estrous cyclicity and accelerates primordial follicle recruitment potentially via dysregulation of the phosphatidylinositol 3-kinase signaling pathway in adult mice

Biology of reproduction, 2014-06, Vol.90 (6), p.136

2014

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Di(2-ethylhexyl) phthalate inhibits antral follicle growth, induces atresia, and inhibits steroid hormone production in cultured mouse antral follicles

Toxicology and applied pharmacology, 2015-04, Vol.284 (1), p.42-53

2015

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Acute Exposure to Di(2-Ethylhexyl) Phthalate in Adulthood Causes Adverse Reproductive Outcomes Later in Life and Accelerates Reproductive Aging in Female Mice

Toxicological sciences, 2016-03, Vol.150 (1), p.97-108

2016

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The effects of phthalates on the ovary

Frontiers in endocrinology (Lausanne), 2015, Vol.6, p.8

2015

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Loss of Fertility in the Absence of Progesterone Receptor Expression in Kisspeptin Neurons of Female Mice

PloS one, 2016-07, Vol.11 (7), p.e0159534

2016

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Mono(2-ethylhexyl) phthalate accelerates early folliculogenesis and inhibits steroidogenesis in cultured mouse whole ovaries and antral follicles

Biology of reproduction, 2015-05, Vol.92 (5), p.120

2015

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Coordinated Regulation Among Progesterone, Prostaglandins, and EGF-Like Factors in Human Ovulatory Follicles

The journal of clinical endocrinology and metabolism, 2017-06, Vol.102 (6), p.1971-1982

2017

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The effects of endocrine‐disrupting chemicals on ovarian‐ and ovulation‐related fertility outcomes

Molecular reproduction and development, 2022-12, Vol.89 (12), p.608-631

2022

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Ovulation is Inhibited by an Environmentally Relevant Phthalate Mixture in Mouse Antral Follicles In Vitro

Toxicological sciences, 2021-01, Vol.179 (2), p.195-205

2021

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Exposure to a phthalate mixture disrupts ovulatory progesterone receptor signaling in human granulosa cells in vitro

Biology of reproduction, 2023-10, Vol.109 (4), p.552

2023

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Di-n-Butyl Phthalate Disrupts the Expression of Genes Involved in Cell Cycle and Apoptotic Pathways in Mouse Ovarian Antral Follicles

Biology of reproduction, 2013, Vol.88 (1), p.23

2013

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Granulosa cell endothelin-2 expression is fundamental for ovulatory follicle rupture

Scientific reports, 2017-04, Vol.7 (1), p.817-11, Article 817

2017

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Di (2-ethylhexyl) phthalate (DEHP) alters proliferation and uterine gland numbers in the uteri of adult exposed mice

Reproductive toxicology (Elmsford, N.Y.), 2018-04, Vol.77, p.70-79

2018

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Neurotensin expression, regulation, and function during the ovulatory period in the mouse ovary

Biology of reproduction, 2023-01, Vol.108 (1), p.107-120

2023

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Ovulatory Induction of SCG2 in Human, Nonhuman Primate, and Rodent Granulosa Cells Stimulates Ovarian Angiogenesis

Endocrinology (Philadelphia), 2018-06, Vol.159 (6), p.2447-2458

2018

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Dioxin exposure reduces the steroidogenic capacity of mouse antral follicles mainly at the level of HSD17B1 without altering atresia

Toxicology and applied pharmacology, 2012-10, Vol.264 (1), p.1-12

2012

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Daily Exposure to Di(2-ethylhexyl) Phthalate Alters Estrous Cyclicity and Accelerates Primordial Follicle Recruitment Potentially Via Dysregulation of the Phosphatidylinositol 3-Kinase Signaling Pathway in Adult Mice1

Biology of reproduction, 2014-06, Vol.90 (6)

2014

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Pregnenolone co-treatment partially restores steroidogenesis, but does not prevent growth inhibition and increased atresia in mouse ovarian antral follicles treated with mono-hydroxy methoxychlor

Toxicology and applied pharmacology, 2013-11, Vol.272 (3), p.780-786

2013

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Mono(2-Ethylhexyl) Phthalate Accelerates Early Folliculogenesis and Inhibits Steroidogenesis in Cultured Mouse Whole Ovaries and Antral Follicles1

Biology of reproduction, 2015-05, Vol.92 (5)

2015

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Di-n-Butyl Phthalate Disrupts the Expression of Genes Involved in Cell Cycle and Apoptotic Pathways in Mouse Ovarian Antral Follicles1

Biology of reproduction, 2013-01, Vol.88 (1)

2013

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Estrogen receptor alpha overexpressing mouse antral follicles are sensitive to atresia induced by methoxychlor and its metabolites

Reproductive toxicology (Elmsford, N.Y.), 2012-06, Vol.33 (3), p.353-360

2012

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Church and State in Ireland: Perspectives of Vatican II

The Irish theological quarterly, 2017-05, Vol.82 (2), p.97-112

Hannon, Patrick

2017

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A brief review of current orthopedic implant device issues: biomechanics and biocompatibility

Biology, engineering and medicine, 2016, Vol.1 (1)

Hannon, Patrick

2016

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NEJM Evidence — A New Journal in the NEJM Group Family

The New England journal of medicine, 2022-01, Vol.386 (2), p.182-183

2022

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Conscience, Law, and Politics

Daily exposure to Di(2-ethylhexyl) phthalate alters estrous cyclicity and accelerates primordial follicle recruitment potentially via dysregulation of the phosphatidylinositol 3-kinase signaling pathway in adult mice

Di(2-ethylhexyl) phthalate inhibits antral follicle growth, induces atresia, and inhibits steroid hormone production in cultured mouse antral follicles

Acute Exposure to Di(2-Ethylhexyl) Phthalate in Adulthood Causes Adverse Reproductive Outcomes Later in Life and Accelerates Reproductive Aging in Female Mice

The effects of phthalates on the ovary

Loss of Fertility in the Absence of Progesterone Receptor Expression in Kisspeptin Neurons of Female Mice

Mono(2-ethylhexyl) phthalate accelerates early folliculogenesis and inhibits steroidogenesis in cultured mouse whole ovaries and antral follicles

Coordinated Regulation Among Progesterone, Prostaglandins, and EGF-Like Factors in Human Ovulatory Follicles

The effects of endocrine‐disrupting chemicals on ovarian‐ and ovulation‐related fertility outcomes

Ovulation is Inhibited by an Environmentally Relevant Phthalate Mixture in Mouse Antral Follicles In Vitro

Exposure to a phthalate mixture disrupts ovulatory progesterone receptor signaling in human granulosa cells in vitro

Di-n-Butyl Phthalate Disrupts the Expression of Genes Involved in Cell Cycle and Apoptotic Pathways in Mouse Ovarian Antral Follicles

Granulosa cell endothelin-2 expression is fundamental for ovulatory follicle rupture

Di (2-ethylhexyl) phthalate (DEHP) alters proliferation and uterine gland numbers in the uteri of adult exposed mice

Neurotensin expression, regulation, and function during the ovulatory period in the mouse ovary

Ovulatory Induction of SCG2 in Human, Nonhuman Primate, and Rodent Granulosa Cells Stimulates Ovarian Angiogenesis

Dioxin exposure reduces the steroidogenic capacity of mouse antral follicles mainly at the level of HSD17B1 without altering atresia

Daily Exposure to Di(2-ethylhexyl) Phthalate Alters Estrous Cyclicity and Accelerates Primordial Follicle Recruitment Potentially Via Dysregulation of the Phosphatidylinositol 3-Kinase Signaling Pathway in Adult Mice1

Pregnenolone co-treatment partially restores steroidogenesis, but does not prevent growth inhibition and increased atresia in mouse ovarian antral follicles treated with mono-hydroxy methoxychlor

Mono(2-Ethylhexyl) Phthalate Accelerates Early Folliculogenesis and Inhibits Steroidogenesis in Cultured Mouse Whole Ovaries and Antral Follicles1

Di-n-Butyl Phthalate Disrupts the Expression of Genes Involved in Cell Cycle and Apoptotic Pathways in Mouse Ovarian Antral Follicles1

Estrogen receptor alpha overexpressing mouse antral follicles are sensitive to atresia induced by methoxychlor and its metabolites

Church and State in Ireland: Perspectives of Vatican II

A brief review of current orthopedic implant device issues: biomechanics and biocompatibility

NEJM Evidence — A New Journal in the NEJM Group Family

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