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Hepatic effects of a highly purified 2,2′,3,4,4′,5,5′-heptachlorbiphenyl (PCB 180) in male and female rats

Toxicology (Amsterdam), 2011-06, Vol.284 (1), p.42-53

2011

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Role of retinoids in biology and toxicology

Reproductive toxicology (Elmsford, N.Y.), 2022-01, Vol.107, p.40-42

Hakansson, H

2022

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Cumulative health risk assessment of 17 perfluoroalkylated and polyfluoroalkylated substances (PFASs) in the Swedish population

Environment international, 2013-09, Vol.59, p.112-123

2013

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Regulatory needs and activities to address the retinoid system in the context of endocrine disruption: The European viewpoint

Reproductive toxicology (Elmsford, N.Y.), 2020-04, Vol.93, p.250-258

2020

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The 2005 World Health Organization Reevaluation of Human and Mammalian Toxic Equivalency Factors for Dioxins and Dioxin-Like Compounds

Toxicological sciences, 2006-10, Vol.93 (2), p.223-241

2006

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Risk to all or none? A comparative analysis of controversies in the health risk assessment of Bisphenol A

Reproductive toxicology (Elmsford, N.Y.), 2010-04, Vol.29 (2), p.132-146

2010

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Risk for animal and human health related to the presence of dioxins and dioxin‐like PCBs in feed and food

EFSA journal, 2018-11, Vol.16 (11), p.e05333-n/a

2018

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Scientific principles for the identification of endocrine-disrupting chemicals: a consensus statement

Archives of toxicology, 2017-02, Vol.91 (2), p.1001-1006

2017

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Gender- and dose-related metabolome alterations in rat offspring after in utero and lactational exposure to PCB 180

Toxicology and applied pharmacology, 2019-05, Vol.370, p.56-64

2019

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Phthalate Diesters and Their Metabolites in Human Breast Milk, Blood or Serum, and Urine as Biomarkers of Exposure in Vulnerable Populations

Environmental health perspectives, 2008-03, Vol.116 (3), p.334-339

2008

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Skeletal and dental effects on rats following in utero/lactational exposure to the non-dioxin-like polychlorinated biphenyl PCB 180

PloS one, 2017-09, Vol.12 (9), p.e0185241

2017

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Effects of a high-fat diet and global aryl hydrocarbon receptor deficiency on energy balance and liver retinoid status in male Sprague-Dawley rats

The Journal of nutritional biochemistry, 2021-09, Vol.95, p.108762, Article 108762

2021

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Bone toxicity induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and the retinoid system: A causality analysis anchored in osteoblast gene expression and mouse data

Reproductive toxicology (Elmsford, N.Y.), 2021-10, Vol.105, p.25-43

2021

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Role of aryl hydrocarbon receptor (AHR) in overall retinoid metabolism: Response comparisons to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) exposure between wild-type and AHR knockout mice

Reproductive toxicology (Elmsford, N.Y.), 2021-04, Vol.101, p.33-49

2021

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Toxicological characterisation of two novel selective aryl hydrocarbon receptor modulators in Sprague-Dawley rats

Toxicology and applied pharmacology, 2017-07, Vol.326, p.54-65

2017

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Individual breast milk consumption and exposure to PCBs and PCDD/Fs in Hungarian infants: A time-course analysis of the first three months of lactation

The Science of the total environment, 2013-04, Vol.449, p.336-344

2013

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Effects of polybrominated diphenyl ethers (PBDEs) and polychlorinated biphenyls (PCBs) on thyroid hormone and vitamin A levels in rats and mice

Archives of toxicology, 2001-06, Vol.75 (4), p.200-208

2001

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A 28-Day Oral Dose Toxicity Study Enhanced to Detect Endocrine Effects of Hexabromocyclododecane in Wistar Rats

Toxicological sciences, 2006-12, Vol.94 (2), p.281-292

2006

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Endocrine, metabolic and apical effects of in utero and lactational exposure to non-dioxin-like 2,2′,3,4,4′,5,5′-heptachlorobiphenyl (PCB 180): A postnatal follow-up study in rats

Reproductive toxicology (Elmsford, N.Y.), 2021-06, Vol.102, p.109-127

2021

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Endocrine effects of tetrabromobisphenol-A (TBBPA) in Wistar rats as tested in a one-generation reproduction study and a subacute toxicity study

Toxicology (Amsterdam), 2008-03, Vol.245 (1), p.76-89

2008

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Estrogen-Like Effects of Cadmium in Vivo Do Not Appear to be Mediated via the Classical Estrogen Receptor Transcriptional Pathway

Environmental health perspectives, 2010-10, Vol.118 (10), p.1389-1394

2010

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Identification of the Tryptophan Photoproduct 6-Formylindolo[3,2-b]carbazole, in Cell Culture Medium, as a Factor That Controls the Background Aryl Hydrocarbon Receptor Activity

Toxicological sciences, 2005-06, Vol.85 (2), p.935-943

2005

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In utero /lactational and adult exposures to 2,3,7,8-tetrachlorodibenzo- p -dioxin (TCDD) show differential effects on craniofacial development and growth in rats

Toxicology (Amsterdam), 2015-11, Vol.337, p.30-38

2015

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Retinoic Acid Drives Aryl Hydrocarbon Receptor Expression and Is Instrumental to Dioxin-Induced Toxicity during Palate Development

Environmental health perspectives, 2011-11, Vol.119 (11), p.1590-1595

2011

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New insights to the role of aryl hydrocarbon receptor in bone phenotype and in dioxin-induced modulation of bone microarchitecture and material properties

Toxicology and applied pharmacology, 2013-11, Vol.273 (1), p.219-226

2013

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Hepatic effects of a highly purified 2,2′,3,4,4′,5,5′-heptachlorbiphenyl (PCB 180) in male and female rats

Role of retinoids in biology and toxicology

Cumulative health risk assessment of 17 perfluoroalkylated and polyfluoroalkylated substances (PFASs) in the Swedish population

Regulatory needs and activities to address the retinoid system in the context of endocrine disruption: The European viewpoint

The 2005 World Health Organization Reevaluation of Human and Mammalian Toxic Equivalency Factors for Dioxins and Dioxin-Like Compounds

Risk to all or none? A comparative analysis of controversies in the health risk assessment of Bisphenol A

Risk for animal and human health related to the presence of dioxins and dioxin‐like PCBs in feed and food

Scientific principles for the identification of endocrine-disrupting chemicals: a consensus statement

Gender- and dose-related metabolome alterations in rat offspring after in utero and lactational exposure to PCB 180

Phthalate Diesters and Their Metabolites in Human Breast Milk, Blood or Serum, and Urine as Biomarkers of Exposure in Vulnerable Populations

Skeletal and dental effects on rats following in utero/lactational exposure to the non-dioxin-like polychlorinated biphenyl PCB 180

Effects of a high-fat diet and global aryl hydrocarbon receptor deficiency on energy balance and liver retinoid status in male Sprague-Dawley rats

Bone toxicity induced by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) and the retinoid system: A causality analysis anchored in osteoblast gene expression and mouse data

Role of aryl hydrocarbon receptor (AHR) in overall retinoid metabolism: Response comparisons to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) exposure between wild-type and AHR knockout mice

Toxicological characterisation of two novel selective aryl hydrocarbon receptor modulators in Sprague-Dawley rats

Individual breast milk consumption and exposure to PCBs and PCDD/Fs in Hungarian infants: A time-course analysis of the first three months of lactation

Effects of polybrominated diphenyl ethers (PBDEs) and polychlorinated biphenyls (PCBs) on thyroid hormone and vitamin A levels in rats and mice

A 28-Day Oral Dose Toxicity Study Enhanced to Detect Endocrine Effects of Hexabromocyclododecane in Wistar Rats

Endocrine, metabolic and apical effects of in utero and lactational exposure to non-dioxin-like 2,2′,3,4,4′,5,5′-heptachlorobiphenyl (PCB 180): A postnatal follow-up study in rats

Endocrine effects of tetrabromobisphenol-A (TBBPA) in Wistar rats as tested in a one-generation reproduction study and a subacute toxicity study

Estrogen-Like Effects of Cadmium in Vivo Do Not Appear to be Mediated via the Classical Estrogen Receptor Transcriptional Pathway

Identification of the Tryptophan Photoproduct 6-Formylindolo[3,2-b]carbazole, in Cell Culture Medium, as a Factor That Controls the Background Aryl Hydrocarbon Receptor Activity

In utero /lactational and adult exposures to 2,3,7,8-tetrachlorodibenzo- p -dioxin (TCDD) show differential effects on craniofacial development and growth in rats

Retinoic Acid Drives Aryl Hydrocarbon Receptor Expression and Is Instrumental to Dioxin-Induced Toxicity during Palate Development

New insights to the role of aryl hydrocarbon receptor in bone phenotype and in dioxin-induced modulation of bone microarchitecture and material properties

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